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Times Co-cited
Similarity
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Katharina Schlacher, Nicole Christ, Nicolas Siaud, Akinori Egashira, Hong Wu, Maria Jasin. Cell 2011
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Replication fork reversal triggers fork degradation in BRCA2-defective cells.
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Rad51-mediated replication fork reversal is a global response to genotoxic treatments in human cells.
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MRE11 and EXO1 nucleases degrade reversed forks and elicit MUS81-dependent fork rescue in BRCA2-deficient cells.
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Rad51 protects nascent DNA from Mre11-dependent degradation and promotes continuous DNA synthesis.
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Causes and consequences of replication stress.
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DNA2 drives processing and restart of reversed replication forks in human cells.
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Replication fork reversal in eukaryotes: from dead end to dynamic response.
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Replication Fork Slowing and Reversal upon DNA Damage Require PCNA Polyubiquitination and ZRANB3 DNA Translocase Activity.
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Human RECQ1 promotes restart of replication forks reversed by DNA topoisomerase I inhibition.
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Hydroxyurea-stalled replication forks become progressively inactivated and require two different RAD51-mediated pathways for restart and repair.
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High speed of fork progression induces DNA replication stress and genomic instability.
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Topoisomerase I poisoning results in PARP-mediated replication fork reversal.
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CtIP-Mediated Fork Protection Synergizes with BRCA1 to Suppress Genomic Instability upon DNA Replication Stress.
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RADX Promotes Genome Stability and Modulates Chemosensitivity by Regulating RAD51 at Replication Forks.
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HLTF's Ancient HIRAN Domain Binds 3' DNA Ends to Drive Replication Fork Reversal.
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ATR phosphorylates SMARCAL1 to prevent replication fork collapse.
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Replication stress activates DNA repair synthesis in mitosis.
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ATR prohibits replication catastrophe by preventing global exhaustion of RPA.
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The DNA translocase FANCM/MHF promotes replication traverse of DNA interstrand crosslinks.
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The shieldin complex mediates 53BP1-dependent DNA repair.
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Co-cited is the co-citation frequency, indicating how many articles cite the article together with the query article. Similarity is the co-citation as percentage of the times cited of the query article or the article in the search results, whichever is the lowest. These numbers are calculated for the last 100 citations when articles are cited more than 100 times.