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Times Cited: 1080
Times Cited: 1080
Times Cited
Times Co-cited
Similarity
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TOX transcriptionally and epigenetically programs CD8+ T cell exhaustion.
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Subsets of exhausted CD8+ T cells differentially mediate tumor control and respond to checkpoint blockade.
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T Cell Factor 1-Expressing Memory-like CD8(+) T Cells Sustain the Immune Response to Chronic Viral Infections.
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TOX and TOX2 transcription factors cooperate with NR4A transcription factors to impose CD8+ T cell exhaustion.
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The transcription factor NFAT promotes exhaustion of activated CD8⁺ T cells.
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Tumor-Specific T Cell Dysfunction Is a Dynamic Antigen-Driven Differentiation Program Initiated Early during Tumorigenesis.
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PD-1 expression on HIV-specific T cells is associated with T-cell exhaustion and disease progression.
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Bioenergetic Insufficiencies Due to Metabolic Alterations Regulated by the Inhibitory Receptor PD-1 Are an Early Driver of CD8(+) T Cell Exhaustion.
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Viral antigen and extensive division maintain virus-specific CD8 T cells during chronic infection.
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Antigen-independent memory CD8 T cells do not develop during chronic viral infection.
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Co-cited is the co-citation frequency, indicating how many articles cite the article together with the query article. Similarity is the co-citation as percentage of the times cited of the query article or the article in the search results, whichever is the lowest. These numbers are calculated for the last 100 citations when articles are cited more than 100 times.