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Times Cited: 50
Times Cited: 50
Times Cited
Times Co-cited
Similarity
Specific deficit of the ON response in visual transmission by targeted disruption of the mGluR6 gene.
M Masu, H Iwakabe, Y Tagawa, T Miyoshi, M Yamashita, Y Fukuda, H Sasaki, K Hiroi, Y Nakamura, R Shigemoto. Cell 1995
M Masu, H Iwakabe, Y Tagawa, T Miyoshi, M Yamashita, Y Fukuda, H Sasaki, K Hiroi, Y Nakamura, R Shigemoto. Cell 1995
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TRPM1 is required for the depolarizing light response in retinal ON-bipolar cells.
Catherine W Morgans, Jianmei Zhang, Brett G Jeffrey, Steve M Nelson, Neal S Burke, Robert M Duvoisin, R Lane Brown. Proc Natl Acad Sci U S A 2009
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GPR179 is required for depolarizing bipolar cell function and is mutated in autosomal-recessive complete congenital stationary night blindness.
Neal S Peachey, Thomas A Ray, Ralph Florijn, Lucy B Rowe, Trijntje Sjoerdsma, Susana Contreras-Alcantara, Kenkichi Baba, Gianluca Tosini, Nikita Pozdeyev, P Michael Iuvone,[...]. Am J Hum Genet 2012
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Night blindness and abnormal cone electroretinogram ON responses in patients with mutations in the GRM6 gene encoding mGluR6.
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A transient receptor potential-like channel mediates synaptic transmission in rod bipolar cells.
Yin Shen, J Alexander Heimel, Maarten Kamermans, Neal S Peachey, Ronald G Gregg, Scott Nawy. J Neurosci 2009
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Mutations in GRM6 cause autosomal recessive congenital stationary night blindness with a distinctive scotopic 15-Hz flicker electroretinogram.
Christina Zeitz, Maria van Genderen, John Neidhardt, Ulrich F O Luhmann, Frank Hoeben, Ursula Forster, Katharina Wycisk, Gábor Mátyás, Carel B Hoyng, Frans Riemslag,[...]. Invest Ophthalmol Vis Sci 2005
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Generation, identification and functional characterization of the nob4 mutation of Grm6 in the mouse.
Lawrence H Pinto, Martha H Vitaterna, Kazuhiro Shimomura, Sandra M Siepka, Victoria Balannik, Erin L McDearmon, Chiaki Omura, Stephen Lumayag, Brandon M Invergo, Brett Glawe,[...]. Vis Neurosci 2007
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TRPM1 is a component of the retinal ON bipolar cell transduction channel in the mGluR6 cascade.
Chieko Koike, Takehisa Obara, Yoshitsugu Uriu, Tomohiro Numata, Rikako Sanuki, Kentarou Miyata, Toshiyuki Koyasu, Shinji Ueno, Kazuo Funabiki, Akiko Tani,[...]. Proc Natl Acad Sci U S A 2010
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A naturally occurring mouse model of X-linked congenital stationary night blindness.
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Nyctalopin expression in retinal bipolar cells restores visual function in a mouse model of complete X-linked congenital stationary night blindness.
Ronald G Gregg, Maarten Kamermans, Jan Klooster, Peter D Lukasiewicz, Neal S Peachey, Kirstan A Vessey, Maureen A McCall. J Neurophysiol 2007
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A role for nyctalopin, a small leucine-rich repeat protein, in localizing the TRP melastatin 1 channel to retinal depolarizing bipolar cell dendrites.
Jillian N Pearring, Pasano Bojang, Yin Shen, Chieko Koike, Takahisa Furukawa, Scott Nawy, Ronald G Gregg. J Neurosci 2011
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Mutations in NYX, encoding the leucine-rich proteoglycan nyctalopin, cause X-linked complete congenital stationary night blindness.
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Mutations in TRPM1 are a common cause of complete congenital stationary night blindness.
Maria M van Genderen, Mieke M C Bijveld, Yvonne B Claassen, Ralph J Florijn, Jillian N Pearring, Francoise M Meire, Maureen A McCall, Frans C C Riemslag, Ronald G Gregg, Arthur A B Bergen,[...]. Am J Hum Genet 2009
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TRPM1 is mutated in patients with autosomal-recessive complete congenital stationary night blindness.
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TRPM1 forms complexes with nyctalopin in vivo and accumulates in postsynaptic compartment of ON-bipolar neurons in mGluR6-dependent manner.
Yan Cao, Ekaterina Posokhova, Kirill A Martemyanov. J Neurosci 2011
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Gbeta5 is required for normal light responses and morphology of retinal ON-bipolar cells.
Anjali Rao, Rebecca Dallman, Scott Henderson, Ching-Kang Chen. J Neurosci 2007
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The light response of ON bipolar neurons requires G[alpha]o.
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The nob2 mouse, a null mutation in Cacna1f: anatomical and functional abnormalities in the outer retina and their consequences on ganglion cell visual responses.
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Congenital stationary night blindness with negative electroretinogram. A new classification.
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Molecular characterization of a novel retinal metabotropic glutamate receptor mGluR6 with a high agonist selectivity for L-2-amino-4-phosphonobutyrate.
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Identification of the gene and the mutation responsible for the mouse nob phenotype.
