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Times Cited: 339
Times Cited: 339
Times Cited
Times Co-cited
Similarity
Altered properties of quantal neurotransmitter release at endplates of mice lacking P/Q-type Ca2+ channels.
Francisco J Urbano, Erika S Piedras-Rentería, Kisun Jun, Hee-Sup Shin, Osvaldo D Uchitel, Richard W Tsien. Proc Natl Acad Sci U S A 2003
Francisco J Urbano, Erika S Piedras-Rentería, Kisun Jun, Hee-Sup Shin, Osvaldo D Uchitel, Richard W Tsien. Proc Natl Acad Sci U S A 2003
25
Calcium channels coupled to neurotransmitter release at neonatal rat neuromuscular junctions.
M D Rosato Siri, O D Uchitel. J Physiol 1999
M D Rosato Siri, O D Uchitel. J Physiol 1999
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Ablation of P/Q-type Ca(2+) channel currents, altered synaptic transmission, and progressive ataxia in mice lacking the alpha(1A)-subunit.
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22
Familial hemiplegic migraine and episodic ataxia type-2 are caused by mutations in the Ca2+ channel gene CACNL1A4.
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Structure and regulation of voltage-gated Ca2+ channels.
W A Catterall. Annu Rev Cell Dev Biol 2000
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Autosomal dominant cerebellar ataxia (SCA6) associated with small polyglutamine expansions in the alpha 1A-voltage-dependent calcium channel.
O Zhuchenko, J Bailey, P Bonnen, T Ashizawa, D W Stockton, C Amos, W B Dobyns, S H Subramony, H Y Zoghbi, C C Lee. Nat Genet 1997
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Structure and function of neuronal Ca2+ channels and their role in neurotransmitter release.
W A Catterall. Cell Calcium 1998
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Loss-of-function EA2 mutations are associated with impaired neuromuscular transmission.
J Jen, J Wan, M Graves, H Yu, A F Mock, C J Coulin, G Kim, Q Yue, D M Papazian, R W Baloh. Neurology 2001
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Differential expression of alpha 1 and beta subunits of voltage dependent Ca2+ channel at the neuromuscular junction of normal and P/Q Ca2+ channel knockout mouse.
R Pagani, M Song, M McEnery, N Qin, R W Tsien, L Toro, E Stefani, O D Uchitel. Neuroscience 2004
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Transmitter release and presynaptic Ca2+ currents blocked by the spider toxin omega-Aga-IVA.
D A Protti, O D Uchitel. Neuroreport 1993
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A Cacna1a knockin migraine mouse model with increased susceptibility to cortical spreading depression.
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Dysfunction of the brain calcium channel CaV2.1 in absence epilepsy and episodic ataxia.
Paola Imbrici, Stephen L Jaffe, Louise H Eunson, Nicholas P Davies, Colin Herd, Robert Robertson, Dimitri M Kullmann, Michael G Hanna. Brain 2004
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Localization of P-type calcium channels in the central nervous system.
D Hillman, S Chen, T T Aung, B Cherksey, M Sugimori, R R Llinás. Proc Natl Acad Sci U S A 1991
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Differential localization of voltage-dependent calcium channel alpha1 subunits at the human and rat neuromuscular junction.
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Non-linear summation of end-plate potentials in the frog and mouse.
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Calcium channel types with distinct presynaptic localization couple differentially to transmitter release in single calyx-type synapses.
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Immunochemical identification and subcellular distribution of the alpha 1A subunits of brain calcium channels.
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Dystonia and cerebellar atrophy in Cacna1a null mice lacking P/Q calcium channel activity.
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Human epilepsy associated with dysfunction of the brain P/Q-type calcium channel.
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Localization of Ca2+ channel subtypes on rat spinal motor neurons, interneurons, and nerve terminals.
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Developmental changes in calcium channel types mediating synaptic transmission in rat auditory brainstem.
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Quantal components of end-plate potentials in the myasthenic syndrome.
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Calcium channel blockers and transmitter release at the normal human neuromuscular junction.
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Different types of calcium channels mediate central synaptic transmission.
T Takahashi, A Momiyama. Nature 1993
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11
Presynaptic Ca2+ channels compete for channel type-preferring slots in altered neurotransmission arising from Ca2+ channelopathy.
Yu-Qing Cao, Erika S Piedras-Rentería, Geoffrey B Smith, Gong Chen, Nobutoshi C Harata, Richard W Tsien. Neuron 2004
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Calcium control of transmitter release at a cerebellar synapse.
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Differential blockade of voltage-sensitive calcium channels at the mouse neuromuscular junction by novel omega-conopeptides and omega-agatoxin-IVA.
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Calcium channels involved in synaptic transmission at the mature and regenerating mouse neuromuscular junction.
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Nomenclature of voltage-gated calcium channels.
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Roles of N-type and Q-type Ca2+ channels in supporting hippocampal synaptic transmission.
D B Wheeler, A Randall, R W Tsien. Science 1994
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Abnormal transmitter release at neuromuscular junctions of mice carrying the tottering alpha(1A) Ca(2+) channel mutation.
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Adaptation of quantal content to decreased postsynaptic sensitivity at single endplates in alpha-bungarotoxin-treated rats.
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Primary structure and functional expression from complementary DNA of a brain calcium channel.
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Complete loss of P/Q calcium channel activity caused by a CACNA1A missense mutation carried by patients with episodic ataxia type 2.
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Absence epilepsy in tottering mutant mice is associated with calcium channel defects.
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Gene dosage-dependent transmitter release changes at neuromuscular synapses of CACNA1A R192Q knockin mice are non-progressive and do not lead to morphological changes or muscle weakness.
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Developmental changes in calcium channel types mediating central synaptic transmission.
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Passive transfer of Lambert-Eaton myasthenic syndrome with IgG from man to mouse depletes the presynaptic membrane active zones.
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Presynaptic Ca(2+) influx at a mouse central synapse with Ca(2+) channel subunit mutations.
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Splicing of alpha 1A subunit gene generates phenotypic variants of P- and Q-type calcium channels.
E Bourinet, T W Soong, K Sutton, S Slaymaker, E Mathews, A Monteil, G W Zamponi, J Nargeot, T P Snutch. Nat Neurosci 1999
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Function and dysfunction of synaptic calcium channels: insights from mouse models.
Daniela Pietrobon. Curr Opin Neurobiol 2005
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Gamma-band deficiency and abnormal thalamocortical activity in P/Q-type channel mutant mice.
Rodolfo R Llinás, Soonwook Choi, Francisco J Urbano, Hee-Sup Shin. Proc Natl Acad Sci U S A 2007
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A synaptic laminin-calcium channel interaction organizes active zones in motor nerve terminals.
Hiroshi Nishimune, Joshua R Sanes, Steven S Carlson. Nature 2004
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Inhibition of acetylcholine release from mouse motor nerve by a P-type calcium channel blocker, omega-agatoxin IVA.
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Coupling of L-type calcium channels to neurotransmitter release at mouse motor nerve terminals.
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Multiple types of calcium channels mediate transmitter release during functional recovery of botulinum toxin type A-poisoned mouse motor nerve terminals.
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Novel modulatory effect of L-type calcium channels at newly formed neuromuscular junctions.
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Calcium-channel antibodies in the Lambert-Eaton syndrome and other paraneoplastic syndromes.
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Biochemical properties and subcellular distribution of an N-type calcium channel alpha 1 subunit.
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8
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