A citation-based method for searching scientific literature

Reha M Toydemir, Anna E Brassington, Pinar Bayrak-Toydemir, Patrycja A Krakowiak, Lynn B Jorde, Frank G Whitby, Nicola Longo, David H Viskochil, John C Carey, Michael J Bamshad. Am J Hum Genet 2006
Times Cited: 86







List of co-cited articles
1272 articles co-cited >1



Times Cited
  Times     Co-cited
Similarity


Skeletal overgrowth and deafness in mice lacking fibroblast growth factor receptor 3.
J S Colvin, B A Bohne, G W Harding, D G McEwen, D M Ornitz. Nat Genet 1996
688
43

Fibroblast growth factor receptor 3 is a negative regulator of bone growth.
C Deng, A Wynshaw-Boris, F Zhou, A Kuo, P Leder. Cell 1996
835
38

Mutations in the transmembrane domain of FGFR3 cause the most common genetic form of dwarfism, achondroplasia.
R Shiang, L M Thompson, Y Z Zhu, D M Church, T J Fielder, M Bocian, S T Winokur, J J Wasmuth. Cell 1994
919
31

Mutations in the gene encoding fibroblast growth factor receptor-3 in achondroplasia.
F Rousseau, J Bonaventure, L Legeai-Mallet, A Pelet, J M Rozet, P Maroteaux, M Le Merrer, A Munnich. Nature 1994
649
27

A novel homozygous mutation in FGFR3 causes tall stature, severe lateral tibial deviation, scoliosis, hearing impairment, camptodactyly, and arachnodactyly.
Periklis Makrythanasis, Samia Temtamy, Mona S Aglan, Ghada A Otaify, Hanan Hamamy, Stylianos E Antonarakis. Hum Mutat 2014
38
63

Thanatophoric dysplasia (types I and II) caused by distinct mutations in fibroblast growth factor receptor 3.
P L Tavormina, R Shiang, L M Thompson, Y Z Zhu, D J Wilkin, R S Lachman, W R Wilcox, D L Rimoin, D H Cohn, J J Wasmuth. Nat Genet 1995
478
26


Overexpression of CNP in chondrocytes rescues achondroplasia through a MAPK-dependent pathway.
Akihiro Yasoda, Yasato Komatsu, Hideki Chusho, Takashi Miyazawa, Ami Ozasa, Masako Miura, Tatsuya Kurihara, Tomohiro Rogi, Shoji Tanaka, Michio Suda,[...]. Nat Med 2004
262
24

Gly369Cys mutation in mouse FGFR3 causes achondroplasia by affecting both chondrogenesis and osteogenesis.
L Chen, R Adar, X Yang, E O Monsonego, C Li, P V Hauschka, A Yayon, C X Deng. J Clin Invest 1999
189
24


A unique point mutation in the fibroblast growth factor receptor 3 gene (FGFR3) defines a new craniosynostosis syndrome.
M Muenke, K W Gripp, D M McDonald-McGinn, K Gaudenz, L A Whitaker, S P Bartlett, R I Markowitz, N H Robin, N Nwokoro, J J Mulvihill,[...]. Am J Hum Genet 1997
288
23


Defective bone mineralization and osteopenia in young adult FGFR3-/- mice.
Gladys Valverde-Franco, Hanlong Liu, David Davidson, Sen Chai, Hector Valderrama-Carvajal, David Goltzman, David M Ornitz, Janet E Henderson. Hum Mol Genet 2004
97
20


A neonatal lethal mutation in FGFR3 uncouples proliferation and differentiation of growth plate chondrocytes in embryos.
T Iwata, L Chen, C Li, D A Ovchinnikov, R R Behringer, C A Francomano, C X Deng. Hum Mol Genet 2000
157
19

Achondroplasia.
William A Horton, Judith G Hall, Jacqueline T Hecht. Lancet 2007
276
19


Overexpression of the C-type natriuretic peptide (CNP) is associated with overgrowth and bone anomalies in an individual with balanced t(2;7) translocation.
Renata Bocciardi, Roberto Giorda, Jens Buttgereit, Stefania Gimelli, Maria Teresa Divizia, Silvana Beri, Silvio Garofalo, Sara Tavella, Margherita Lerone, Orsetta Zuffardi,[...]. Hum Mutat 2007
90
18


