P S Gaynon, A L Carrel. Adv Exp Med Biol 1999
Times Cited: 101
Times Cited: 101
Times Cited
Times Co-cited
Similarity
Microarray analysis uncovers the induction of the proapoptotic BH3-only protein Bim in multiple models of glucocorticoid-induced apoptosis.
Zhengqi Wang, Michael H Malone, Huiling He, Karen S McColl, Clark W Distelhorst. J Biol Chem 2003
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The molecular basis of glucocorticoid-induced apoptosis of lymphoblastic leukemia cells.
R Kofler. Histochem Cell Biol 2000
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17
Recent insights into the mechanism of glucocorticosteroid-induced apoptosis.
C W Distelhorst. Cell Death Differ 2002
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Mechanisms of glucocorticoid-mediated apoptosis in hematological malignancies.
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Glucocorticoid-induced apoptosis and glucocorticoid resistance: molecular mechanisms and clinical relevance.
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Prednisone response is the strongest predictor of treatment outcome in infant acute lymphoblastic leukemia.
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Molecular determinants of glucocorticoid sensitivity and resistance in acute lymphoblastic leukemia.
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Long-term results of four consecutive trials in childhood ALL performed by the ALL-BFM study group from 1981 to 1995. Berlin-Frankfurt-Münster.
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Glucocorticoid-induced cell death requires autoinduction of glucocorticoid receptor expression in human leukemic T cells.
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Glucocorticoid resistance in childhood leukemia.
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Glucocorticoid receptors and resistance to glucocorticoids in hematologic malignancies.
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Identification of glucocorticoid-response genes in children with acute lymphoblastic leukemia.
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Comparison of the antileukemic activity in vitro of dexamethasone and prednisolone in childhood acute lymphoblastic leukemia.
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[Corticosteroid-dependent reduction of leukocyte count in blood as a prognostic factor in acute lymphoblastic leukemia in childhood (therapy study ALL-BFM 83)].
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Dexamethasone resistance in B-cell precursor childhood acute lymphoblastic leukemia occurs downstream of ligand-induced nuclear translocation of the glucocorticoid receptor.
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Glucocorticoid-induced apoptosis of human leukemic cells is caused by the repressive function of the glucocorticoid receptor.
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Prednisolone resistance in childhood acute lymphoblastic leukemia: vitro-vivo correlations and cross-resistance to other drugs.
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Proapoptotic Bcl-2 relative Bim required for certain apoptotic responses, leukocyte homeostasis, and to preclude autoimmunity.
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Glucocorticoid receptors in lymphoma cells in culture: relationship to glucocorticoid killing activity.
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Identification of genes regulated by dexamethasone in multiple myeloma cells using oligonucleotide arrays.
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Glucocorticoid-receptor-gene defects and resistance to glucocorticoid-induced apoptosis in human leukemic cell lines.
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Cognitive sequelae in children treated for acute lymphoblastic leukemia with dexamethasone or prednisone.
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8
Expression profiling of glucocorticoid-treated T-ALL cell lines: rapid repression of multiple genes involved in RNA-, protein- and nucleotide synthesis.
P Obexer, U Certa, R Kofler, A Helmberg. Oncogene 2001
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Osteonecrosis as a complication of treating acute lymphoblastic leukemia in children: a report from the Children's Cancer Group.
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8
Role of apical caspases and glucocorticoid-regulated genes in glucocorticoid-induced apoptosis of pre-B leukemic cells.
Sonia L Planey, Marc T Abrams, Noreen M Robertson, Gerald Litwack. Cancer Res 2003
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Steroid receptor interactions with heat shock protein and immunophilin chaperones.
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Glucocorticoid resistance in childhood leukaemia: mechanisms and modulation.
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18
Gene-expression patterns in drug-resistant acute lymphoblastic leukemia cells and response to treatment.
Amy Holleman, Meyling H Cheok, Monique L den Boer, Wenjian Yang, Anjo J P Veerman, Karin M Kazemier, Deqing Pei, Cheng Cheng, Ching-Hon Pui, Mary V Relling,[...]. N Engl J Med 2004
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8
Inhibition of glucocorticoid-induced apoptosis by targeting the major splice variants of BIM mRNA with small interfering RNA and short hairpin RNA.
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Marc T Abrams, Noreen M Robertson, Kyonggeun Yoon, Eric Wickstrom. J Biol Chem 2004
8
Glucocorticoid resistance in two key models of acute lymphoblastic leukemia occurs at the level of the glucocorticoid receptor.
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14
Glucocorticoid-induced thymocyte apoptosis is associated with endogenous endonuclease activation.
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Glucocorticoid receptor gene mutations in leukemic cells acquired in vitro and in vivo.
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DNA binding of the glucocorticoid receptor is not essential for survival.
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Bcl-2 interferes with the execution phase, but not upstream events, in glucocorticoid-induced leukemia apoptosis.
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Lower incidence of meningeal leukemia when prednisone is replaced by dexamethasone in the treatment of acute lymphocytic leukemia.
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Differences in cerebrospinal fluid penetration of corticosteroids: possible relationship to the prevention of meningeal leukemia.
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Analysis of gene expression patterns during glucocorticoid-induced apoptosis using oligonucleotide arrays.
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Substituting dexamethasone for prednisone complicates remission induction in children with acute lymphoblastic leukemia.
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Resistance of human leukemic CEM-C1 cells is overcome by synergism between glucocorticoid and protein kinase A pathways: correlation with c-Myc suppression.
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Suppression of c-myc is a critical step in glucocorticoid-induced human leukemic cell lysis.
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Improved outcome for children with acute lymphoblastic leukemia: results of Dana-Farber Consortium Protocol 91-01.
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Identification of the activation-labile gene: a single point mutation in the human glucocorticoid receptor presents as two distinct receptor phenotypes.
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Low glucocorticoid receptor alpha/beta ratio in T-cell lymphoblastic leukemia.
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The use of glucocorticoids in acute lymphoblastic leukemia of childhood. Molecular, cellular, and clinical considerations.
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Gene expression profile of human lymphoid CEM cells sensitive and resistant to glucocorticoid-evoked apoptosis.
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S L Planey, G Litwack. Biochem Biophys Res Commun 2000
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