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Times Cited: 860
Times Cited: 860
Times Cited
Times Co-cited
Similarity
Deep brain stimulation for treatment-resistant depression.
Helen S Mayberg, Andres M Lozano, Valerie Voon, Heather E McNeely, David Seminowicz, Clement Hamani, Jason M Schwalb, Sidney H Kennedy. Neuron 2005
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Resting-state functional connectivity in major depression: abnormally increased contributions from subgenual cingulate cortex and thalamus.
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Subgenual prefrontal cortex abnormalities in mood disorders.
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Regional metabolic effects of fluoxetine in major depression: serial changes and relationship to clinical response.
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Brain structural and functional abnormalities in mood disorders: implications for neurocircuitry models of depression.
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Changes in regional brain glucose metabolism measured with positron emission tomography after paroxetine treatment of major depression.
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The mesolimbic dopamine reward circuit in depression.
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Increased amygdala and decreased dorsolateral prefrontal BOLD responses in unipolar depression: related and independent features.
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Morphometric evidence for neuronal and glial prefrontal cell pathology in major depression.
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Chronic behavioral stress induces apical dendritic reorganization in pyramidal neurons of the medial prefrontal cortex.
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Can't shake that feeling: event-related fMRI assessment of sustained amygdala activity in response to emotional information in depressed individuals.
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mTOR-dependent synapse formation underlies the rapid antidepressant effects of NMDA antagonists.
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Neurobiology of emotion perception II: Implications for major psychiatric disorders.
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A default mode of brain function.
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Glial reduction in the subgenual prefrontal cortex in mood disorders.
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Acute and longer-term outcomes in depressed outpatients requiring one or several treatment steps: a STAR*D report.
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Essential role of BDNF in the mesolimbic dopamine pathway in social defeat stress.
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Antidepressant effect of optogenetic stimulation of the medial prefrontal cortex.
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Molecular adaptations underlying susceptibility and resistance to social defeat in brain reward regions.
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Rapid regulation of depression-related behaviours by control of midbrain dopamine neurons.
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Resting-state functional MRI in depression unmasks increased connectivity between networks via the dorsal nexus.
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A meta-analytic study of changes in brain activation in depression.
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Functional neuroimaging of major depressive disorder: a meta-analysis and new integration of base line activation and neural response data.
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Nucleus accumbens medium spiny neuron subtypes mediate depression-related outcomes to social defeat stress.
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Failure to regulate: counterproductive recruitment of top-down prefrontal-subcortical circuitry in major depression.
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Rethinking feelings: an FMRI study of the cognitive regulation of emotion.
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Reduced caudate and nucleus accumbens response to rewards in unmedicated individuals with major depressive disorder.
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Functional anatomical abnormalities in limbic and prefrontal cortical structures in major depression.
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A meta-analysis of neurofunctional imaging studies of emotion and cognition in major depression.
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Chronic stress alters dendritic morphology in rat medial prefrontal cortex.
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Meta-analysis of volumetric abnormalities in cortico-striatal-pallidal-thalamic circuits in major depressive disorder.
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Distinct regions of prefrontal cortex mediate resistance and vulnerability to depression.
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Lack of ventral striatal response to positive stimuli in depressed versus normal subjects.
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Frontal and parietal metabolic disturbances in unipolar depression.
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DeltaFosB in brain reward circuits mediates resilience to stress and antidepressant responses.
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A standardized protocol for repeated social defeat stress in mice.
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Co-cited is the co-citation frequency, indicating how many articles cite the article together with the query article. Similarity is the co-citation as percentage of the times cited of the query article or the article in the search results, whichever is the lowest. These numbers are calculated for the last 100 citations when articles are cited more than 100 times.