Constitutive activation of MEK1 in chondrocytes causes Stat1-independent achondroplasia-like dwarfism and rescues the Fgfr3-deficient mouse phenotype.
Shunichi Murakami, Gener Balmes, Sandra McKinney, Zhaoping Zhang, David Givol, Benoit de Crombrugghe. Genes Dev 2004
199
17

Coordination of chondrogenesis and osteogenesis by fibroblast growth factor 18.
Zhonghao Liu, Jingsong Xu, Jennifer S Colvin, David M Ornitz. Genes Dev 2002
338
16

Mutations in different components of FGF signaling in LADD syndrome.
Edyta Rohmann, Han G Brunner, Hülya Kayserili, Oya Uyguner, Gudrun Nürnberg, Erin D Lew, Angus Dobbie, Veraragavan P Eswarakumar, Abdullah Uzumcu, Melike Ulubil-Emeroglu,[...]. Nat Genet 2006
128
16

Gain-of-function mutation in FGFR3 in mice leads to decreased bone mass by affecting both osteoblastogenesis and osteoclastogenesis.
Nan Su, Qidi Sun, Can Li, Xiumin Lu, Huabing Qi, Siyu Chen, Jing Yang, Xiaolan Du, Ling Zhao, Qifen He,[...]. Hum Mol Genet 2010
64
21

Evaluation of the therapeutic potential of a CNP analog in a Fgfr3 mouse model recapitulating achondroplasia.
Florence Lorget, Nabil Kaci, Jeff Peng, Catherine Benoist-Lasselin, Emilie Mugniery, Todd Oppeneer, Dan J Wendt, Sean M Bell, Sherry Bullens, Stuart Bunting,[...]. Am J Hum Genet 2012
102
16


Skeletal overgrowth is mediated by deficiency in a specific isoform of fibroblast growth factor receptor 3.
Veraragavan P Eswarakumar, Joseph Schlessinger. Proc Natl Acad Sci U S A 2007
39
33

A novel skeletal dysplasia with developmental delay and acanthosis nigricans is caused by a Lys650Met mutation in the fibroblast growth factor receptor 3 gene.
P L Tavormina, G A Bellus, M K Webster, M J Bamshad, A E Fraley, I McIntosh, J Szabo, W Jiang, E W Jabs, W R Wilcox,[...]. Am J Hum Genet 1999
116
15

A cluster of translocation breakpoints in 2q37 is associated with overexpression of NPPC in patients with a similar overgrowth phenotype.
Anne Moncla, Chantal Missirian, Pierre Cacciagli, Eve Balzamo, Laurence Legeai-Mallet, Jean-Luc Jouve, Brigitte Chabrol, Martine Le Merrer, Ghislaine Plessis, Laurent Villard,[...]. Hum Mutat 2007
61
21

Mutations in the transmembrane natriuretic peptide receptor NPR-B impair skeletal growth and cause acromesomelic dysplasia, type Maroteaux.
Cynthia F Bartels, Hulya Bükülmez, Pius Padayatti, David K Rhee, Conny van Ravenswaaij-Arts, Richard M Pauli, Stefan Mundlos, David Chitayat, Ling-Yu Shih, Lihadh I Al-Gazali,[...]. Am J Hum Genet 2004
227
15

A single-base change in the tyrosine kinase II domain of ovine FGFR3 causes hereditary chondrodysplasia in sheep.
J E Beever, M A Smit, S N Meyers, T S Hadfield, C Bottema, J Albretsen, N E Cockett. Anim Genet 2006
37
32

FGF signaling inhibits chondrocyte proliferation and regulates bone development through the STAT-1 pathway.
M Sahni, D C Ambrosetti, A Mansukhani, R Gertner, D Levy, C Basilico. Genes Dev 1999
280
13

A network of transcriptional and signaling events is activated by FGF to induce chondrocyte growth arrest and differentiation.
Lisa Dailey, Emmanuel Laplantine, Riccardo Priore, Claudio Basilico. J Cell Biol 2003
120
13

