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Times Co-cited
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Association analyses identify 38 susceptibility loci for inflammatory bowel disease and highlight shared genetic risk across populations.
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Nod2 is a general sensor of peptidoglycan through muramyl dipeptide (MDP) detection.
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Genome-wide association study implicates immune activation of multiple integrin genes in inflammatory bowel disease.
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NOD2-mediated dysbiosis predisposes mice to transmissible colitis and colorectal cancer.
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ATG16L1 and NOD2 interact in an autophagy-dependent antibacterial pathway implicated in Crohn's disease pathogenesis.
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Faecalibacterium prausnitzii is an anti-inflammatory commensal bacterium identified by gut microbiota analysis of Crohn disease patients.
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The microbiome in inflammatory bowel disease: current status and the future ahead.
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Genome-wide association study identifies new susceptibility loci for Crohn disease and implicates autophagy in disease pathogenesis.
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Toward an integrated clinical, molecular and serological classification of inflammatory bowel disease: report of a Working Party of the 2005 Montreal World Congress of Gastroenterology.
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Paneth cells as a site of origin for intestinal inflammation.
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NOD2 (CARD15) mutations in Crohn's disease are associated with diminished mucosal alpha-defensin expression.
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The ubiquitin ligase XIAP recruits LUBAC for NOD2 signaling in inflammation and innate immunity.
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Genome-wide meta-analysis increases to 71 the number of confirmed Crohn's disease susceptibility loci.
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Virus-plus-susceptibility gene interaction determines Crohn's disease gene Atg16L1 phenotypes in intestine.
Ken Cadwell, Khushbu K Patel, Nicole S Maloney, Ta-Chiang Liu, Aylwin C Y Ng, Chad E Storer, Richard D Head, Ramnik Xavier, Thaddeus S Stappenbeck, Herbert W Virgin. Cell 2010
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Ustekinumab as Induction and Maintenance Therapy for Crohn's Disease.
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Deep resequencing of GWAS loci identifies independent rare variants associated with inflammatory bowel disease.
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Commensal microbe-derived butyrate induces the differentiation of colonic regulatory T cells.
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Koichi S Kobayashi, Mathias Chamaillard, Yasunori Ogura, Octavian Henegariu, Naohiro Inohara, Gabriel Nuñez, Richard A Flavell. Science 2005
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Inflammatory bowel disease and mutations affecting the interleukin-10 receptor.
Erik-Oliver Glocker, Daniel Kotlarz, Kaan Boztug, E Michael Gertz, Alejandro A Schäffer, Fatih Noyan, Mario Perro, Jana Diestelhorst, Anna Allroth, Dhaarini Murugan,[...]. N Engl J Med 2009
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Nod2 is required for the regulation of commensal microbiota in the intestine.
Tanja Petnicki-Ocwieja, Tomas Hrncir, Yuen-Joyce Liu, Amlan Biswas, Tomas Hudcovic, Helena Tlaskalova-Hogenova, Koichi S Kobayashi. Proc Natl Acad Sci U S A 2009
Tanja Petnicki-Ocwieja, Tomas Hrncir, Yuen-Joyce Liu, Amlan Biswas, Tomas Hudcovic, Helena Tlaskalova-Hogenova, Koichi S Kobayashi. Proc Natl Acad Sci U S A 2009
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Multi-omics of the gut microbial ecosystem in inflammatory bowel diseases.
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Single-Cell Analysis of Crohn's Disease Lesions Identifies a Pathogenic Cellular Module Associated with Resistance to Anti-TNF Therapy.
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RICK/Rip2/CARDIAK mediates signalling for receptors of the innate and adaptive immune systems.
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Endosomes are specialized platforms for bacterial sensing and NOD2 signalling.
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Co-cited is the co-citation frequency, indicating how many articles cite the article together with the query article. Similarity is the co-citation as percentage of the times cited of the query article or the article in the search results, whichever is the lowest. These numbers are calculated for the last 100 citations when articles are cited more than 100 times.