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Times Cited: 147
Times Cited
Times Co-cited
Similarity
Astrocytes expressing ALS-linked mutated SOD1 release factors selectively toxic to motor neurons.
Makiko Nagai, Diane B Re, Tetsuya Nagata, Alcmène Chalazonitis, Thomas M Jessell, Hynek Wichterle, Serge Przedborski. Nat Neurosci 2007
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Astrocytes as determinants of disease progression in inherited amyotrophic lateral sclerosis.
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Selective loss of glial glutamate transporter GLT-1 in amyotrophic lateral sclerosis.
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Focal loss of the glutamate transporter EAAT2 in a transgenic rat model of SOD1 mutant-mediated amyotrophic lateral sclerosis (ALS).
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Onset and progression in inherited ALS determined by motor neurons and microglia.
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Astrocytes from familial and sporadic ALS patients are toxic to motor neurons.
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Non-cell autonomous effect of glia on motor neurons in an embryonic stem cell-based ALS model.
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Mutations in Cu/Zn superoxide dismutase gene are associated with familial amyotrophic lateral sclerosis.
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Expanded GGGGCC hexanucleotide repeat in noncoding region of C9ORF72 causes chromosome 9p-linked FTD and ALS.
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A hexanucleotide repeat expansion in C9ORF72 is the cause of chromosome 9p21-linked ALS-FTD.
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Wild-type nonneuronal cells extend survival of SOD1 mutant motor neurons in ALS mice.
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Ubiquitinated TDP-43 in frontotemporal lobar degeneration and amyotrophic lateral sclerosis.
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ALS-linked SOD1 mutant G85R mediates damage to astrocytes and promotes rapidly progressive disease with SOD1-containing inclusions.
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Focal transplantation-based astrocyte replacement is neuroprotective in a model of motor neuron disease.
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Astrocyte loss of mutant SOD1 delays ALS disease onset and progression in G85R transgenic mice.
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Mutations in the FUS/TLS gene on chromosome 16 cause familial amyotrophic lateral sclerosis.
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Non-cell autonomous toxicity in neurodegenerative disorders: ALS and beyond.
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Focal degeneration of astrocytes in amyotrophic lateral sclerosis.
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Wild-type microglia extend survival in PU.1 knockout mice with familial amyotrophic lateral sclerosis.
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Non-cell-autonomous effect of human SOD1 G37R astrocytes on motor neurons derived from human embryonic stem cells.
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Mutations in FUS, an RNA processing protein, cause familial amyotrophic lateral sclerosis type 6.
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Dysregulation of astrocyte-motoneuron cross-talk in mutant superoxide dismutase 1-related amyotrophic lateral sclerosis.
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Mutations in UBQLN2 cause dominant X-linked juvenile and adult-onset ALS and ALS/dementia.
Han-Xiang Deng, Wenjie Chen, Seong-Tshool Hong, Kym M Boycott, George H Gorrie, Nailah Siddique, Yi Yang, Faisal Fecto, Yong Shi, Hong Zhai,[...]. Nature 2011
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Neurotoxic reactive astrocytes are induced by activated microglia.
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Human embryonic stem cell-derived motor neurons are sensitive to the toxic effect of glial cells carrying an ALS-causing mutation.
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TDP-43 mutations in familial and sporadic amyotrophic lateral sclerosis.
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ALS: a disease of motor neurons and their nonneuronal neighbors.
Séverine Boillée, Christine Vande Velde, Don W Cleveland. Neuron 2006
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Decreased glutamate transport by the brain and spinal cord in amyotrophic lateral sclerosis.
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The BH4 domain of Bcl-X(L) rescues astrocyte degeneration in amyotrophic lateral sclerosis by modulating intracellular calcium signals.
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Expression of ALS-linked TDP-43 mutant in astrocytes causes non-cell-autonomous motor neuron death in rats.
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Mutations of optineurin in amyotrophic lateral sclerosis.
Hirofumi Maruyama, Hiroyuki Morino, Hidefumi Ito, Yuishin Izumi, Hidemasa Kato, Yasuhito Watanabe, Yoshimi Kinoshita, Masaki Kamada, Hiroyuki Nodera, Hidenori Suzuki,[...]. Nature 2010
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Amyotrophic lateral sclerosis.
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Mutations in the profilin 1 gene cause familial amyotrophic lateral sclerosis.
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The role of excitotoxicity in the pathogenesis of amyotrophic lateral sclerosis.
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Nrf2 activation in astrocytes protects against neurodegeneration in mouse models of familial amyotrophic lateral sclerosis.
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Increased expression of the glial glutamate transporter EAAT2 modulates excitotoxicity and delays the onset but not the outcome of ALS in mice.
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Beta-lactam antibiotics offer neuroprotection by increasing glutamate transporter expression.
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Restricted expression of G86R Cu/Zn superoxide dismutase in astrocytes results in astrocytosis but does not cause motoneuron degeneration.
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Mutant SOD1 in cell types other than motor neurons and oligodendrocytes accelerates onset of disease in ALS mice.
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Astrocyte pathology and the absence of non-cell autonomy in an induced pluripotent stem cell model of TDP-43 proteinopathy.
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State of play in amyotrophic lateral sclerosis genetics.
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Direct conversion of patient fibroblasts demonstrates non-cell autonomous toxicity of astrocytes to motor neurons in familial and sporadic ALS.
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Phenotypically aberrant astrocytes that promote motoneuron damage in a model of inherited amyotrophic lateral sclerosis.
Pablo Díaz-Amarilla, Silvia Olivera-Bravo, Emiliano Trias, Andrea Cragnolini, Laura Martínez-Palma, Patricia Cassina, Joseph Beckman, Luis Barbeito. Proc Natl Acad Sci U S A 2011
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Neuroinflammation in amyotrophic lateral sclerosis: role of glial activation in motor neuron disease.
Thomas Philips, Wim Robberecht. Lancet Neurol 2011
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Necroptosis drives motor neuron death in models of both sporadic and familial ALS.
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Astrocyte-derived TGF-β1 accelerates disease progression in ALS mice by interfering with the neuroprotective functions of microglia and T cells.
Fumito Endo, Okiru Komine, Noriko Fujimori-Tonou, Masahisa Katsuno, Shijie Jin, Seiji Watanabe, Gen Sobue, Mari Dezawa, Tony Wyss-Coray, Koji Yamanaka. Cell Rep 2015
Fumito Endo, Okiru Komine, Noriko Fujimori-Tonou, Masahisa Katsuno, Shijie Jin, Seiji Watanabe, Gen Sobue, Mari Dezawa, Tony Wyss-Coray, Koji Yamanaka. Cell Rep 2015
11
Co-cited is the co-citation frequency, indicating how many articles cite the article together with the query article. Similarity is the co-citation as percentage of the times cited of the query article or the article in the search results, whichever is the lowest. These numbers are calculated for the last 100 citations when articles are cited more than 100 times.