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The genetic basis of early T-cell precursor acute lymphoblastic leukaemia.
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Early T-cell precursor leukaemia: a subtype of very high-risk acute lymphoblastic leukaemia.
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HOXA genes are included in genetic and biologic networks defining human acute T-cell leukemia (T-ALL).
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Activating mutations of NOTCH1 in human T cell acute lymphoblastic leukemia.
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The genetics and mechanisms of T cell acute lymphoblastic leukaemia.
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Immature MEF2C-dysregulated T-cell leukemia patients have an early T-cell precursor acute lymphoblastic leukemia gene signature and typically have non-rearranged T-cell receptors.
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ETV6 mutations in early immature human T cell leukemias.
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An early thymic precursor phenotype predicts outcome exclusively in HOXA-overexpressing adult T-cell acute lymphoblastic leukemia: a Group for Research in Adult Acute Lymphoblastic Leukemia study.
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ABT-199 mediated inhibition of BCL-2 as a novel therapeutic strategy in T-cell acute lymphoblastic leukemia.
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Targeted sequencing identifies associations between IL7R-JAK mutations and epigenetic modulators in T-cell acute lymphoblastic leukemia.
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Oncogene regulation. An oncogenic super-enhancer formed through somatic mutation of a noncoding intergenic element.
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Genomic analyses identify recurrent MEF2D fusions in acute lymphoblastic leukaemia.
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JAK/STAT pathway inhibition overcomes IL7-induced glucocorticoid resistance in a subset of human T-cell acute lymphoblastic leukemias.
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The TLX1 oncogene drives aneuploidy in T cell transformation.
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Oncogenic IL7R gain-of-function mutations in childhood T-cell acute lymphoblastic leukemia.
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Clinical and molecular characterization of early T-cell precursor leukemia: a high-risk subgroup in adult T-ALL with a high frequency of FLT3 mutations.
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The genetic basis and cell of origin of mixed phenotype acute leukaemia.
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The cardiac homeobox gene NKX2-5 is deregulated by juxtaposition with BCL11B in pediatric T-ALL cell lines via a novel t(5;14)(q35.1;q32.2).
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Absence of biallelic TCRgamma deletion predicts early treatment failure in pediatric T-cell acute lymphoblastic leukemia.
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Maturation stage of T-cell acute lymphoblastic leukemia determines BCL-2 versus BCL-XL dependence and sensitivity to ABT-199.
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Deregulation of a homeobox gene, HOX11, by the t(10;14) in T cell leukemia.
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MEF2C is activated by multiple mechanisms in a subset of T-acute lymphoblastic leukemia cell lines.
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Core transcriptional regulatory circuit controlled by the TAL1 complex in human T cell acute lymphoblastic leukemia.
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NKX3.1 is a direct TAL1 target gene that mediates proliferation of TAL1-expressing human T cell acute lymphoblastic leukemia.
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Whole-exome sequencing in adult ETP-ALL reveals a high rate of DNMT3A mutations.
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A new recurrent and specific cryptic translocation, t(5;14)(q35;q32), is associated with expression of the Hox11L2 gene in T acute lymphoblastic leukemia.
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TLX homeodomain oncogenes mediate T cell maturation arrest in T-ALL via interaction with ETS1 and suppression of TCRα gene expression.
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High expression of myocyte enhancer factor 2C (MEF2C) is associated with adverse-risk features and poor outcome in pediatric acute myeloid leukemia: a report from the Children's Oncology Group.
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The 2016 revision to the World Health Organization classification of myeloid neoplasms and acute leukemia.
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Homing and invasiveness of MLL/ENL leukemic cells is regulated by MEF2C.
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Co-cited is the co-citation frequency, indicating how many articles cite the article together with the query article. Similarity is the co-citation as percentage of the times cited of the query article or the article in the search results, whichever is the lowest. These numbers are calculated for the last 100 citations when articles are cited more than 100 times.