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Times Cited: 791
Times Cited: 791
Times Cited
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Similarity
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Maria Manczak, Marcus J Calkins, P Hemachandra Reddy. Hum Mol Genet 2011
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Mitochondrial Abeta: a potential focal point for neuronal metabolic dysfunction in Alzheimer's disease.
Casper Caspersen, Ning Wang, Jun Yao, Alexander Sosunov, Xi Chen, Joyce W Lustbader, Hong Wei Xu, David Stern, Guy McKhann, Shi Du Yan. FASEB J 2005
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Impaired balance of mitochondrial fission and fusion in Alzheimer's disease.
Xinglong Wang, Bo Su, Hyoung-gon Lee, Xinyi Li, George Perry, Mark A Smith, Xiongwei Zhu. J Neurosci 2009
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Cyclophilin D deficiency attenuates mitochondrial and neuronal perturbation and ameliorates learning and memory in Alzheimer's disease.
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The amyloid beta-peptide is imported into mitochondria via the TOM import machinery and localized to mitochondrial cristae.
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Amyloid-beta overproduction causes abnormal mitochondrial dynamics via differential modulation of mitochondrial fission/fusion proteins.
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ABAD directly links Abeta to mitochondrial toxicity in Alzheimer's disease.
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The Alzheimer's disease mitochondrial cascade hypothesis: progress and perspectives.
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Impaired mitochondrial biogenesis, defective axonal transport of mitochondria, abnormal mitochondrial dynamics and synaptic degeneration in a mouse model of Alzheimer's disease.
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Oxidative stress and mitochondrial dysfunction in Alzheimer's disease.
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Mitochondrial bioenergetic deficit precedes Alzheimer's pathology in female mouse model of Alzheimer's disease.
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Differential expression of oxidative phosphorylation genes in patients with Alzheimer's disease: implications for early mitochondrial dysfunction and oxidative damage.
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Amyloid beta, mitochondrial structural and functional dynamics in Alzheimer's disease.
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Abnormal interaction between the mitochondrial fission protein Drp1 and hyperphosphorylated tau in Alzheimer's disease neurons: implications for mitochondrial dysfunction and neuronal damage.
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Mitochondria dysfunction in the pathogenesis of Alzheimer's disease: recent advances.
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Impaired mitochondrial calcium efflux contributes to disease progression in models of Alzheimer's disease.
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Mitophagy and Alzheimer's Disease: Cellular and Molecular Mechanisms.
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A key role for MAM in mediating mitochondrial dysfunction in Alzheimer disease.
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Gene expression profiles of transcripts in amyloid precursor protein transgenic mice: up-regulation of mitochondrial metabolism and apoptotic genes is an early cellular change in Alzheimer's disease.
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Impaired mitochondrial biogenesis contributes to mitochondrial dysfunction in Alzheimer's disease.
Baiyang Sheng, Xinglong Wang, Bo Su, Hyoung-gon Lee, Gemma Casadesus, George Perry, Xiongwei Zhu. J Neurochem 2012
Baiyang Sheng, Xinglong Wang, Bo Su, Hyoung-gon Lee, Gemma Casadesus, George Perry, Xiongwei Zhu. J Neurochem 2012
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Abnormal mitochondrial dynamics and synaptic degeneration as early events in Alzheimer's disease: implications to mitochondria-targeted antioxidant therapeutics.
P Hemachandra Reddy, Raghav Tripathi, Quang Troung, Karuna Tirumala, Tejaswini P Reddy, Vishwanath Anekonda, Ulziibat P Shirendeb, Marcus J Calkins, Arubala P Reddy, Peizhong Mao,[...]. Biochim Biophys Acta 2012
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Amyloid beta impairs mitochondrial anterograde transport and degenerates synapses in Alzheimer's disease neurons.
Marcus J Calkins, P Hemachandra Reddy. Biochim Biophys Acta 2011
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Beta-amyloid inhibits integrated mitochondrial respiration and key enzyme activities.
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Modulation of the endoplasmic reticulum-mitochondria interface in Alzheimer's disease and related models.
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Co-cited is the co-citation frequency, indicating how many articles cite the article together with the query article. Similarity is the co-citation as percentage of the times cited of the query article or the article in the search results, whichever is the lowest. These numbers are calculated for the last 100 citations when articles are cited more than 100 times.