A citation-based method for searching scientific literature

Francesca Maltecca, Raffaella Magnoni, Federica Cerri, Gregory A Cox, Angelo Quattrini, Giorgio Casari. J Neurosci 2009
Times Cited: 75







List of co-cited articles
965 articles co-cited >1



Times Cited
  Times     Co-cited
Similarity


Mutations in the mitochondrial protease gene AFG3L2 cause dominant hereditary ataxia SCA28.
Daniela Di Bella, Federico Lazzaro, Alfredo Brusco, Massimo Plumari, Giorgio Battaglia, Annalisa Pastore, Adele Finardi, Claudia Cagnoli, Filippo Tempia, Marina Frontali,[...]. Nat Genet 2010
207
64

The mitochondrial protease AFG3L2 is essential for axonal development.
Francesca Maltecca, Asadollah Aghaie, David G Schroeder, Laura Cassina, Benjamin A Taylor, Sandra J Phillips, Mariachiara Malaguti, Stefano Previtali, Jean-Louis Guénet, Angelo Quattrini,[...]. J Neurosci 2008
77
52

Spastic paraplegia and OXPHOS impairment caused by mutations in paraplegin, a nuclear-encoded mitochondrial metalloprotease.
G Casari, M De Fusco, S Ciarmatori, M Zeviani, M Mora, P Fernandez, G De Michele, A Filla, S Cocozza, R Marconi,[...]. Cell 1998
597
45

The m-AAA protease defective in hereditary spastic paraplegia controls ribosome assembly in mitochondria.
Mark Nolden, Sarah Ehses, Mirko Koppen, Andrea Bernacchia, Elena I Rugarli, Thomas Langer. Cell 2005
272
44

Whole-exome sequencing identifies homozygous AFG3L2 mutations in a spastic ataxia-neuropathy syndrome linked to mitochondrial m-AAA proteases.
Tyler Mark Pierson, David Adams, Florian Bonn, Paola Martinelli, Praveen F Cherukuri, Jamie K Teer, Nancy F Hansen, Pedro Cruz, James C Mullikin For The Nisc Comparative Sequencing Program, Robert W Blakesley,[...]. PLoS Genet 2011
137
44

Variable and tissue-specific subunit composition of mitochondrial m-AAA protease complexes linked to hereditary spastic paraplegia.
Mirko Koppen, Metodi D Metodiev, Giorgio Casari, Elena I Rugarli, Thomas Langer. Mol Cell Biol 2007
132
40

Axonal degeneration in paraplegin-deficient mice is associated with abnormal mitochondria and impairment of axonal transport.
Fatima Ferreirinha, Angelo Quattrini, Marinella Pirozzi, Valentina Valsecchi, Giorgia Dina, Vania Broccoli, Alberto Auricchio, Fiorella Piemonte, Giulia Tozzi, Laura Gaeta,[...]. J Clin Invest 2004
204
32

Genetic interaction between the m-AAA protease isoenzymes reveals novel roles in cerebellar degeneration.
Paola Martinelli, Veronica La Mattina, Andrea Bernacchia, Raffaella Magnoni, Federica Cerri, Gregory Cox, Angelo Quattrini, Giorgio Casari, Elena I Rugarli. Hum Mol Genet 2009
41
56

AFG3L2 supports mitochondrial protein synthesis and Purkinje cell survival.
Eva R Almajan, Ricarda Richter, Lars Paeger, Paola Martinelli, Esther Barth, Thorsten Decker, Nils-Göran Larsson, Peter Kloppenburg, Thomas Langer, Elena I Rugarli. J Clin Invest 2012
72
31

Loss of m-AAA protease in mitochondria causes complex I deficiency and increased sensitivity to oxidative stress in hereditary spastic paraplegia.
Luigia Atorino, Laura Silvestri, Mirko Koppen, Laura Cassina, Andrea Ballabio, Roberto Marconi, Thomas Langer, Giorgio Casari. J Cell Biol 2003
187
29

SCA28, a novel form of autosomal dominant cerebellar ataxia on chromosome 18p11.22-q11.2.
Claudia Cagnoli, Caterina Mariotti, Franco Taroni, Marco Seri, Alessandro Brussino, Chiara Michielotto, Marina Grisoli, Daniela Di Bella, Nicola Migone, Cinzia Gellera,[...]. Brain 2006
81
28

Missense mutations in the AFG3L2 proteolytic domain account for ∼1.5% of European autosomal dominant cerebellar ataxias.
Claudia Cagnoli, Giovanni Stevanin, Alessandro Brussino, Marco Barberis, Cecilia Mancini, Russell L Margolis, Susan E Holmes, Marcello Nobili, Sylvie Forlani, Sergio Padovan,[...]. Hum Mutat 2010
58
36