Mutations that cause osteoglophonic dysplasia define novel roles for FGFR1 in bone elongation.
Kenneth E White, Jose M Cabral, Siobhan I Davis, Tonya Fishburn, Wayne E Evans, Shoji Ichikawa, Joanna Fields, Xijie Yu, Nick J Shaw, Neil J McLellan,[...]. Am J Hum Genet 2005
196
13

FGF signaling in the developing endochondral skeleton.
David M Ornitz. Cytokine Growth Factor Rev 2005
242
13


FGFR3 promotes synchondrosis closure and fusion of ossification centers through the MAPK pathway.
Takehiko Matsushita, William R Wilcox, Yuk Yu Chan, Aya Kawanami, Hülya Bükülmez, Gener Balmes, Pavel Krejci, Pertchoui B Mekikian, Kazuyuki Otani, Isakichi Yamaura,[...]. Hum Mol Genet 2009
73
16

FGF18 is required for normal cell proliferation and differentiation during osteogenesis and chondrogenesis.
Norihiko Ohbayashi, Masaki Shibayama, Yoko Kurotaki, Mayumi Imanishi, Toshihiko Fujimori, Nobuyuki Itoh, Shinji Takada. Genes Dev 2002
327
12

Activation of Stat1 by mutant fibroblast growth-factor receptor in thanatophoric dysplasia type II dwarfism.
W C Su, M Kitagawa, N Xue, B Xie, S Garofalo, J Cho, C Deng, W A Horton, X Y Fu. Nature 1997
251
12

Unique expression pattern of the FGF receptor 3 gene during mouse organogenesis.
K Peters, D Ornitz, S Werner, L Williams. Dev Biol 1993
409
12

Conditional inactivation of FGF receptor 2 reveals an essential role for FGF signaling in the regulation of osteoblast function and bone growth.
Kai Yu, Jingsong Xu, Zhonghao Liu, Drazen Sosic, Jiansu Shao, Eric N Olson, Dwight A Towler, David M Ornitz. Development 2003
453
12



Dwarfism and early death in mice lacking C-type natriuretic peptide.
H Chusho, N Tamura, Y Ogawa, A Yasoda, M Suda, T Miyazawa, K Nakamura, K Nakao, T Kurihara, Y Komatsu,[...]. Proc Natl Acad Sci U S A 2001
328
12

Interaction of fibroblast growth factor and C-natriuretic peptide signaling in regulation of chondrocyte proliferation and extracellular matrix homeostasis.
Pavel Krejci, Bernard Masri, Vincent Fontaine, Pertchoui B Mekikian, Maryann Weis, Herve Prats, William R Wilcox. J Cell Sci 2005
104
12

Postnatal soluble FGFR3 therapy rescues achondroplasia symptoms and restores bone growth in mice.
Stéphanie Garcia, Béatrice Dirat, Thomas Tognacci, Nathalie Rochet, Xavier Mouska, Stéphanie Bonnafous, Stéphanie Patouraux, Albert Tran, Philippe Gual, Yannick Le Marchand-Brustel,[...]. Sci Transl Med 2013
57
19

Spatio-temporal expression of FGFR 1, 2 and 3 genes during human embryo-fetal ossification.
A L Delezoide, C Benoist-Lasselin, L Legeai-Mallet, M Le Merrer, A Munnich, M Vekemans, J Bonaventure. Mech Dev 1998
136
11

Disruption of the fibroblast growth factor-2 gene results in decreased bone mass and bone formation.
A Montero, Y Okada, M Tomita, M Ito, H Tsurukami, T Nakamura, T Doetschman, J D Coffin, M M Hurley. J Clin Invest 2000
341
11


A mouse model for achondroplasia produced by targeting fibroblast growth factor receptor 3.
Y Wang, M K Spatz, K Kannan, H Hayk, A Avivi, M Gorivodsky, M Pines, A Yayon, P Lonai, D Givol. Proc Natl Acad Sci U S A 1999
167
11



Co-cited is the co-citation frequency, indicating how many articles cite the article together with the query article. Similarity is the co-citation as percentage of the times cited of the query article or the article in the search results, whichever is the lowest. These numbers are calculated for the last 100 citations when articles are cited more than 100 times.