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Times Cited: 619
Times Cited: 619
Times Cited
Times Co-cited
Similarity
ABAD directly links Abeta to mitochondrial toxicity in Alzheimer's disease.
Joyce W Lustbader, Maurizio Cirilli, Chang Lin, Hong Wei Xu, Kazuhiro Takuma, Ning Wang, Casper Caspersen, Xi Chen, Susan Pollak, Michael Chaney,[...]. Science 2004
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Mitophagy inhibits amyloid-β and tau pathology and reverses cognitive deficits in models of Alzheimer's disease.
Evandro F Fang, Yujun Hou, Konstantinos Palikaras, Bryan A Adriaanse, Jesse S Kerr, Beimeng Yang, Sofie Lautrup, Md Mahdi Hasan-Olive, Domenica Caponio, Xiuli Dan,[...]. Nat Neurosci 2019
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Impaired mitochondrial dynamics and abnormal interaction of amyloid beta with mitochondrial protein Drp1 in neurons from patients with Alzheimer's disease: implications for neuronal damage.
Maria Manczak, Marcus J Calkins, P Hemachandra Reddy. Hum Mol Genet 2011
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Early deficits in synaptic mitochondria in an Alzheimer's disease mouse model.
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Impaired balance of mitochondrial fission and fusion in Alzheimer's disease.
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Mitochondrial dysfunction is a trigger of Alzheimer's disease pathophysiology.
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Accumulation of amyloid precursor protein in the mitochondrial import channels of human Alzheimer's disease brain is associated with mitochondrial dysfunction.
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A "mitochondrial cascade hypothesis" for sporadic Alzheimer's disease.
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Cyclophilin D deficiency attenuates mitochondrial and neuronal perturbation and ameliorates learning and memory in Alzheimer's disease.
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Impaired mitochondrial biogenesis, defective axonal transport of mitochondria, abnormal mitochondrial dynamics and synaptic degeneration in a mouse model of Alzheimer's disease.
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Amyloid-beta and tau synergistically impair the oxidative phosphorylation system in triple transgenic Alzheimer's disease mice.
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Parkin-mediated mitophagy in mutant hAPP neurons and Alzheimer's disease patient brains.
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Brain glucose hypometabolism and oxidative stress in preclinical Alzheimer's disease.
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Differential expression of oxidative phosphorylation genes in patients with Alzheimer's disease: implications for early mitochondrial dysfunction and oxidative damage.
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Amyloid beta, mitochondrial dysfunction and synaptic damage: implications for cognitive decline in aging and Alzheimer's disease.
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Amyloid-beta overproduction causes abnormal mitochondrial dynamics via differential modulation of mitochondrial fission/fusion proteins.
Xinglong Wang, Bo Su, Sandra L Siedlak, Paula I Moreira, Hisashi Fujioka, Yang Wang, Gemma Casadesus, Xiongwei Zhu. Proc Natl Acad Sci U S A 2008
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The ubiquitin kinase PINK1 recruits autophagy receptors to induce mitophagy.
Michael Lazarou, Danielle A Sliter, Lesley A Kane, Shireen A Sarraf, Chunxin Wang, Jonathon L Burman, Dionisia P Sideris, Adam I Fogel, Richard J Youle. Nature 2015
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Increased mitochondrial calcium levels associated with neuronal death in a mouse model of Alzheimer's disease.
Maria Calvo-Rodriguez, Steven S Hou, Austin C Snyder, Elizabeth K Kharitonova, Alyssa N Russ, Sudeshna Das, Zhanyun Fan, Alona Muzikansky, Monica Garcia-Alloza, Alberto Serrano-Pozo,[...]. Nat Commun 2020
Maria Calvo-Rodriguez, Steven S Hou, Austin C Snyder, Elizabeth K Kharitonova, Alyssa N Russ, Sudeshna Das, Zhanyun Fan, Alona Muzikansky, Monica Garcia-Alloza, Alberto Serrano-Pozo,[...]. Nat Commun 2020
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S-nitrosylation of Drp1 mediates beta-amyloid-related mitochondrial fission and neuronal injury.
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Inhibition of Drp1 Ameliorates Synaptic Depression, Aβ Deposition, and Cognitive Impairment in an Alzheimer's Disease Model.
Seung Hyun Baek, So Jung Park, Jae In Jeong, Sung Hyun Kim, Jihoon Han, Jae Won Kyung, Sang-Ha Baik, Yuri Choi, Bo Youn Choi, Jin Su Park,[...]. J Neurosci 2017
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Mitochondria are a direct site of A beta accumulation in Alzheimer's disease neurons: implications for free radical generation and oxidative damage in disease progression.
Maria Manczak, Thimmappa S Anekonda, Edward Henson, Byung S Park, Joseph Quinn, P Hemachandra Reddy. Hum Mol Genet 2006
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Impaired mitochondrial biogenesis contributes to mitochondrial dysfunction in Alzheimer's disease.
Baiyang Sheng, Xinglong Wang, Bo Su, Hyoung-gon Lee, Gemma Casadesus, George Perry, Xiongwei Zhu. J Neurochem 2012
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The amyloid hypothesis of Alzheimer's disease at 25 years.
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Mitochondria dysfunction in the pathogenesis of Alzheimer's disease: recent advances.
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GLUT1 reductions exacerbate Alzheimer's disease vasculo-neuronal dysfunction and degeneration.
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Mitochondria-targeted catalase reduces abnormal APP processing, amyloid β production and BACE1 in a mouse model of Alzheimer's disease: implications for neuroprotection and lifespan extension.
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Large-scale proteomic analysis of Alzheimer's disease brain and cerebrospinal fluid reveals early changes in energy metabolism associated with microglia and astrocyte activation.
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White Matter Lipids as a Ketogenic Fuel Supply in Aging Female Brain: Implications for Alzheimer's Disease.
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Inhibition of mitochondrial fragmentation protects against Alzheimer's disease in rodent model.
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Co-cited is the co-citation frequency, indicating how many articles cite the article together with the query article. Similarity is the co-citation as percentage of the times cited of the query article or the article in the search results, whichever is the lowest. These numbers are calculated for the last 100 citations when articles are cited more than 100 times.