A citation-based method for searching scientific literature

Mingzhao Xing. Nat Rev Cancer 2013
Times Cited: 851

List of co-cited articles
563 articles co-cited >1

Times Cited
  Times     Co-cited

Congenital hypothyroidism, dwarfism, and hearing impairment caused by a missense mutation in the mouse dual oxidase 2 gene, Duox2.
Kenneth R Johnson, Coleen C Marden, Patricia Ward-Bailey, Leona H Gagnon, Roderick T Bronson, Leah Rae Donahue. Mol Endocrinol 2007

Defective oxidative phosphorylation in thyroid oncocytic carcinoma is associated with pathogenic mitochondrial DNA mutations affecting complexes I and III.
Elena Bonora, Anna Maria Porcelli, Giuseppe Gasparre, Annalisa Biondi, Anna Ghelli, Valerio Carelli, Alessandra Baracca, Giovanni Tallini, Andrea Martinuzzi, Giorgio Lenaz,[...]. Cancer Res 2006

Thyroid Na+/I- symporter. Mechanism, stoichiometry, and specificity.
S Eskandari, D D Loo, G Dai, O Levy, E M Wright, N Carrasco. J Biol Chem 1997

The KCNQ1-KCNE2 K⁺ channel is required for adequate thyroid I⁻ uptake.
Kerry Purtell, Monika Paroder-Belenitsky, Andrea Reyna-Neyra, Juan P Nicola, Wade Koba, Eugene Fine, Nancy Carrasco, Geoffrey W Abbott. FASEB J 2012

Overexpression of NOX4 predicts poor prognosis and promotes tumor progression in human colorectal cancer.
Xiao-Lu Lin, Li Yang, Seng-Wang Fu, Wen-Feng Lin, Yun-Jie Gao, Hao-Yan Chen, Zhi-Zheng Ge. Oncotarget 2017

(V600E)BRAF is associated with disabled feedback inhibition of RAF-MEK signaling and elevated transcriptional output of the pathway.
Christine A Pratilas, Barry S Taylor, Qing Ye, Agnes Viale, Chris Sander, David B Solit, Neal Rosen. Proc Natl Acad Sci U S A 2009

Modulation of oxidative stress as an anticancer strategy.
Chiara Gorrini, Isaac S Harris, Tak W Mak. Nat Rev Drug Discov 2013

Inactivating mutations in the gene for thyroid oxidase 2 (THOX2) and congenital hypothyroidism.
José C Moreno, Hennie Bikker, Marlies J E Kempers, A S Paul van Trotsenburg, Frank Baas, Jan J M de Vijlder, Thomas Vulsma, C Ris-Stalpers. N Engl J Med 2002

Excess iodide induces an acute inhibition of the sodium/iodide symporter in thyroid male rat cells by increasing reactive oxygen species.
Alejandro A Arriagada, Eduardo Albornoz, Ma Cecilia Opazo, Alvaro Becerra, Gonzalo Vidal, Carlos Fardella, Luis Michea, Nancy Carrasco, Felipe Simon, Alvaro A Elorza,[...]. Endocrinology 2015

Regulation of ROS signal transduction by NADPH oxidase 4 localization.
Kai Chen, Michael T Kirber, Hui Xiao, Yu Yang, John F Keaney. J Cell Biol 2008

Iodide excess induces apoptosis in thyroid cells through a p53-independent mechanism involving oxidative stress.
M Vitale, T Di Matola, F D'Ascoli, S Salzano, F Bogazzi, G Fenzi, E Martino, G Rossi. Endocrinology 2000

ROS-generating NADPH oxidase NOX4 is a critical mediator in oncogenic H-Ras-induced DNA damage and subsequent senescence.
U Weyemi, O Lagente-Chevallier, M Boufraqech, F Prenois, F Courtin, B Caillou, M Talbot, M Dardalhon, A Al Ghuzlan, J-M Bidart,[...]. Oncogene 2012

Essential role of NADPH oxidase-dependent reactive oxygen species generation in regulating microRNA-21 expression and function in prostate cancer.
Sarvesh Jajoo, Debashree Mukherjea, Tejbeer Kaur, Kelly E Sheehan, Sandeep Sheth, Vikrant Borse, Leonard P Rybak, Vickram Ramkumar. Antioxid Redox Signal 2013

Direct interaction of the novel Nox proteins with p22phox is required for the formation of a functionally active NADPH oxidase.
Rashmi K Ambasta, Pravir Kumar, Kathy K Griendling, Harald H H W Schmidt, Rudi Busse, Ralf P Brandes. J Biol Chem 2004

Co-cited is the co-citation frequency, indicating how many articles cite the article together with the query article. Similarity is the co-citation as percentage of the times cited of the query article or the article in the search results, whichever is the lowest. These numbers are calculated for the last 100 citations when articles are cited more than 100 times.