R E Steinert, C Feinle-Bisset, N Geary, C Beglinger. J Anim Sci 2013
Times Cited: 35
Times Cited: 35
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Anorexia induction by the trichothecene deoxynivalenol (vomitoxin) is mediated by the release of the gut satiety hormone peptide YY.
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Role of cholecystokinin in anorexia induction following oral exposure to the 8-ketotrichothecenes deoxynivalenol, 15-acetyldeoxynivalenol, 3-acetyldeoxynivalenol, fusarenon X, and nivalenol.
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Characterization of deoxynivalenol-induced anorexia using mouse bioassay.
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Effects of intraduodenal fatty acids on appetite, antropyloroduodenal motility, and plasma CCK and GLP-1 in humans vary with their chain length.
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Diet: friend or foe of enteroendocrine cells--how it interacts with enteroendocrine cells.
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Comparison of murine anorectic responses to the 8-ketotrichothecenes 3-acetyldeoxynivalenol, 15-acetyldeoxynivalenol, fusarenon X and nivalenol.
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Immunochemical assessment of deoxynivalenol tissue distribution following oral exposure in the mouse.
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Gut hormone PYY(3-36) physiologically inhibits food intake.
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Acute effects of PYY3-36 on food intake and hypothalamic neuropeptide expression in the mouse.
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Deoxynivalenol: mechanisms of action, human exposure, and toxicological relevance.
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Evidence that intestinal glucagon-like peptide-1 plays a physiological role in satiety.
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G-protein-coupled receptors in intestinal chemosensation.
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Digestive physiology of the pig symposium: G protein-coupled receptors in nutrient chemosensation and gastrointestinal hormone secretion.
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Nesfatin-1 neurons in paraventricular and supraoptic nuclei of the rat hypothalamus coexpress oxytocin and vasopressin and are activated by refeeding.
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A role for glucagon-like peptide-1 in the central regulation of feeding.
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From the gut to the brain: journey and pathophysiological effects of the food-associated trichothecene mycotoxin deoxynivalenol.
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Peptide YY3-36 and 5-hydroxytryptamine mediate emesis induction by trichothecene deoxynivalenol (vomitoxin).
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The role of gut hormones in the regulation of body weight and energy homeostasis.
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Central inflammation and sickness-like behavior induced by the food contaminant deoxynivalenol: a PGE2-independent mechanism.
Clémence Girardet, Marion S Bonnet, Rajae Jdir, Medhi Sadoud, Sylvie Thirion, Catherine Tardivel, Julien Roux, Bruno Lebrun, Lourdes Mounien, Jérôme Trouslard,[...]. Toxicol Sci 2011
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Intraduodenal protein modulates antropyloroduodenal motility, hormone release, glycemia, appetite, and energy intake in lean men.
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Postprandial suppression of plasma ghrelin level is proportional to ingested caloric load but does not predict intermeal interval in humans.
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Effects of 8-week exposure of the B6C3F1 mouse to dietary deoxynivalenol (vomitoxin) and zearalenone.
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Cholecystokinin decreases food intake in rats.
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Comparison of Anorectic Potencies of the Trichothecenes T-2 Toxin, HT-2 Toxin and Satratoxin G to the Ipecac Alkaloid Emetine.
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Deoxynivalenol (Vomitoxin)-Induced Cholecystokinin and Glucagon-Like Peptide-1 Release in the STC-1 Enteroendocrine Cell Model Is Mediated by Calcium-Sensing Receptor and Transient Receptor Potential Ankyrin-1 Channel.
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Co-cited is the co-citation frequency, indicating how many articles cite the article together with the query article. Similarity is the co-citation as percentage of the times cited of the query article or the article in the search results, whichever is the lowest. These numbers are calculated for the last 100 citations when articles are cited more than 100 times.