Claudia Lukas, Velibor Savic, Simon Bekker-Jensen, Carsten Doil, Beate Neumann, Ronni Sølvhøj Pedersen, Merete Grøfte, Kok Lung Chan, Ian David Hickson, Jiri Bartek, Jiri Lukas. Nat Cell Biol 2011
Times Cited: 463
Times Cited: 463
Times Cited
Times Co-cited
Similarity
Replication stress activates DNA repair synthesis in mitosis.
Sheroy Minocherhomji, Songmin Ying, Victoria A Bjerregaard, Sara Bursomanno, Aiste Aleliunaite, Wei Wu, Hocine W Mankouri, Huahao Shen, Ying Liu, Ian D Hickson. Nature 2015
Sheroy Minocherhomji, Songmin Ying, Victoria A Bjerregaard, Sara Bursomanno, Aiste Aleliunaite, Wei Wu, Hocine W Mankouri, Huahao Shen, Ying Liu, Ian D Hickson. Nature 2015
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Replication stress induces 53BP1-containing OPT domains in G1 cells.
Jeanine A Harrigan, Rimma Belotserkovskaya, Julia Coates, Daniela S Dimitrova, Sophie E Polo, Charles R Bradshaw, Peter Fraser, Stephen P Jackson. J Cell Biol 2011
Jeanine A Harrigan, Rimma Belotserkovskaya, Julia Coates, Daniela S Dimitrova, Sophie E Polo, Charles R Bradshaw, Peter Fraser, Stephen P Jackson. J Cell Biol 2011
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Replication stress induces sister-chromatid bridging at fragile site loci in mitosis.
Kok Lung Chan, Timea Palmai-Pallag, Songmin Ying, Ian D Hickson. Nat Cell Biol 2009
Kok Lung Chan, Timea Palmai-Pallag, Songmin Ying, Ian D Hickson. Nat Cell Biol 2009
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Causes and consequences of replication stress.
Michelle K Zeman, Karlene A Cimprich. Nat Cell Biol 2014
Michelle K Zeman, Karlene A Cimprich. Nat Cell Biol 2014
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ERCC1 and MUS81-EME1 promote sister chromatid separation by processing late replication intermediates at common fragile sites during mitosis.
Valeria Naim, Therese Wilhelm, Michelle Debatisse, Filippo Rosselli. Nat Cell Biol 2013
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RAD52 Facilitates Mitotic DNA Synthesis Following Replication Stress.
Rahul Bhowmick, Sheroy Minocherhomji, Ian D Hickson. Mol Cell 2016
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53BP1 nuclear bodies enforce replication timing at under-replicated DNA to limit heritable DNA damage.
Julian Spies, Claudia Lukas, Kumar Somyajit, Maj-Britt Rask, Jiri Lukas, Kai John Neelsen. Nat Cell Biol 2019
Julian Spies, Claudia Lukas, Kumar Somyajit, Maj-Britt Rask, Jiri Lukas, Kai John Neelsen. Nat Cell Biol 2019
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DNA replication stress as a hallmark of cancer.
Morgane Macheret, Thanos D Halazonetis. Annu Rev Pathol 2015
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MUS81 promotes common fragile site expression.
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ATR prohibits replication catastrophe by preventing global exhaustion of RPA.
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Activation of the DNA damage checkpoint and genomic instability in human precancerous lesions.
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DNA damage response as a candidate anti-cancer barrier in early human tumorigenesis.
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Intragenic origins due to short G1 phases underlie oncogene-induced DNA replication stress.
Morgane Macheret, Thanos D Halazonetis. Nature 2018
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BLM is required for faithful chromosome segregation and its localization defines a class of ultrafine anaphase bridges.
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Unreplicated DNA remaining from unperturbed S phases passes through mitosis for resolution in daughter cells.
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A distinct replication fork protection pathway connects Fanconi anemia tumor suppressors to RAD51-BRCA1/2.
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Replication stress links structural and numerical cancer chromosomal instability.
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Nucleotide deficiency promotes genomic instability in early stages of cancer development.
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DNA synthesis by Pol η promotes fragile site stability by preventing under-replicated DNA in mitosis.
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Mammalian RAD52 Functions in Break-Induced Replication Repair of Collapsed DNA Replication Forks.
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Unresolved recombination intermediates lead to ultra-fine anaphase bridges, chromosome breaks and aberrations.
Ying Wai Chan, Kasper Fugger, Stephen C West. Nat Cell Biol 2018
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Double-strand break repair-independent role for BRCA2 in blocking stalled replication fork degradation by MRE11.
Katharina Schlacher, Nicole Christ, Nicolas Siaud, Akinori Egashira, Hong Wu, Maria Jasin. Cell 2011
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Rad51-mediated replication fork reversal is a global response to genotoxic treatments in human cells.
Ralph Zellweger, Damian Dalcher, Karun Mutreja, Matteo Berti, Jonas A Schmid, Raquel Herrador, Alessandro Vindigni, Massimo Lopes. J Cell Biol 2015
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Replication stress and cancer.
Hélène Gaillard, Tatiana García-Muse, Andrés Aguilera. Nat Rev Cancer 2015
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An intrinsic S/G2 checkpoint enforced by ATR.
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The FANC pathway and BLM collaborate during mitosis to prevent micro-nucleation and chromosome abnormalities.
Valeria Naim, Filippo Rosselli. Nat Cell Biol 2009
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MRE11 and EXO1 nucleases degrade reversed forks and elicit MUS81-dependent fork rescue in BRCA2-deficient cells.
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Mechanisms of Oncogene-Induced Replication Stress: Jigsaw Falling into Place.
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The essential kinase ATR: ensuring faithful duplication of a challenging genome.
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Replication fork reversal triggers fork degradation in BRCA2-defective cells.
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Cell-type-specific replication initiation programs set fragility of the FRA3B fragile site.
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PICH, a centromere-associated SNF2 family ATPase, is regulated by Plk1 and required for the spindle checkpoint.
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ATR activates the S-M checkpoint during unperturbed growth to ensure sufficient replication prior to mitotic onset.
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Sensing DNA damage through ATRIP recognition of RPA-ssDNA complexes.
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Restoration of Replication Fork Stability in BRCA1- and BRCA2-Deficient Cells by Inactivation of SNF2-Family Fork Remodelers.
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Replication fork stability confers chemoresistance in BRCA-deficient cells.
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Endogenous Replication Stress in Mother Cells Leads to Quiescence of Daughter Cells.
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FANCD2 Facilitates Replication through Common Fragile Sites.
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Co-cited is the co-citation frequency, indicating how many articles cite the article together with the query article. Similarity is the co-citation as percentage of the times cited of the query article or the article in the search results, whichever is the lowest. These numbers are calculated for the last 100 citations when articles are cited more than 100 times.