Gene mutation profiles and prognostic implications in Korean patients with T-lymphoblastic leukemia.
Hee Jae Huh, Soo Hyun Lee, Keon Hee Yoo, Ki Woong Sung, Hong Hoe Koo, Jun Ho Jang, Kihyun Kim, Seok Jin Kim, Won Seog Kim, Chul Won Jung, Ki-O Lee, Sun-Hee Kim, Hee-Jin Kim. Ann Hematol 2013
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Mutations of PHF6 are associated with mutations of NOTCH1, JAK1 and rearrangement of SET-NUP214 in T-cell acute lymphoblastic leukemia.
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PHF6 mutations in T-cell acute lymphoblastic leukemia.
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Activating mutations of NOTCH1 in human T cell acute lymphoblastic leukemia.
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The molecular profile of adult T-cell acute lymphoblastic leukemia: mutations in RUNX1 and DNMT3A are associated with poor prognosis in T-ALL.
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The genomic landscape of pediatric and young adult T-lineage acute lymphoblastic leukemia.
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Somatic mutation of PHF6 gene in T-cell acute lymphoblatic leukemia, acute myelogenous leukemia and hepatocellular carcinoma.
Nam Jin Yoo, Yoo Ri Kim, Sug Hyung Lee. Acta Oncol 2012
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FBW7 mutations in leukemic cells mediate NOTCH pathway activation and resistance to gamma-secretase inhibitors.
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The SCFFBW7 ubiquitin ligase complex as a tumor suppressor in T cell leukemia.
Benjamin J Thompson, Silvia Buonamici, Maria Luisa Sulis, Teresa Palomero, Tomas Vilimas, Giuseppe Basso, Adolfo Ferrando, Iannis Aifantis. J Exp Med 2007
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NOTCH1 mutations in T-cell acute lymphoblastic leukemia: prognostic significance and implication in multifactorial leukemogenesis.
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Gene expression signatures define novel oncogenic pathways in T cell acute lymphoblastic leukemia.
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Gain-of-function mutations in interleukin-7 receptor-α (IL7R) in childhood acute lymphoblastic leukemias.
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Oncogenic IL7R gain-of-function mutations in childhood T-cell acute lymphoblastic leukemia.
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FBXW7 and NOTCH1 mutations in childhood T cell acute lymphoblastic leukaemia and T cell non-Hodgkin lymphoma.
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The genetic basis of early T-cell precursor acute lymphoblastic leukaemia.
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PHF6 regulates cell cycle progression by suppressing ribosomal RNA synthesis.
Jiadong Wang, Justin Wai-chung Leung, Zihua Gong, Lin Feng, Xiaobing Shi, Junjie Chen. J Biol Chem 2013
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PHF6 interacts with the nucleosome remodeling and deacetylation (NuRD) complex.
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Mutations in PHF6 are associated with Börjeson-Forssman-Lehmann syndrome.
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Activating NOTCH1 mutations predict favorable early treatment response and long-term outcome in childhood precursor T-cell lymphoblastic leukemia.
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Genetic mutational profiling analysis of T cell acute lymphoblastic leukemia reveal mutant FBXW7 as a prognostic indicator for inferior survival.
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A cooperative microRNA-tumor suppressor gene network in acute T-cell lymphoblastic leukemia (T-ALL).
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T-cell acute lymphoblastic leukemia in association with Börjeson-Forssman-Lehmann syndrome due to a mutation in PHF6.
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Protein and gene expression analysis of Phf6, the gene mutated in the Börjeson-Forssman-Lehmann Syndrome of intellectual disability and obesity.
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NOTCH1 directly regulates c-MYC and activates a feed-forward-loop transcriptional network promoting leukemic cell growth.
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The X-linked intellectual disability protein PHF6 associates with the PAF1 complex and regulates neuronal migration in the mammalian brain.
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The molecular basis of T cell acute lymphoblastic leukemia.
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Mutational loss of PTEN induces resistance to NOTCH1 inhibition in T-cell leukemia.
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Toward a NOTCH1/FBXW7/RAS/PTEN-based oncogenetic risk classification of adult T-cell acute lymphoblastic leukemia: a Group for Research in Adult Acute Lymphoblastic Leukemia study.
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NOTCH1 and/or FBXW7 mutations predict for initial good prednisone response but not for improved outcome in pediatric T-cell acute lymphoblastic leukemia patients treated on DCOG or COALL protocols.
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Targeted sequencing identifies associations between IL7R-JAK mutations and epigenetic modulators in T-cell acute lymphoblastic leukemia.
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Novel activating mutations lacking cysteine in type I cytokine receptors in acute lymphoblastic leukemia.
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MEK and PI3K-AKT inhibitors synergistically block activated IL7 receptor signaling in T-cell acute lymphoblastic leukemia.
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NOTCH1 and FBXW7 mutations have a favorable impact on early response to treatment, but not on outcome, in children with T-cell acute lymphoblastic leukemia (T-ALL) treated on EORTC trials 58881 and 58951.
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The favorable effect of activating NOTCH1 receptor mutations on long-term outcome in T-ALL patients treated on the ALL-BFM 2000 protocol can be separated from FBXW7 loss of function.
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The clinical picture of the Börjeson-Forssman-Lehmann syndrome in males and heterozygous females with PHF6 mutations.
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PHF6 Deletions May Cause Borjeson-Forssman-Lehmann Syndrome in Females.
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A novel PHF6 mutation results in enhanced exon skipping and mild Börjeson-Forssman-Lehmann syndrome.
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High frequency of PTEN, PI3K, and AKT abnormalities in T-cell acute lymphoblastic leukemia.
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A genome-scale in vivo loss-of-function screen identifies Phf6 as a lineage-specific regulator of leukemia cell growth.
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Cell biology of IL-7, a key lymphotrophin.
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Ras pathway mutations are prevalent in relapsed childhood acute lymphoblastic leukemia and confer sensitivity to MEK inhibition.
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Co-cited is the co-citation frequency, indicating how many articles cite the article together with the query article. Similarity is the co-citation as percentage of the times cited of the query article or the article in the search results, whichever is the lowest. These numbers are calculated for the last 100 citations when articles are cited more than 100 times.