Guoqi Liu, Xiaomi Chen, John J Bissler, Richard R Sinden, Michael Leffak. Nat Chem Biol 2010
Times Cited: 108
Times Cited: 108
Times Cited
Times Co-cited
Similarity
Mechanisms of trinucleotide repeat instability during human development.
Cynthia T McMurray. Nat Rev Genet 2010
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Trinucleotide repeats that expand in human disease form hairpin structures in vitro.
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Repeat instability as the basis for human diseases and as a potential target for therapy.
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OGG1 initiates age-dependent CAG trinucleotide expansion in somatic cells.
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Slipped (CTG)*(CAG) repeats can be correctly repaired, escape repair or undergo error-prone repair.
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Detection of slipped-DNAs at the trinucleotide repeats of the myotonic dystrophy type I disease locus in patient tissues.
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Double-strand break repair pathways protect against CAG/CTG repeat expansions, contractions and repeat-mediated chromosomal fragility in Saccharomyces cerevisiae.
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Stability of a CTG/CAG trinucleotide repeat in yeast is dependent on its orientation in the genome.
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The balancing act of DNA repeat expansions.
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Transcription promotes contraction of CAG repeat tracts in human cells.
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17
Zinc-finger directed double-strand breaks within CAG repeat tracts promote repeat instability in human cells.
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Disease-associated repeat instability and mismatch repair.
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Isolated short CTG/CAG DNA slip-outs are repaired efficiently by hMutSbeta, but clustered slip-outs are poorly repaired.
Gagan B Panigrahi, Meghan M Slean, Jodie P Simard, Opher Gileadi, Christopher E Pearson. Proc Natl Acad Sci U S A 2010
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22
CTG repeat instability and size variation timing in DNA repair-deficient mice.
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MSH2 ATPase domain mutation affects CTG*CAG repeat instability in transgenic mice.
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22
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14
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14
The role of break-induced replication in large-scale expansions of (CAG)n/(CTG)n repeats.
Jane C Kim, Samantha T Harris, Teresa Dinter, Kartik A Shah, Sergei M Mirkin. Nat Struct Mol Biol 2017
Jane C Kim, Samantha T Harris, Teresa Dinter, Kartik A Shah, Sergei M Mirkin. Nat Struct Mol Biol 2017
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Structural analysis of slipped-strand DNA (S-DNA) formed in (CTG)n. (CAG)n repeats from the myotonic dystrophy locus.
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Coordination between polymerase beta and FEN1 can modulate CAG repeat expansion.
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14
Replication inhibitors modulate instability of an expanded trinucleotide repeat at the myotonic dystrophy type 1 disease locus in human cells.
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Zhi Yang, Rachel Lau, Julien L Marcadier, David Chitayat, Christopher E Pearson. Am J Hum Genet 2003
21
Huntington's and myotonic dystrophy hESCs: down-regulated trinucleotide repeat instability and mismatch repair machinery expression upon differentiation.
Anna Seriola, Claudia Spits, Jodie P Simard, Pierre Hilven, Patrick Haentjens, Christopher E Pearson, Karen Sermon. Hum Mol Genet 2011
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13
MutSβ and histone deacetylase complexes promote expansions of trinucleotide repeats in human cells.
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DNA base excision repair: a mechanism of trinucleotide repeat expansion.
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Highly specific contractions of a single CAG/CTG trinucleotide repeat by TALEN in yeast.
Guy-Franck Richard, David Viterbo, Varun Khanna, Valentine Mosbach, Lauriane Castelain, Bernard Dujon. PLoS One 2014
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