A citation-based method for searching scientific literature

T Iwata, L Chen, C Li, D A Ovchinnikov, R R Behringer, C A Francomano, C X Deng. Hum Mol Genet 2000
Times Cited: 157







List of co-cited articles
1402 articles co-cited >1



Times Cited
  Times     Co-cited
Similarity


Fibroblast growth factor receptor 3 is a negative regulator of bone growth.
C Deng, A Wynshaw-Boris, F Zhou, A Kuo, P Leder. Cell 1996
834
50

Skeletal overgrowth and deafness in mice lacking fibroblast growth factor receptor 3.
J S Colvin, B A Bohne, G W Harding, D G McEwen, D M Ornitz. Nat Genet 1996
687
49



Gly369Cys mutation in mouse FGFR3 causes achondroplasia by affecting both chondrogenesis and osteogenesis.
L Chen, R Adar, X Yang, E O Monsonego, C Li, P V Hauschka, A Yayon, C X Deng. J Clin Invest 1999
188
39

Constitutive activation of MEK1 in chondrocytes causes Stat1-independent achondroplasia-like dwarfism and rescues the Fgfr3-deficient mouse phenotype.
Shunichi Murakami, Gener Balmes, Sandra McKinney, Zhaoping Zhang, David Givol, Benoit de Crombrugghe. Genes Dev 2004
198
32



Coordination of chondrogenesis and osteogenesis by fibroblast growth factor 18.
Zhonghao Liu, Jingsong Xu, Jennifer S Colvin, David M Ornitz. Genes Dev 2002
337
30

Mutations in the gene encoding fibroblast growth factor receptor-3 in achondroplasia.
F Rousseau, J Bonaventure, L Legeai-Mallet, A Pelet, J M Rozet, P Maroteaux, M Le Merrer, A Munnich. Nature 1994
649
29



A mouse model for achondroplasia produced by targeting fibroblast growth factor receptor 3.
Y Wang, M K Spatz, K Kannan, H Hayk, A Avivi, M Gorivodsky, M Pines, A Yayon, P Lonai, D Givol. Proc Natl Acad Sci U S A 1999
166
27

FGF18 is required for normal cell proliferation and differentiation during osteogenesis and chondrogenesis.
Norihiko Ohbayashi, Masaki Shibayama, Yoko Kurotaki, Mayumi Imanishi, Toshihiko Fujimori, Nobuyuki Itoh, Shinji Takada. Genes Dev 2002
327
27

Mutations in the transmembrane domain of FGFR3 cause the most common genetic form of dwarfism, achondroplasia.
R Shiang, L M Thompson, Y Z Zhu, D M Church, T J Fielder, M Bocian, S T Winokur, J J Wasmuth. Cell 1994
917
26

Interaction of FGF, Ihh/Pthlh, and BMP signaling integrates chondrocyte proliferation and hypertrophic differentiation.
Eleonora Minina, Conny Kreschel, Michael C Naski, David M Ornitz, Andrea Vortkamp. Dev Cell 2002
310
26

FGF signaling in the developing endochondral skeleton.
David M Ornitz. Cytokine Growth Factor Rev 2005
242
24


Cellular signaling by fibroblast growth factor receptors.
V P Eswarakumar, I Lax, J Schlessinger. Cytokine Growth Factor Rev 2005
23


Overexpression of CNP in chondrocytes rescues achondroplasia through a MAPK-dependent pathway.
Akihiro Yasoda, Yasato Komatsu, Hideki Chusho, Takashi Miyazawa, Ami Ozasa, Masako Miura, Tatsuya Kurihara, Tomohiro Rogi, Shoji Tanaka, Michio Suda,[...]. Nat Med 2004
262
21

FGF signaling inhibits chondrocyte proliferation and regulates bone development through the STAT-1 pathway.
M Sahni, D C Ambrosetti, A Mansukhani, R Gertner, D Levy, C Basilico. Genes Dev 1999
279
21

Unique expression pattern of the FGF receptor 3 gene during mouse organogenesis.
K Peters, D Ornitz, S Werner, L Williams. Dev Biol 1993
409
21


Thanatophoric dysplasia (types I and II) caused by distinct mutations in fibroblast growth factor receptor 3.
P L Tavormina, R Shiang, L M Thompson, Y Z Zhu, D J Wilkin, R S Lachman, W R Wilcox, D L Rimoin, D H Cohn, J J Wasmuth. Nat Genet 1995
477
20


Conditional inactivation of FGF receptor 2 reveals an essential role for FGF signaling in the regulation of osteoblast function and bone growth.
Kai Yu, Jingsong Xu, Zhonghao Liu, Drazen Sosic, Jiansu Shao, Eric N Olson, Dwight A Towler, David M Ornitz. Development 2003
452
20

Restrained chondrocyte proliferation and maturation with abnormal growth plate vascularization and ossification in human FGFR-3(G380R) transgenic mice.
O Segev, I Chumakov, Z Nevo, D Givol, L Madar-Shapiro, Y Sheinin, M Weinreb, A Yayon. Hum Mol Genet 2000
77
25

