G V Rebec, S K Conroy, S J Barton. Neuroscience 2006
Times Cited: 67
Times Cited: 67
Times Cited
Times Co-cited
Similarity
61
Exon 1 of the HD gene with an expanded CAG repeat is sufficient to cause a progressive neurological phenotype in transgenic mice.
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Electrophysiological and morphological changes in striatal spiny neurons in R6/2 Huntington's disease transgenic mice.
G J Klapstein, R S Fisher, H Zanjani, C Cepeda, E S Jokel, M F Chesselet, M S Levine. J Neurophysiol 2001
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The corticostriatal pathway in Huntington's disease.
Carlos Cepeda, Nanping Wu, Véronique M André, Damian M Cummings, Michael S Levine. Prog Neurobiol 2007
Carlos Cepeda, Nanping Wu, Véronique M André, Damian M Cummings, Michael S Levine. Prog Neurobiol 2007
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Dysregulated information processing by medium spiny neurons in striatum of freely behaving mouse models of Huntington's disease.
Benjamin R Miller, Adam G Walker, Anand S Shah, Scott J Barton, George V Rebec. J Neurophysiol 2008
Benjamin R Miller, Adam G Walker, Anand S Shah, Scott J Barton, George V Rebec. J Neurophysiol 2008
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Altered information processing in the prefrontal cortex of Huntington's disease mouse models.
Adam G Walker, Benjamin R Miller, Jenna N Fritsch, Scott J Barton, George V Rebec. J Neurosci 2008
Adam G Walker, Benjamin R Miller, Jenna N Fritsch, Scott J Barton, George V Rebec. J Neurosci 2008
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Alterations in cortical excitation and inhibition in genetic mouse models of Huntington's disease.
Damian M Cummings, Véronique M André, Besim O Uzgil, Steven M Gee, Yvette E Fisher, Carlos Cepeda, Michael S Levine. J Neurosci 2009
Damian M Cummings, Véronique M André, Besim O Uzgil, Steven M Gee, Yvette E Fisher, Carlos Cepeda, Michael S Levine. J Neurosci 2009
37
Transient and progressive electrophysiological alterations in the corticostriatal pathway in a mouse model of Huntington's disease.
Carlos Cepeda, Raymond S Hurst, Christopher R Calvert, Elizabeth Hernández-Echeagaray, Oanh K Nguyen, Emily Jocoy, Lindsey J Christian, Marjorie A Ariano, Michael S Levine. J Neurosci 2003
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Differential loss of striatal projection neurons in Huntington disease.
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Age-dependent alterations of corticostriatal activity in the YAC128 mouse model of Huntington disease.
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Time course of early motor and neuropathological anomalies in a knock-in mouse model of Huntington's disease with 140 CAG repeats.
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Changes in cortical and striatal neurons predict behavioral and electrophysiological abnormalities in a transgenic murine model of Huntington's disease.
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Full-length human mutant huntingtin with a stable polyglutamine repeat can elicit progressive and selective neuropathogenesis in BACHD mice.
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Dysregulated Neuronal Activity Patterns Implicate Corticostriatal Circuit Dysfunction in Multiple Rodent Models of Huntington's Disease.
Benjamin R Miller, Adam G Walker, Scott J Barton, George V Rebec. Front Syst Neurosci 2011
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NMDA receptor function in mouse models of Huntington disease.
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Pathological cell-cell interactions elicited by a neuropathogenic form of mutant Huntingtin contribute to cortical pathogenesis in HD mice.
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Up-regulation of GLT1 expression increases glutamate uptake and attenuates the Huntington's disease phenotype in the R6/2 mouse.
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Impaired glutamate transport and glutamate-glutamine cycling: downstream effects of the Huntington mutation.
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Enhanced sensitivity to N-methyl-D-aspartate receptor activation in transgenic and knockin mouse models of Huntington's disease.
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Ascorbate treatment attenuates the Huntington behavioral phenotype in mice.
George V Rebec, Scott J Barton, Ann M Marseilles, Kristin Collins. Neuroreport 2003
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25
Dysregulation of ascorbate release in the striatum of behaving mice expressing the Huntington's disease gene.
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Pathophysiology of Huntington's disease: time-dependent alterations in synaptic and receptor function.
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Differential loss of striatal projection systems in Huntington's disease: a quantitative immunohistochemical study.
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Aberrant cortical synaptic plasticity and dopaminergic dysfunction in a mouse model of Huntington's disease.
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A YAC mouse model for Huntington's disease with full-length mutant huntingtin, cytoplasmic toxicity, and selective striatal neurodegeneration.
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Altered brain neurotransmitter receptors in transgenic mice expressing a portion of an abnormal human huntington disease gene.
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Differential susceptibility to excitotoxic stress in YAC128 mouse models of Huntington disease between initiation and progression of disease.
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Early increase in extrasynaptic NMDA receptor signaling and expression contributes to phenotype onset in Huntington's disease mice.
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Glutamate uptake is reduced in prefrontal cortex in Huntington's disease.
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Striatal potassium channel dysfunction in Huntington's disease transgenic mice.
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Decreased expression of striatal signaling genes in a mouse model of Huntington's disease.
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17
Co-cited is the co-citation frequency, indicating how many articles cite the article together with the query article. Similarity is the co-citation as percentage of the times cited of the query article or the article in the search results, whichever is the lowest. These numbers are calculated for the last 100 citations when articles are cited more than 100 times.