Yunfu Lin, Vincent Dion, John H Wilson. Nat Struct Mol Biol 2006
Times Cited: 122
Times Cited: 122
Times Cited
Times Co-cited
Similarity
Transcription-induced CAG repeat contraction in human cells is mediated in part by transcription-coupled nucleotide excision repair.
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Mechanisms of trinucleotide repeat instability during human development.
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Intergenerational and striatal CAG repeat instability in Huntington's disease knock-in mice involve different DNA repair genes.
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Transcription destabilizes triplet repeats.
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Convergent transcription through a long CAG tract destabilizes repeats and induces apoptosis.
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Slipped (CTG)*(CAG) repeats can be correctly repaired, escape repair or undergo error-prone repair.
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Determinants of R-loop formation at convergent bidirectionally transcribed trinucleotide repeats.
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Trinucleotide repeats that expand in human disease form hairpin structures in vitro.
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MSH2-dependent germinal CTG repeat expansions are produced continuously in spermatogonia from DM1 transgenic mice.
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Dnmt1 deficiency promotes CAG repeat expansion in the mouse germline.
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Transcription-coupled DNA repair: two decades of progress and surprises.
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CTCF cis-regulates trinucleotide repeat instability in an epigenetic manner: a novel basis for mutational hot spot determination.
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