Defective bone mineralization and osteopenia in young adult FGFR3-/- mice.
Gladys Valverde-Franco, Hanlong Liu, David Davidson, Sen Chai, Hector Valderrama-Carvajal, David Goltzman, David M Ornitz, Janet E Henderson. Hum Mol Genet 2004
96
19

Activation of Stat1 by mutant fibroblast growth-factor receptor in thanatophoric dysplasia type II dwarfism.
W C Su, M Kitagawa, N Xue, B Xie, S Garofalo, J Cho, C Deng, W A Horton, X Y Fu. Nature 1997
251
19

PTH/PTHrP receptor in early development and Indian hedgehog-regulated bone growth.
B Lanske, A C Karaplis, K Lee, A Luz, A Vortkamp, A Pirro, M Karperien, L H Defize, C Ho, R C Mulligan,[...]. Science 1996
952
18

Achondroplasia.
William A Horton, Judith G Hall, Jacqueline T Hecht. Lancet 2007
269
18

Regulation of rate of cartilage differentiation by Indian hedgehog and PTH-related protein.
A Vortkamp, K Lee, B Lanske, G V Segre, H M Kronenberg, C J Tabin. Science 1996
17

A network of transcriptional and signaling events is activated by FGF to induce chondrocyte growth arrest and differentiation.
Lisa Dailey, Emmanuel Laplantine, Riccardo Priore, Claudio Basilico. J Cell Biol 2003
120
17

A novel mutation in FGFR3 causes camptodactyly, tall stature, and hearing loss (CATSHL) syndrome.
Reha M Toydemir, Anna E Brassington, Pinar Bayrak-Toydemir, Patrycja A Krakowiak, Lynn B Jorde, Frank G Whitby, Nicola Longo, David H Viskochil, John C Carey, Michael J Bamshad. Am J Hum Genet 2006
85
20


FGFR3 promotes synchondrosis closure and fusion of ossification centers through the MAPK pathway.
Takehiko Matsushita, William R Wilcox, Yuk Yu Chan, Aya Kawanami, Hülya Bükülmez, Gener Balmes, Pavel Krejci, Pertchoui B Mekikian, Kazuyuki Otani, Isakichi Yamaura,[...]. Hum Mol Genet 2009
73
23

Gain-of-function mutation in FGFR3 in mice leads to decreased bone mass by affecting both osteoblastogenesis and osteoclastogenesis.
Nan Su, Qidi Sun, Can Li, Xiumin Lu, Huabing Qi, Siyu Chen, Jing Yang, Xiaolan Du, Ling Zhao, Qifen He,[...]. Hum Mol Genet 2010
64
26

Development of the endochondral skeleton.
Fanxin Long, David M Ornitz. Cold Spring Harb Perspect Biol 2013
290
17

A recurrent mutation in the tyrosine kinase domain of fibroblast growth factor receptor 3 causes hypochondroplasia.
G A Bellus, I McIntosh, E A Smith, A S Aylsworth, I Kaitila, W A Horton, G A Greenhaw, J T Hecht, C A Francomano. Nat Genet 1995
332
16


Fibroblast growth factor (FGF) 18 signals through FGF receptor 3 to promote chondrogenesis.
David Davidson, Antoine Blanc, Dominic Filion, Huifen Wang, Paul Plut, Gerald Pfeffer, Michael D Buschmann, Janet E Henderson. J Biol Chem 2005
169
15



The IIIc alternative of Fgfr2 is a positive regulator of bone formation.
Vereragavan P Eswarakumar, Efrat Monsonego-Ornan, Mark Pines, Ileana Antonopoulou, Gillian M Morriss-Kay, Peter Lonai. Development 2002
192
14

Lethal skeletal dysplasia from targeted disruption of the parathyroid hormone-related peptide gene.
A C Karaplis, A Luz, J Glowacki, R T Bronson, V L Tybulewicz, H M Kronenberg, R C Mulligan. Genes Dev 1994
871
14

Spatio-temporal expression of FGFR 1, 2 and 3 genes during human embryo-fetal ossification.
A L Delezoide, C Benoist-Lasselin, L Legeai-Mallet, M Le Merrer, A Munnich, M Vekemans, J Bonaventure. Mech Dev 1998
136
14

Fibroblast growth factor receptor 3 mutations promote apoptosis but do not alter chondrocyte proliferation in thanatophoric dysplasia.
L Legeai-Mallet, C Benoist-Lasselin, A L Delezoide, A Munnich, J Bonaventure. J Biol Chem 1998
115
14

Skeletal dysplasia and defective chondrocyte differentiation by targeted overexpression of fibroblast growth factor 9 in transgenic mice.
S Garofalo, M Kliger-Spatz, J L Cooke, O Wolstin, G P Lunstrum, S M Moshkovitz, W A Horton, A Yayon. J Bone Miner Res 1999
75
17

Disruption of the fibroblast growth factor-2 gene results in decreased bone mass and bone formation.
A Montero, Y Okada, M Tomita, M Ito, H Tsurukami, T Nakamura, T Doetschman, J D Coffin, M M Hurley. J Clin Invest 2000
340
13


Co-cited is the co-citation frequency, indicating how many articles cite the article together with the query article. Similarity is the co-citation as percentage of the times cited of the query article or the article in the search results, whichever is the lowest. These numbers are calculated for the last 100 citations when articles are cited more than 100 times.