Regulation of OPA1 processing and mitochondrial fusion by m-AAA protease isoenzymes and OMA1.
Sarah Ehses, Ines Raschke, Giuseppe Mancuso, Andrea Bernacchia, Stefan Geimer, Daniel Tondera, Jean-Claude Martinou, Benedikt Westermann, Elena I Rugarli, Thomas Langer. J Cell Biol 2009
359
26

Early onset and slow progression of SCA28, a rare dominant ataxia in a large four-generation family with a novel AFG3L2 mutation.
Ulf Edener, Janine Wöllner, Ute Hehr, Zacharias Kohl, Stefan Schilling, Friedmar Kreuz, Peter Bauer, Veronica Bernard, Gabriele Gillessen-Kaesbach, Christine Zühlke. Eur J Hum Genet 2010
37
45


Spinocerebellar ataxia type 28: a novel autosomal dominant cerebellar ataxia characterized by slow progression and ophthalmoparesis.
Caterina Mariotti, Alfredo Brusco, Daniela Di Bella, Claudia Cagnoli, Marco Seri, Cinzia Gellera, Stefano Di Donato, Franco Taroni. Cerebellum 2008
36
44

Purkinje neuron Ca2+ influx reduction rescues ataxia in SCA28 model.
Francesca Maltecca, Elisa Baseggio, Francesco Consolato, Davide Mazza, Paola Podini, Samuel M Young, Ilaria Drago, Ben A Bahr, Aldamaria Puliti, Franca Codazzi,[...]. J Clin Invest 2015
45
33


The m-AAA Protease Associated with Neurodegeneration Limits MCU Activity in Mitochondria.
Tim König, Simon E Tröder, Kavya Bakka, Anne Korwitz, Ricarda Richter-Dennerlein, Philipp A Lampe, Maria Patron, Mareike Mühlmeister, Sergio Guerrero-Castillo, Ulrich Brandt,[...]. Mol Cell 2016
90
18

An intersubunit signaling network coordinates ATP hydrolysis by m-AAA proteases.
Steffen Augustin, Florian Gerdes, Sukyeong Lee, Francis T F Tsai, Thomas Langer, Takashi Tatsuta. Mol Cell 2009
74
17

Spectrin mutations cause spinocerebellar ataxia type 5.
Yoshio Ikeda, Katherine A Dick, Marcy R Weatherspoon, Dan Gincel, Karen R Armbrust, Joline C Dalton, Giovanni Stevanin, Alexandra Dürr, Christine Zühlke, Katrin Bürk,[...]. Nat Genet 2006
242
17

Deletion at ITPR1 underlies ataxia in mice and spinocerebellar ataxia 15 in humans.
Joyce van de Leemput, Jayanth Chandran, Melanie A Knight, Lynne A Holtzclaw, Sonja Scholz, Mark R Cookson, Henry Houlden, Katrina Gwinn-Hardy, Hon-Chung Fung, Xian Lin,[...]. PLoS Genet 2007
192
17

Respiratory dysfunction by AFG3L2 deficiency causes decreased mitochondrial calcium uptake via organellar network fragmentation.
Francesca Maltecca, Diego De Stefani, Laura Cassina, Francesco Consolato, Michal Wasilewski, Luca Scorrano, Rosario Rizzuto, Giorgio Casari. Hum Mol Genet 2012
39
33

m-AAA protease-driven membrane dislocation allows intramembrane cleavage by rhomboid in mitochondria.
Takashi Tatsuta, Steffen Augustin, Mark Nolden, Björn Friedrichs, Thomas Langer. EMBO J 2007
86
16

Regulation of mitochondrial morphology through proteolytic cleavage of OPA1.
Naotada Ishihara, Yuu Fujita, Toshihiko Oka, Katsuyoshi Mihara. EMBO J 2006
565
16

Identification and characterization of AFG3L2, a novel paraplegin-related gene.
S Banfi, M T Bassi, G Andolfi, A Marchitiello, S Zanotta, A Ballabio, G Casari, B Franco. Genomics 1999
60
20


Bergmann glia expression of polyglutamine-expanded ataxin-7 produces neurodegeneration by impairing glutamate transport.
Sara K Custer, Gwenn A Garden, Nishi Gill, Udo Rueb, Randell T Libby, Christian Schultz, Stephan J Guyenet, Thomas Deller, Lesnick E Westrum, Bryce L Sopher,[...]. Nat Neurosci 2006
172
16

The i-AAA protease YME1L and OMA1 cleave OPA1 to balance mitochondrial fusion and fission.
Ruchika Anand, Timothy Wai, Michael J Baker, Nikolay Kladt, Astrid C Schauss, Elena Rugarli, Thomas Langer. J Cell Biol 2014
362
16

New roles for mitochondrial proteases in health, ageing and disease.
Pedro M Quirós, Thomas Langer, Carlos López-Otín. Nat Rev Mol Cell Biol 2015
271
16

Membrane protein degradation by AAA proteases in mitochondria: extraction of substrates from either membrane surface.
K Leonhard, B Guiard, G Pellecchia, A Tzagoloff, W Neupert, T Langer. Mol Cell 2000
140
14


