Benjamin R Miller, Adam G Walker, Scott J Barton, George V Rebec. Front Syst Neurosci 2011
Times Cited: 55
Times Cited: 55
Times Cited
Times Co-cited
Similarity
49
Altered information processing in the prefrontal cortex of Huntington's disease mouse models.
Adam G Walker, Benjamin R Miller, Jenna N Fritsch, Scott J Barton, George V Rebec. J Neurosci 2008
Adam G Walker, Benjamin R Miller, Jenna N Fritsch, Scott J Barton, George V Rebec. J Neurosci 2008
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Exon 1 of the HD gene with an expanded CAG repeat is sufficient to cause a progressive neurological phenotype in transgenic mice.
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Dysfunctional behavioral modulation of corticostriatal communication in the R6/2 mouse model of Huntington's disease.
S Lee Hong, Desirée Cossyleon, Wajeeha A Hussain, Lauren J Walker, Scott J Barton, George V Rebec. PLoS One 2012
S Lee Hong, Desirée Cossyleon, Wajeeha A Hussain, Lauren J Walker, Scott J Barton, George V Rebec. PLoS One 2012
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Dysregulated information processing by medium spiny neurons in striatum of freely behaving mouse models of Huntington's disease.
Benjamin R Miller, Adam G Walker, Anand S Shah, Scott J Barton, George V Rebec. J Neurophysiol 2008
Benjamin R Miller, Adam G Walker, Anand S Shah, Scott J Barton, George V Rebec. J Neurophysiol 2008
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Hyperactive striatal neurons in symptomatic Huntington R6/2 mice: variations with behavioral state and repeated ascorbate treatment.
G V Rebec, S K Conroy, S J Barton. Neuroscience 2006
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Alterations in cortical excitation and inhibition in genetic mouse models of Huntington's disease.
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Pathophysiology of Huntington's disease: time-dependent alterations in synaptic and receptor function.
L A Raymond, V M André, C Cepeda, C M Gladding, A J Milnerwood, M S Levine. Neuroscience 2011
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The corticostriatal pathway in Huntington's disease.
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Electrophysiological and morphological changes in striatal spiny neurons in R6/2 Huntington's disease transgenic mice.
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Comprehensive behavioral and molecular characterization of a new knock-in mouse model of Huntington's disease: zQ175.
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Characterization of neurophysiological and behavioral changes, MRI brain volumetry and 1H MRS in zQ175 knock-in mouse model of Huntington's disease.
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Full-length human mutant huntingtin with a stable polyglutamine repeat can elicit progressive and selective neuropathogenesis in BACHD mice.
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Genetic mouse models of Huntington's disease: focus on electrophysiological mechanisms.
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Up-regulation of GLT1 expression increases glutamate uptake and attenuates the Huntington's disease phenotype in the R6/2 mouse.
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Age-dependent alterations of corticostriatal activity in the YAC128 mouse model of Huntington disease.
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Co-cited is the co-citation frequency, indicating how many articles cite the article together with the query article. Similarity is the co-citation as percentage of the times cited of the query article or the article in the search results, whichever is the lowest. These numbers are calculated for the last 100 citations when articles are cited more than 100 times.