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Times Cited: 790
Times Cited: 790
Times Cited
Times Co-cited
Similarity
Onset and progression in inherited ALS determined by motor neurons and microglia.
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Astrocytes from familial and sporadic ALS patients are toxic to motor neurons.
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Astrocytes expressing ALS-linked mutated SOD1 release factors selectively toxic to motor neurons.
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Mutations in Cu/Zn superoxide dismutase gene are associated with familial amyotrophic lateral sclerosis.
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Neurotoxic reactive astrocytes are induced by activated microglia.
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Direct conversion of patient fibroblasts demonstrates non-cell autonomous toxicity of astrocytes to motor neurons in familial and sporadic ALS.
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Expanded GGGGCC hexanucleotide repeat in noncoding region of C9ORF72 causes chromosome 9p-linked FTD and ALS.
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A hexanucleotide repeat expansion in C9ORF72 is the cause of chromosome 9p21-linked ALS-FTD.
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Wild-type nonneuronal cells extend survival of SOD1 mutant motor neurons in ALS mice.
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Kevin A Guttenplan, Maya K Weigel, Drew I Adler, Julien Couthouis, Shane A Liddelow, Aaron D Gitler, Ben A Barres. Nat Commun 2020
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C9orf72 is required for proper macrophage and microglial function in mice.
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Human embryonic stem cell-derived motor neurons are sensitive to the toxic effect of glial cells carrying an ALS-causing mutation.
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Astrocyte loss of mutant SOD1 delays ALS disease onset and progression in G85R transgenic mice.
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Sporadic ALS Astrocytes Induce Neuronal Degeneration In Vivo.
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Human iPSC-derived astrocytes from ALS patients with mutated C9ORF72 show increased oxidative stress and neurotoxicity.
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Necroptosis drives motor neuron death in models of both sporadic and familial ALS.
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Progressive Motor Neuron Pathology and the Role of Astrocytes in a Human Stem Cell Model of VCP-Related ALS.
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13
Focal loss of the glutamate transporter EAAT2 in a transgenic rat model of SOD1 mutant-mediated amyotrophic lateral sclerosis (ALS).
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Oligodendrocytes contribute to motor neuron death in ALS via SOD1-dependent mechanism.
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Micro-RNAs secreted through astrocyte-derived extracellular vesicles cause neuronal network degeneration in C9orf72 ALS.
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A neurodegeneration-specific gene-expression signature of acutely isolated microglia from an amyotrophic lateral sclerosis mouse model.
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Amyotrophic lateral sclerosis patient iPSC-derived astrocytes impair autophagy via non-cell autonomous mechanisms.
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ALS-linked SOD1 mutant G85R mediates damage to astrocytes and promotes rapidly progressive disease with SOD1-containing inclusions.
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Reactive Astrocytes: Production, Function, and Therapeutic Potential.
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Astrocytes expressing ALS-linked mutant FUS induce motor neuron death through release of tumor necrosis factor-alpha.
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Expression of ALS-linked TDP-43 mutant in astrocytes causes non-cell-autonomous motor neuron death in rats.
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Focal transplantation-based astrocyte replacement is neuroprotective in a model of motor neuron disease.
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Oligodendrocyte dysfunction in the pathogenesis of amyotrophic lateral sclerosis.
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Phenotypically aberrant astrocytes that promote motoneuron damage in a model of inherited amyotrophic lateral sclerosis.
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Co-cited is the co-citation frequency, indicating how many articles cite the article together with the query article. Similarity is the co-citation as percentage of the times cited of the query article or the article in the search results, whichever is the lowest. These numbers are calculated for the last 100 citations when articles are cited more than 100 times.