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Times Cited: 162
Times Cited: 162
Times Cited
Times Co-cited
Similarity
Mutation of the calcium channel gene Cacna1f disrupts calcium signaling, synaptic transmission and cellular organization in mouse retina.
Fiona Mansergh, Noelle C Orton, John P Vessey, Melanie R Lalonde, William K Stell, Francois Tremblay, Steven Barnes, Derrick E Rancourt, N Torben Bech-Hansen. Hum Mol Genet 2005
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Essential role of Ca2+-binding protein 4, a Cav1.4 channel regulator, in photoreceptor synaptic function.
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The presynaptic active zone protein bassoon is essential for photoreceptor ribbon synapse formation in the retina.
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An L-type calcium-channel gene mutated in incomplete X-linked congenital stationary night blindness.
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Dysregulation of Ca(v)1.4 channels disrupts the maturation of photoreceptor synaptic ribbons in congenital stationary night blindness type 2.
Xiaoni Liu, Vasily Kerov, Françoise Haeseleer, Anurima Majumder, Nikolai Artemyev, Sheila A Baker, Amy Lee. Channels (Austin) 2013
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Effects of presynaptic mutations on a postsynaptic Cacna1s calcium channel colocalized with mGluR6 at mouse photoreceptor ribbon synapses.
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Structural and functional abnormalities of retinal ribbon synapses due to Cacna2d4 mutation.
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Hanna Regus-Leidig, Jenny Atorf, Andreas Feigenspan, Jan Kremers, Marion A Maw, Johann Helmut Brandstätter. PLoS One 2014
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Mutation in the auxiliary calcium-channel subunit CACNA2D4 causes autosomal recessive cone dystrophy.
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A CACNA1F mutation identified in an X-linked retinal disorder shifts the voltage dependence of Cav1.4 channel activation.
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The CACNA1F gene encodes an L-type calcium channel with unique biophysical properties and tissue distribution.
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Immunocytochemical localization of the postsynaptic density protein PSD-95 in the mammalian retina.
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Pikachurin, a dystroglycan ligand, is essential for photoreceptor ribbon synapse formation.
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Mutations in NYX, encoding the leucine-rich proteoglycan nyctalopin, cause X-linked complete congenital stationary night blindness.
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The diverse roles of ribbon synapses in sensory neurotransmission.
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Clinical manifestations of a unique X-linked retinal disorder in a large New Zealand family with a novel mutation in CACNA1F, the gene responsible for CSNB2.
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Cone contacts, mosaics, and territories of bipolar cells in the mouse retina.
Heinz Wässle, Christian Puller, Frank Müller, Silke Haverkamp. J Neurosci 2009
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Generation, identification and functional characterization of the nob4 mutation of Grm6 in the mouse.
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TRPM1 is a component of the retinal ON bipolar cell transduction channel in the mGluR6 cascade.
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RIBEYE, a component of synaptic ribbons: a protein's journey through evolution provides insight into synaptic ribbon function.
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Deletion of the presynaptic scaffold CAST reduces active zone size in rod photoreceptors and impairs visual processing.
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Guidance-cue control of horizontal cell morphology, lamination, and synapse formation in the mammalian outer retina.
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X linked cone-rod dystrophy, CORDX3, is caused by a mutation in the CACNA1F gene.
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Congenital stationary night blindness: an analysis and update of genotype-phenotype correlations and pathogenic mechanisms.
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TRPM1 is required for the depolarizing light response in retinal ON-bipolar cells.
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Dendrites of rod bipolar cells sprout in normal aging retina.
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Mutations in CABP4, the gene encoding the Ca2+-binding protein 4, cause autosomal recessive night blindness.
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12
GPR179 is required for depolarizing bipolar cell function and is mutated in autosomal-recessive complete congenital stationary night blindness.
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C-terminal modulator controls Ca2+-dependent gating of Ca(v)1.4 L-type Ca2+ channels.
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Localization of the alpha(1F) calcium channel subunit in the rat retina.
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Co-cited is the co-citation frequency, indicating how many articles cite the article together with the query article. Similarity is the co-citation as percentage of the times cited of the query article or the article in the search results, whichever is the lowest. These numbers are calculated for the last 100 citations when articles are cited more than 100 times.