Malay Mandal, Kelly M Crusio, Fanyong Meng, Suqing Liu, Marcus Kinsella, Marcus R Clark, Osamu Takeuchi, Iannis Aifantis. Proc Natl Acad Sci U S A 2008
Times Cited: 34
Times Cited: 34
Times Cited
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Similarity
Notch promotes survival of pre-T cells at the beta-selection checkpoint by regulating cellular metabolism.
Maria Ciofani, Juan Carlos Zúñiga-Pflücker. Nat Immunol 2005
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A developmental pathway involving four phenotypically and functionally distinct subsets of CD3-CD4-CD8- triple-negative adult mouse thymocytes defined by CD44 and CD25 expression.
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BH3-only Bcl-2 family member Bim is required for apoptosis of autoreactive thymocytes.
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Proapoptotic Bcl-2 relative Bim required for certain apoptotic responses, leukocyte homeostasis, and to preclude autoimmunity.
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Induction of T cell development from hematopoietic progenitor cells by delta-like-1 in vitro.
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The BH3-only protein bid is dispensable for DNA damage- and replicative stress-induced apoptosis or cell-cycle arrest.
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Obligatory role for cooperative signaling by pre-TCR and Notch during thymocyte differentiation.
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Heterogeneity among DN1 prothymocytes reveals multiple progenitors with different capacities to generate T cell and non-T cell lineages.
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Bclx regulates the survival of double-positive thymocytes.
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Delta-like 4 is the essential, nonredundant ligand for Notch1 during thymic T cell lineage commitment.
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Zoned out: functional mapping of stromal signaling microenvironments in the thymus.
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Gene expression patterns define novel roles for E47 in cell cycle progression, cytokine-mediated signaling, and T lineage development.
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Inactivation of Notch1 impairs VDJbeta rearrangement and allows pre-TCR-independent survival of early alpha beta Lineage Thymocytes.
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Early thymocyte development is regulated by modulation of E2A protein activity.
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Deficient T cell fate specification in mice with an induced inactivation of Notch1.
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Launching the T-cell-lineage developmental programme.
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E2A and HEB are required to block thymocyte proliferation prior to pre-TCR expression.
Jason Wojciechowski, Anne Lai, Motonari Kondo, Yuan Zhuang. J Immunol 2007
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Developmental and molecular characterization of emerging beta- and gammadelta-selected pre-T cells in the adult mouse thymus.
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Pre-TCR signaling and inactivation of p53 induces crucial cell survival pathways in pre-T cells.
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Deficiency in Bak and Bax perturbs thymic selection and lymphoid homeostasis.
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Life and death in the thymus--cell death signaling during T cell development.
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Akt promotes cell survival by phosphorylating and inhibiting a Forkhead transcription factor.
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SLP-65 regulates immunoglobulin light chain gene recombination through the PI(3)K-PKB-Foxo pathway.
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Combined deficiency of proapoptotic regulators Bim and Fas results in the early onset of systemic autoimmunity.
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Clonal deletion of thymocytes can occur in the cortex with no involvement of the medulla.
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Requirement for cyclin D3 in lymphocyte development and T cell leukemias.
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NOTCH1 directly regulates c-MYC and activates a feed-forward-loop transcriptional network promoting leukemic cell growth.
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Molecular pathogenesis of T-cell leukaemia and lymphoma.
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Bid, a Bcl2 interacting protein, mediates cytochrome c release from mitochondria in response to activation of cell surface death receptors.
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Proapoptotic BAX and BAK: a requisite gateway to mitochondrial dysfunction and death.
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Proapoptotic BID is an ATM effector in the DNA-damage response.
Iris Kamer, Rachel Sarig, Yehudit Zaltsman, Hagit Niv, Galia Oberkovitz, Limor Regev, Gal Haimovich, Yaniv Lerenthal, Richard C Marcellus, Atan Gross. Cell 2005
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