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The complete form of X-linked congenital stationary night blindness is caused by mutations in a gene encoding a leucine-rich repeat protein.
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Whole-exome sequencing identifies LRIT3 mutations as a cause of autosomal-recessive complete congenital stationary night blindness.
Christina Zeitz, Samuel G Jacobson, Christian P Hamel, Kinga Bujakowska, Marion Neuillé, Elise Orhan, Xavier Zanlonghi, Marie-Elise Lancelot, Christelle Michiels, Sharon B Schwartz,[...]. Am J Hum Genet 2013
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Whole-exome sequencing identifies mutations in GPR179 leading to autosomal-recessive complete congenital stationary night blindness.
Isabelle Audo, Kinga Bujakowska, Elise Orhan, Charlotte M Poloschek, Sabine Defoort-Dhellemmes, Isabelle Drumare, Susanne Kohl, Tien D Luu, Odile Lecompte, Eberhart Zrenner,[...]. Am J Hum Genet 2012
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Regulators of G protein signaling RGS7 and RGS11 determine the onset of the light response in ON bipolar neurons.
Yan Cao, Johan Pahlberg, Ignacio Sarria, Naomi Kamasawa, Alapakkam P Sampath, Kirill A Martemyanov. Proc Natl Acad Sci U S A 2012
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The metabotropic receptor mGluR6 may signal through G(o), but not phosphodiesterase, in retinal bipolar cells.
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Orphan receptor GPR179 forms macromolecular complexes with components of metabotropic signaling cascade in retina ON-bipolar neurons.
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Depolarizing bipolar cell dysfunction due to a Trpm1 point mutation.
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Lrit3 deficient mouse (nob6): a novel model of complete congenital stationary night blindness (cCSNB).
Marion Neuillé, Said El Shamieh, Elise Orhan, Christelle Michiels, Aline Antonio, Marie-Elise Lancelot, Christel Condroyer, Kinga Bujakowska, Olivier Poch, José-Alain Sahel,[...]. PLoS One 2014
Marion Neuillé, Said El Shamieh, Elise Orhan, Christelle Michiels, Aline Antonio, Marie-Elise Lancelot, Christel Condroyer, Kinga Bujakowska, Olivier Poch, José-Alain Sahel,[...]. PLoS One 2014
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Mutation of the calcium channel gene Cacna1f disrupts calcium signaling, synaptic transmission and cellular organization in mouse retina.
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Essential role of Ca2+-binding protein 4, a Cav1.4 channel regulator, in photoreceptor synaptic function.
Françoise Haeseleer, Yoshikazu Imanishi, Tadao Maeda, Daniel E Possin, Akiko Maeda, Amy Lee, Fred Rieke, Krzysztof Palczewski. Nat Neurosci 2004
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Developmentally regulated postsynaptic localization of a metabotropic glutamate receptor in rat rod bipolar cells.
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Retina-specific GTPase accelerator RGS11/G beta 5S/R9AP is a constitutive heterotrimer selectively targeted to mGluR6 in ON-bipolar neurons.
Yan Cao, Ikuo Masuho, Haruhisa Okawa, Keqiang Xie, Junko Asami, Paul J Kammermeier, Dennis M Maddox, Takahisa Furukawa, Takayoshi Inoue, Alapakkam P Sampath,[...]. J Neurosci 2009
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22
Recessive mutations of the gene TRPM1 abrogate ON bipolar cell function and cause complete congenital stationary night blindness in humans.
Zheng Li, Panagiotis I Sergouniotis, Michel Michaelides, Donna S Mackay, Genevieve A Wright, Sophie Devery, Anthony T Moore, Graham E Holder, Anthony G Robson, Andrew R Webster. Am J Hum Genet 2009
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Congenital stationary night blindness: an analysis and update of genotype-phenotype correlations and pathogenic mechanisms.
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GPR158/179 regulate G protein signaling by controlling localization and activity of the RGS7 complexes.
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Loss-of-function mutations in a calcium-channel alpha1-subunit gene in Xp11.23 cause incomplete X-linked congenital stationary night blindness.
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Structural and functional abnormalities of retinal ribbon synapses due to Cacna2d4 mutation.
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Localization of nyctalopin in the mammalian retina.
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Comparisons of structural and functional abnormalities in mouse b-wave mutants.
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TRPM1 mutations are associated with the complete form of congenital stationary night blindness.
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TRPM1: the endpoint of the mGluR6 signal transduction cascade in retinal ON-bipolar cells.
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TRPM1: a vertebrate TRP channel responsible for retinal ON bipolar function.
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mGluR6 deletion renders the TRPM1 channel in retina inactive.
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Signal transmission along retinal rods and the origin of the electroretinographic a-wave.
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Light response of retinal ON bipolar cells requires a specific splice variant of Galpha(o).
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Pikachurin, a dystroglycan ligand, is essential for photoreceptor ribbon synapse formation.
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Gbeta5-RGS complexes co-localize with mGluR6 in retinal ON-bipolar cells.
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Genetic inactivation of an inwardly rectifying potassium channel (Kir4.1 subunit) in mice: phenotypic impact in retina.
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Co-cited is the co-citation frequency, indicating how many articles cite the article together with the query article. Similarity is the co-citation as percentage of the times cited of the query article or the article in the search results, whichever is the lowest. These numbers are calculated for the last 100 citations when articles are cited more than 100 times.