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HOXA genes are included in genetic and biologic networks defining human acute T-cell leukemia (T-ALL).
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Integrated transcript and genome analyses reveal NKX2-1 and MEF2C as potential oncogenes in T cell acute lymphoblastic leukemia.
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The molecular basis of T cell acute lymphoblastic leukemia.
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High frequency of PTEN, PI3K, and AKT abnormalities in T-cell acute lymphoblastic leukemia.
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Mutational loss of PTEN induces resistance to NOTCH1 inhibition in T-cell leukemia.
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c-Myc is an important direct target of Notch1 in T-cell acute lymphoblastic leukemia/lymphoma.
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Recurrent SPI1 (PU.1) fusions in high-risk pediatric T cell acute lymphoblastic leukemia.
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A NOTCH1-driven MYC enhancer promotes T cell development, transformation and acute lymphoblastic leukemia.
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Genome-wide analysis of genetic alterations in acute lymphoblastic leukaemia.
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PAX5-driven subtypes of B-progenitor acute lymphoblastic leukemia.
Zhaohui Gu, Michelle L Churchman, Kathryn G Roberts, Ian Moore, Xin Zhou, Joy Nakitandwe, Kohei Hagiwara, Stephane Pelletier, Sebastien Gingras, Hartmut Berns,[...]. Nat Genet 2019
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NOTCH1 and FBXW7 mutations have a favorable impact on early response to treatment, but not on outcome, in children with T-cell acute lymphoblastic leukemia (T-ALL) treated on EORTC trials 58881 and 58951.
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HOX11L2 expression defines a clinical subtype of pediatric T-ALL associated with poor prognosis.
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TLX homeodomain oncogenes mediate T cell maturation arrest in T-ALL via interaction with ETS1 and suppression of TCRα gene expression.
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PTEN microdeletions in T-cell acute lymphoblastic leukemia are caused by illegitimate RAG-mediated recombination events.
Rui D Mendes, Leonor M Sarmento, Kirsten Canté-Barrett, Linda Zuurbier, Jessica G C A M Buijs-Gladdines, Vanda Póvoa, Willem K Smits, Miguel Abecasis, J Andres Yunes, Edwin Sonneveld,[...]. Blood 2014
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Outcome for children and young people with Early T-cell precursor acute lymphoblastic leukaemia treated on a contemporary protocol, UKALL 2003.
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An early thymic precursor phenotype predicts outcome exclusively in HOXA-overexpressing adult T-cell acute lymphoblastic leukemia: a Group for Research in Adult Acute Lymphoblastic Leukemia study.
Jonathan Bond, Tony Marchand, Aurore Touzart, Agata Cieslak, Amélie Trinquand, Laurent Sutton, Isabelle Radford-Weiss, Ludovic Lhermitte, Salvatore Spicuglia, Hervé Dombret,[...]. Haematologica 2016
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Identification of fusion genes and characterization of transcriptome features in T-cell acute lymphoblastic leukemia.
Bing Chen, Lu Jiang, Meng-Ling Zhong, Jian-Feng Li, Ben-Shang Li, Li-Jun Peng, Yu-Ting Dai, Bo-Wen Cui, Tian-Qi Yan, Wei-Na Zhang,[...]. Proc Natl Acad Sci U S A 2018
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The BCL11B tumor suppressor is mutated across the major molecular subtypes of T-cell acute lymphoblastic leukemia.
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Targetable kinase-activating lesions in Ph-like acute lymphoblastic leukemia.
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The significance of PTEN and AKT aberrations in pediatric T-cell acute lymphoblastic leukemia.
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A new recurrent and specific cryptic translocation, t(5;14)(q35;q32), is associated with expression of the Hox11L2 gene in T acute lymphoblastic leukemia.
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NOTCH1 directly regulates c-MYC and activates a feed-forward-loop transcriptional network promoting leukemic cell growth.
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Exome sequencing identifies mutation in CNOT3 and ribosomal genes RPL5 and RPL10 in T-cell acute lymphoblastic leukemia.
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Co-cited is the co-citation frequency, indicating how many articles cite the article together with the query article. Similarity is the co-citation as percentage of the times cited of the query article or the article in the search results, whichever is the lowest. These numbers are calculated for the last 100 citations when articles are cited more than 100 times.