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Times Cited: 135
Times Cited: 135
Times Cited
Times Co-cited
Similarity
Ablation of P/Q-type Ca(2+) channel currents, altered synaptic transmission, and progressive ataxia in mice lacking the alpha(1A)-subunit.
K Jun, E S Piedras-Rentería, S M Smith, D B Wheeler, S B Lee, T G Lee, H Chin, M E Adams, R H Scheller, R W Tsien,[...]. Proc Natl Acad Sci U S A 1999
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Familial hemiplegic migraine and episodic ataxia type-2 are caused by mutations in the Ca2+ channel gene CACNL1A4.
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Absence epilepsy in tottering mutant mice is associated with calcium channel defects.
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Autosomal dominant cerebellar ataxia (SCA6) associated with small polyglutamine expansions in the alpha 1A-voltage-dependent calcium channel.
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A Cacna1a knockin migraine mouse model with increased susceptibility to cortical spreading depression.
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Immunochemical identification and subcellular distribution of the alpha 1A subunits of brain calcium channels.
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Single tottering mutations responsible for the neuropathic phenotype of the P-type calcium channel.
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Specific kinetic alterations of human CaV2.1 calcium channels produced by mutation S218L causing familial hemiplegic migraine and delayed cerebral edema and coma after minor head trauma.
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Functional consequences of mutations in the human alpha1A calcium channel subunit linked to familial hemiplegic migraine.
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Whole-cell and single-channel analysis of P-type calcium currents in cerebellar Purkinje cells of leaner mutant mice.
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Functional consequences of P/Q-type Ca2+ channel Cav2.1 missense mutations associated with episodic ataxia type 2 and progressive ataxia.
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Complete loss of P/Q calcium channel activity caused by a CACNA1A missense mutation carried by patients with episodic ataxia type 2.
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Familial hemiplegic migraine mutations change alpha1A Ca2+ channel kinetics.
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The clinical spectrum of familial hemiplegic migraine associated with mutations in a neuronal calcium channel.
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Ducky mouse phenotype of epilepsy and ataxia is associated with mutations in the Cacna2d2 gene and decreased calcium channel current in cerebellar Purkinje cells.
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Two novel alleles of tottering with distinct Ca(v)2.1 calcium channel neuropathologies.
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Co-cited is the co-citation frequency, indicating how many articles cite the article together with the query article. Similarity is the co-citation as percentage of the times cited of the query article or the article in the search results, whichever is the lowest. These numbers are calculated for the last 100 citations when articles are cited more than 100 times.