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Times Cited: 394
Times Cited: 394
Times Cited
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Similarity
Inflammation directs memory precursor and short-lived effector CD8(+) T cell fates via the graded expression of T-bet transcription factor.
Nikhil S Joshi, Weiguo Cui, Anmol Chandele, Heung Kyu Lee, David R Urso, James Hagman, Laurent Gapin, Susan M Kaech. Immunity 2007
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Defining CD8+ T cells that provide the proliferative burst after PD-1 therapy.
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Essential role of the Wnt pathway effector Tcf-1 for the establishment of functional CD8 T cell memory.
Grégoire Jeannet, Caroline Boudousquié, Noémie Gardiol, Joonsoo Kang, Joerg Huelsken, Werner Held. Proc Natl Acad Sci U S A 2010
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T Cell Factor 1-Expressing Memory-like CD8(+) T Cells Sustain the Immune Response to Chronic Viral Infections.
Daniel T Utzschneider, Mélanie Charmoy, Vijaykumar Chennupati, Laurène Pousse, Daniela Pais Ferreira, Sandra Calderon-Copete, Maxime Danilo, Francesca Alfei, Maike Hofmann, Dominik Wieland,[...]. Immunity 2016
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Intratumoral Tcf1+PD-1+CD8+ T Cells with Stem-like Properties Promote Tumor Control in Response to Vaccination and Checkpoint Blockade Immunotherapy.
Imran Siddiqui, Karin Schaeuble, Vijaykumar Chennupati, Silvia A Fuertes Marraco, Sandra Calderon-Copete, Daniela Pais Ferreira, Santiago J Carmona, Leonardo Scarpellino, David Gfeller, Sylvain Pradervand,[...]. Immunity 2019
Imran Siddiqui, Karin Schaeuble, Vijaykumar Chennupati, Silvia A Fuertes Marraco, Sandra Calderon-Copete, Daniela Pais Ferreira, Santiago J Carmona, Leonardo Scarpellino, David Gfeller, Sylvain Pradervand,[...]. Immunity 2019
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The TCF1-Bcl6 axis counteracts type I interferon to repress exhaustion and maintain T cell stemness.
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Transcriptional control of effector and memory CD8+ T cell differentiation.
Susan M Kaech, Weiguo Cui. Nat Rev Immunol 2012
Susan M Kaech, Weiguo Cui. Nat Rev Immunol 2012
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TOX reinforces the phenotype and longevity of exhausted T cells in chronic viral infection.
Francesca Alfei, Kristiyan Kanev, Maike Hofmann, Ming Wu, Hazem E Ghoneim, Patrick Roelli, Daniel T Utzschneider, Madlaina von Hoesslin, Jolie G Cullen, Yiping Fan,[...]. Nature 2019
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TOX transcriptionally and epigenetically programs CD8+ T cell exhaustion.
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Single-cell RNA-seq reveals TOX as a key regulator of CD8+ T cell persistence in chronic infection.
Chen Yao, Hong-Wei Sun, Neal E Lacey, Yun Ji, E Ashley Moseman, Han-Yu Shih, Elisabeth F Heuston, Martha Kirby, Stacie Anderson, Jun Cheng,[...]. Nat Immunol 2019
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CD8 T Cell Exhaustion During Chronic Viral Infection and Cancer.
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TCF-1-Centered Transcriptional Network Drives an Effector versus Exhausted CD8 T Cell-Fate Decision.
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TOX is a critical regulator of tumour-specific T cell differentiation.
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Subsets of exhausted CD8+ T cells differentially mediate tumor control and respond to checkpoint blockade.
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Wnt signaling arrests effector T cell differentiation and generates CD8+ memory stem cells.
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Defining T Cell States Associated with Response to Checkpoint Immunotherapy in Melanoma.
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Checkpoint Blockade Immunotherapy Induces Dynamic Changes in PD-1-CD8+ Tumor-Infiltrating T Cells.
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Effector and memory CD8+ T cell fate coupled by T-bet and eomesodermin.
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Epigenetic stability of exhausted T cells limits durability of reinvigoration by PD-1 blockade.
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The transcriptional regulators Id2 and Id3 control the formation of distinct memory CD8+ T cell subsets.
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Runx3 programs CD8+ T cell residency in non-lymphoid tissues and tumours.
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CXCR5(+) follicular cytotoxic T cells control viral infection in B cell follicles.
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Transcriptional repressor Blimp-1 promotes CD8(+) T cell terminal differentiation and represses the acquisition of central memory T cell properties.
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Progenitor and terminal subsets of CD8+ T cells cooperate to contain chronic viral infection.
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Follicular CXCR5- expressing CD8(+) T cells curtail chronic viral infection.
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TOX and TOX2 transcription factors cooperate with NR4A transcription factors to impose CD8+ T cell exhaustion.
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A human memory T cell subset with stem cell-like properties.
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Differentiation of CD8 memory T cells depends on Foxo1.
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Hobit and Blimp1 instruct a universal transcriptional program of tissue residency in lymphocytes.
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Suppression of Tcf1 by Inflammatory Cytokines Facilitates Effector CD8 T Cell Differentiation.
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Developmental Relationships of Four Exhausted CD8+ T Cell Subsets Reveals Underlying Transcriptional and Epigenetic Landscape Control Mechanisms.
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Proliferating Transitory T Cells with an Effector-like Transcriptional Signature Emerge from PD-1+ Stem-like CD8+ T Cells during Chronic Infection.
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Cutting edge: The transcription factor eomesodermin enables CD8+ T cells to compete for the memory cell niche.
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The developmental pathway for CD103(+)CD8+ tissue-resident memory T cells of skin.
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The Chemokine Receptor CX3CR1 Defines Three Antigen-Experienced CD8 T Cell Subsets with Distinct Roles in Immune Surveillance and Homeostasis.
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Constitutive activation of Wnt signaling favors generation of memory CD8 T cells.
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Selective expression of the interleukin 7 receptor identifies effector CD8 T cells that give rise to long-lived memory cells.
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Molecular and cellular insights into T cell exhaustion.
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The transcription factor NFAT promotes exhaustion of activated CD8⁺ T cells.
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Restoring function in exhausted CD8 T cells during chronic viral infection.
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The epigenetic landscape of T cell exhaustion.
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A critical role for TCF-1 in T-lineage specification and differentiation.
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Epigenetic signature of PD-1+ TCF1+ CD8 T cells that act as resource cells during chronic viral infection and respond to PD-1 blockade.
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BACH2 regulates CD8(+) T cell differentiation by controlling access of AP-1 factors to enhancers.
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CD4+ T Cell Help Is Required for the Formation of a Cytolytic CD8+ T Cell Subset that Protects against Chronic Infection and Cancer.
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Co-cited is the co-citation frequency, indicating how many articles cite the article together with the query article. Similarity is the co-citation as percentage of the times cited of the query article or the article in the search results, whichever is the lowest. These numbers are calculated for the last 100 citations when articles are cited more than 100 times.