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Times Co-cited
Similarity
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Functional impact of global rare copy number variation in autism spectrum disorders.
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The contribution of de novo coding mutations to autism spectrum disorder.
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Synaptic, transcriptional and chromatin genes disrupted in autism.
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De novo mutations revealed by whole-exome sequencing are strongly associated with autism.
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Sporadic autism exomes reveal a highly interconnected protein network of de novo mutations.
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Strong association of de novo copy number mutations with autism.
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Multiple recurrent de novo CNVs, including duplications of the 7q11.23 Williams syndrome region, are strongly associated with autism.
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Identification of common genetic risk variants for autism spectrum disorder.
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Genetic relationship between five psychiatric disorders estimated from genome-wide SNPs.
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Excess of rare, inherited truncating mutations in autism.
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Autism genome-wide copy number variation reveals ubiquitin and neuronal genes.
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Integrative functional genomic analyses implicate specific molecular pathways and circuits in autism.
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Coexpression networks implicate human midfetal deep cortical projection neurons in the pathogenesis of autism.
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Global prevalence of autism and other pervasive developmental disorders.
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Multiplex targeted sequencing identifies recurrently mutated genes in autism spectrum disorders.
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Mutations of the X-linked genes encoding neuroligins NLGN3 and NLGN4 are associated with autism.
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Using whole-exome sequencing to identify inherited causes of autism.
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Co-cited is the co-citation frequency, indicating how many articles cite the article together with the query article. Similarity is the co-citation as percentage of the times cited of the query article or the article in the search results, whichever is the lowest. These numbers are calculated for the last 100 citations when articles are cited more than 100 times.