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Novel mutations target distinct subgroups of medulloblastoma.
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Molecular subgroups of medulloblastoma: the current consensus.
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Subgroup-specific structural variation across 1,000 medulloblastoma genomes.
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Medulloblastoma comprises four distinct molecular variants.
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Novel molecular subgroups for clinical classification and outcome prediction in childhood medulloblastoma: a cohort study.
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Genome sequencing of SHH medulloblastoma predicts genotype-related response to smoothened inhibition.
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The 2016 World Health Organization Classification of Tumors of the Central Nervous System: a summary.
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Subtypes of medulloblastoma have distinct developmental origins.
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Risk stratification of childhood medulloblastoma in the molecular era: the current consensus.
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The genetic landscape of the childhood cancer medulloblastoma.
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Integrative genomic analysis of medulloblastoma identifies a molecular subgroup that drives poor clinical outcome.
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Active medulloblastoma enhancers reveal subgroup-specific cellular origins.
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Subgroup-specific prognostic implications of TP53 mutation in medulloblastoma.
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Divergent clonal selection dominates medulloblastoma at recurrence.
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Integrated genomics identifies five medulloblastoma subtypes with distinct genetic profiles, pathway signatures and clinicopathological features.
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Enhancer hijacking activates GFI1 family oncogenes in medulloblastoma.
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Spectrum and prevalence of genetic predisposition in medulloblastoma: a retrospective genetic study and prospective validation in a clinical trial cohort.
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Resolving medulloblastoma cellular architecture by single-cell genomics.
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Medulloblastoma: clinicopathological correlates of SHH, WNT, and non-SHH/WNT molecular subgroups.
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Genomics identifies medulloblastoma subgroups that are enriched for specific genetic alterations.
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Childhood cerebellar tumours mirror conserved fetal transcriptional programs.
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A mouse model of the most aggressive subgroup of human medulloblastoma.
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Pediatric and adult sonic hedgehog medulloblastomas are clinically and molecularly distinct.
Paul A Northcott, Thomas Hielscher, Adrian Dubuc, Stephen Mack, David Shih, Marc Remke, Hani Al-Halabi, Steffen Albrecht, Nada Jabado, Charles G Eberhart,[...]. Acta Neuropathol 2011
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DNA methylation-based classification of central nervous system tumours.
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Risk-adapted craniospinal radiotherapy followed by high-dose chemotherapy and stem-cell rescue in children with newly diagnosed medulloblastoma (St Jude Medulloblastoma-96): long-term results from a prospective, multicentre trial.
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Recurrence patterns across medulloblastoma subgroups: an integrated clinical and molecular analysis.
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Cancer-associated DDX3X mutations drive stress granule assembly and impair global translation.
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BET bromodomain inhibition of MYC-amplified medulloblastoma.
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Genome sequencing of pediatric medulloblastoma links catastrophic DNA rearrangements with TP53 mutations.
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Aberrant patterns of H3K4 and H3K27 histone lysine methylation occur across subgroups in medulloblastoma.
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Combined MYC and P53 defects emerge at medulloblastoma relapse and define rapidly progressive, therapeutically targetable disease.
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Aberrant ERBB4-SRC Signaling as a Hallmark of Group 4 Medulloblastoma Revealed by Integrative Phosphoproteomic Profiling.
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Cytogenetic prognostication within medulloblastoma subgroups.
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Acquisition of granule neuron precursor identity is a critical determinant of progenitor cell competence to form Shh-induced medulloblastoma.
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Proteomics, Post-translational Modifications, and Integrative Analyses Reveal Molecular Heterogeneity within Medulloblastoma Subgroups.
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Second-generation molecular subgrouping of medulloblastoma: an international meta-analysis of Group 3 and Group 4 subtypes.
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Exome sequencing identifies somatic mutations of DDX3X in natural killer/T-cell lymphoma.
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An animal model of MYC-driven medulloblastoma.
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HDAC and PI3K Antagonists Cooperate to Inhibit Growth of MYC-Driven Medulloblastoma.
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The 2007 WHO classification of tumours of the central nervous system.
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beta-Catenin status predicts a favorable outcome in childhood medulloblastoma: the United Kingdom Children's Cancer Study Group Brain Tumour Committee.
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New Brain Tumor Entities Emerge from Molecular Classification of CNS-PNETs.
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Irreversible growth plate fusions in children with medulloblastoma treated with a targeted hedgehog pathway inhibitor.
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Co-cited is the co-citation frequency, indicating how many articles cite the article together with the query article. Similarity is the co-citation as percentage of the times cited of the query article or the article in the search results, whichever is the lowest. These numbers are calculated for the last 100 citations when articles are cited more than 100 times.