J Savill. Kidney Blood Press Res 2000
Times Cited: 35
Times Cited: 35
Times Cited
Times Co-cited
Similarity
Macrophages that have ingested apoptotic cells in vitro inhibit proinflammatory cytokine production through autocrine/paracrine mechanisms involving TGF-beta, PGE2, and PAF.
V A Fadok, D L Bratton, A Konowal, P W Freed, J Y Westcott, P M Henson. J Clin Invest 1998
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Apoptotic cells induce migration of phagocytes via caspase-3-mediated release of a lipid attraction signal.
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Phosphatidylserine-dependent ingestion of apoptotic cells promotes TGF-beta1 secretion and the resolution of inflammation.
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Appearance of phosphatidylserine on apoptotic cells requires calcium-mediated nonspecific flip-flop and is enhanced by loss of the aminophospholipid translocase.
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Exposure of phosphatidylserine is a general feature in the phagocytosis of apoptotic lymphocytes by macrophages.
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Phagocytosis and clearance of apoptotic cells is mediated by MER.
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CD95/CD95L-mediated apoptosis of the hepatic stellate cell. A mechanism terminating uncontrolled hepatic stellate cell proliferation during hepatic tissue repair.
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Co-cited is the co-citation frequency, indicating how many articles cite the article together with the query article. Similarity is the co-citation as percentage of the times cited of the query article or the article in the search results, whichever is the lowest. These numbers are calculated for the last 100 citations when articles are cited more than 100 times.