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Times Cited: 895
Times Cited: 895
Times Cited
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Similarity
Shank3 mutant mice display autistic-like behaviours and striatal dysfunction.
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Autism-like behavioral phenotypes in BTBR T+tf/J mice.
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Mechanisms Underlying Microbial-Mediated Changes in Social Behavior in Mouse Models of Autism Spectrum Disorder.
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Autistic-like behaviour and cerebellar dysfunction in Purkinje cell Tsc1 mutant mice.
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Autism-associated neuroligin-3 mutations commonly impair striatal circuits to boost repetitive behaviors.
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The cerebellum, sensitive periods, and autism.
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Microbial Reconstitution Reverses Maternal Diet-Induced Social and Synaptic Deficits in Offspring.
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Behavioral phenotypes of genetic mouse models of autism.
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Adult restoration of Shank3 expression rescues selective autistic-like phenotypes.
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Dysfunction in GABA signalling mediates autism-like stereotypies and Rett syndrome phenotypes.
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The maternal interleukin-17a pathway in mice promotes autism-like phenotypes in offspring.
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Social deficits in IRSp53 mutant mice improved by NMDAR and mGluR5 suppression.
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CHD8 haploinsufficiency results in autistic-like phenotypes in mice.
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Excitatory/Inhibitory Balance and Circuit Homeostasis in Autism Spectrum Disorders.
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Increased Excitation-Inhibition Ratio Stabilizes Synapse and Circuit Excitability in Four Autism Mouse Models.
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Autistic-like behaviours and hyperactivity in mice lacking ProSAP1/Shank2.
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Dysfunctional cerebellar Purkinje cells contribute to autism-like behaviour in Shank2-deficient mice.
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Absence of CNTNAP2 leads to epilepsy, neuronal migration abnormalities, and core autism-related deficits.
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Marble burying and nestlet shredding as tests of repetitive, compulsive-like behaviors in mice.
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Autism spectrum disorder.
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Identification of common genetic risk variants for autism spectrum disorder.
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Chd8 Mutation Leads to Autistic-like Behaviors and Impaired Striatal Circuits.
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The valproic acid-induced rodent model of autism.
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Autistic-Like Traits and Cerebellar Dysfunction in Purkinje Cell PTEN Knock-Out Mice.
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Social deficits in Shank3-deficient mouse models of autism are rescued by histone deacetylase (HDAC) inhibition.
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Foetal oestrogens and autism.
Simon Baron-Cohen, Alexandros Tsompanidis, Bonnie Auyeung, Bent Nørgaard-Pedersen, David M Hougaard, Morsi Abdallah, Arieh Cohen, Alexa Pohl. Mol Psychiatry 2020
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A review of research trends in physiological abnormalities in autism spectrum disorders: immune dysregulation, inflammation, oxidative stress, mitochondrial dysfunction and environmental toxicant exposures.
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Multiplex targeted sequencing identifies recurrently mutated genes in autism spectrum disorders.
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Co-cited is the co-citation frequency, indicating how many articles cite the article together with the query article. Similarity is the co-citation as percentage of the times cited of the query article or the article in the search results, whichever is the lowest. These numbers are calculated for the last 100 citations when articles are cited more than 100 times.