Mitochondrial AAA proteases--towards a molecular understanding of membrane-bound proteolytic machines.
Florian Gerdes, Takashi Tatsuta, Thomas Langer. Biochim Biophys Acta 2012
83
14

Inducible proteolytic inactivation of OPA1 mediated by the OMA1 protease in mammalian cells.
Brian Head, Lorena Griparic, Mandana Amiri, Shilpa Gandre-Babbe, Alexander M van der Bliek. J Cell Biol 2009
300
14


A novel missense mutation in AFG3L2 associated with late onset and slow progression of spinocerebellar ataxia type 28.
Anna Mareike Löbbe, Jun-Suk Kang, Rüdiger Hilker, Holger Hackstein, Ulrich Müller, Dagmar Nolte. J Mol Neurosci 2014
18
61

Loss of the m-AAA protease subunit AFG₃L₂ causes mitochondrial transport defects and tau hyperphosphorylation.
Arun Kumar Kondadi, Shuaiyu Wang, Sara Montagner, Nikolay Kladt, Anne Korwitz, Paola Martinelli, David Herholz, Michael J Baker, Astrid C Schauss, Thomas Langer,[...]. EMBO J 2014
45
24

Autocatalytic processing of m-AAA protease subunits in mitochondria.
Mirko Koppen, Florian Bonn, Sarah Ehses, Thomas Langer. Mol Biol Cell 2009
37
27

Chaperone-like activity of the AAA domain of the yeast Yme1 AAA protease.
K Leonhard, A Stiegler, W Neupert, T Langer. Nature 1999
161
13

Deranged calcium signaling and neurodegeneration in spinocerebellar ataxia type 2.
Jing Liu, Tie-Shan Tang, Huiping Tu, Omar Nelson, Emily Herndon, Duong P Huynh, Stefan M Pulst, Ilya Bezprozvanny. J Neurosci 2009
182
13

Autosomal dominant cerebellar ataxia (SCA6) associated with small polyglutamine expansions in the alpha 1A-voltage-dependent calcium channel.
O Zhuchenko, J Bailey, P Bonnen, T Ashizawa, D W Stockton, C Amos, W B Dobyns, S H Subramony, H Y Zoghbi, C C Lee. Nat Genet 1997
13

Electron cryomicroscopy structure of a membrane-anchored mitochondrial AAA protease.
Sukyeong Lee, Steffen Augustin, Takashi Tatsuta, Florian Gerdes, Thomas Langer, Francis T F Tsai. J Biol Chem 2011
43
23

Stress-induced OMA1 activation and autocatalytic turnover regulate OPA1-dependent mitochondrial dynamics.
Michael J Baker, Philipp A Lampe, Diana Stojanovski, Anne Korwitz, Ruchika Anand, Takashi Tatsuta, Thomas Langer. EMBO J 2014
153
13

OPA1 processing controls mitochondrial fusion and is regulated by mRNA splicing, membrane potential, and Yme1L.
Zhiyin Song, Hsiuchen Chen, Maja Fiket, Christiane Alexander, David C Chan. J Cell Biol 2007
499
12

Regulation of the mitochondrial dynamin-like protein Opa1 by proteolytic cleavage.
Lorena Griparic, Takayuki Kanazawa, Alexander M van der Bliek. J Cell Biol 2007
320
12

Missense mutations in the regulatory domain of PKC gamma: a new mechanism for dominant nonepisodic cerebellar ataxia.
Dong-Hui Chen, Zoran Brkanac, Christophe L M J Verlinde, Xiao-Jian Tan, Laura Bylenok, David Nochlin, Mark Matsushita, Hillary Lipe, John Wolff, Magali Fernandez,[...]. Am J Hum Genet 2003
175
12

Autosomal dominant cerebellar ataxias: clinical features, genetics, and pathogenesis.
Ludger Schöls, Peter Bauer, Thorsten Schmidt, Thorsten Schulte, Olaf Riess. Lancet Neurol 2004
649
12

Presequence-dependent folding ensures MrpL32 processing by the m-AAA protease in mitochondria.
Florian Bonn, Takashi Tatsuta, Carmelina Petrungaro, Jan Riemer, Thomas Langer. EMBO J 2011
52
17

Regulation of mitochondrial phospholipids by Ups1/PRELI-like proteins depends on proteolysis and Mdm35.
Christoph Potting, Claudia Wilmes, Tanja Engmann, Christof Osman, Thomas Langer. EMBO J 2010
113
12

Mitochondrial fusion protects against neurodegeneration in the cerebellum.
Hsiuchen Chen, J Michael McCaffery, David C Chan. Cell 2007
594
12


Co-cited is the co-citation frequency, indicating how many articles cite the article together with the query article. Similarity is the co-citation as percentage of the times cited of the query article or the article in the search results, whichever is the lowest. These numbers are calculated for the last 100 citations when articles are cited more than 100 times.