Valeria Naim, Filippo Rosselli. Nat Cell Biol 2009
Times Cited: 233
Times Cited: 233
Times Cited
Times Co-cited
Similarity
Replication stress induces sister-chromatid bridging at fragile site loci in mitosis.
Kok Lung Chan, Timea Palmai-Pallag, Songmin Ying, Ian D Hickson. Nat Cell Biol 2009
Kok Lung Chan, Timea Palmai-Pallag, Songmin Ying, Ian D Hickson. Nat Cell Biol 2009
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BLM is required for faithful chromosome segregation and its localization defines a class of ultrafine anaphase bridges.
Kok-Lung Chan, Phillip S North, Ian D Hickson. EMBO J 2007
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ERCC1 and MUS81-EME1 promote sister chromatid separation by processing late replication intermediates at common fragile sites during mitosis.
Valeria Naim, Therese Wilhelm, Michelle Debatisse, Filippo Rosselli. Nat Cell Biol 2013
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Replication stress activates DNA repair synthesis in mitosis.
Sheroy Minocherhomji, Songmin Ying, Victoria A Bjerregaard, Sara Bursomanno, Aiste Aleliunaite, Wei Wu, Hocine W Mankouri, Huahao Shen, Ying Liu, Ian D Hickson. Nature 2015
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MUS81 promotes common fragile site expression.
Songmin Ying, Sheroy Minocherhomji, Kok Lung Chan, Timea Palmai-Pallag, Wai Kit Chu, Theresa Wass, Hocine W Mankouri, Ying Liu, Ian D Hickson. Nat Cell Biol 2013
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53BP1 nuclear bodies form around DNA lesions generated by mitotic transmission of chromosomes under replication stress.
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PICH, a centromere-associated SNF2 family ATPase, is regulated by Plk1 and required for the spindle checkpoint.
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A distinct replication fork protection pathway connects Fanconi anemia tumor suppressors to RAD51-BRCA1/2.
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Replication stress induces 53BP1-containing OPT domains in G1 cells.
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DNA polymerase alpha inhibition by aphidicolin induces gaps and breaks at common fragile sites in human chromosomes.
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RAD52 Facilitates Mitotic DNA Synthesis Following Replication Stress.
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Unresolved recombination intermediates lead to ultra-fine anaphase bridges, chromosome breaks and aberrations.
Ying Wai Chan, Kasper Fugger, Stephen C West. Nat Cell Biol 2018
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Collisions between replication and transcription complexes cause common fragile site instability at the longest human genes.
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FANCD2 Facilitates Replication through Common Fragile Sites.
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The Fanconi anaemia pathway: new players and new functions.
Raphael Ceccaldi, Prabha Sarangi, Alan D D'Andrea. Nat Rev Mol Cell Biol 2016
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Cytokinesis failure occurs in Fanconi anemia pathway-deficient murine and human bone marrow hematopoietic cells.
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TopBP1 is required at mitosis to reduce transmission of DNA damage to G1 daughter cells.
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The Fanconi Anemia Pathway Protects Genome Integrity from R-loops.
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The Fanconi Anemia Pathway Maintains Genome Stability by Coordinating Replication and Transcription.
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Causes and consequences of replication stress.
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Double-strand break repair-independent role for BRCA2 in blocking stalled replication fork degradation by MRE11.
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The BLM helicase contributes to telomere maintenance through processing of late-replicating intermediate structures.
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Chromothripsis from DNA damage in micronuclei.
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Replication stress links structural and numerical cancer chromosomal instability.
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DNA synthesis by Pol η promotes fragile site stability by preventing under-replicated DNA in mitosis.
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Cell-type-specific replication initiation programs set fragility of the FRA3B fragile site.
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Break-induced replication repair of damaged forks induces genomic duplications in human cells.
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The Fanconi anemia pathway is required for the DNA replication stress response and for the regulation of common fragile site stability.
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DNA breaks and chromosome pulverization from errors in mitosis.
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Mammalian RAD52 Functions in Break-Induced Replication Repair of Collapsed DNA Replication Forks.
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Chromosome segregation errors as a cause of DNA damage and structural chromosome aberrations.
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The origins and processing of ultra fine anaphase DNA bridges.
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FANCD2 binds MCM proteins and controls replisome function upon activation of s phase checkpoint signaling.
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PICH promotes sister chromatid disjunction and co-operates with topoisomerase II in mitosis.
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Rif1 Is Required for Resolution of Ultrafine DNA Bridges in Anaphase to Ensure Genomic Stability.
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PICH and BLM limit histone association with anaphase centromeric DNA threads and promote their resolution.
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Chromothripsis and Kataegis Induced by Telomere Crisis.
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Replication of a common fragile site, FRA3B, occurs late in S phase and is delayed further upon induction: implications for the mechanism of fragile site induction.
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Co-cited is the co-citation frequency, indicating how many articles cite the article together with the query article. Similarity is the co-citation as percentage of the times cited of the query article or the article in the search results, whichever is the lowest. These numbers are calculated for the last 100 citations when articles are cited more than 100 times.