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Times Cited: 117
Times Cited: 117
Times Cited
Times Co-cited
Similarity
Ubiquitinated TDP-43 in frontotemporal lobar degeneration and amyotrophic lateral sclerosis.
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Astrocyte pathology and the absence of non-cell autonomy in an induced pluripotent stem cell model of TDP-43 proteinopathy.
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Astrocytes expressing ALS-linked mutated SOD1 release factors selectively toxic to motor neurons.
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Astrocytes as determinants of disease progression in inherited amyotrophic lateral sclerosis.
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TDP-43 mutations in familial and sporadic amyotrophic lateral sclerosis.
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Mutations in FUS, an RNA processing protein, cause familial amyotrophic lateral sclerosis type 6.
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Mutations in Cu/Zn superoxide dismutase gene are associated with familial amyotrophic lateral sclerosis.
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Mutations in the FUS/TLS gene on chromosome 16 cause familial amyotrophic lateral sclerosis.
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TDP-43 mutant transgenic mice develop features of ALS and frontotemporal lobar degeneration.
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Genomic analysis of reactive astrogliosis.
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Reactive astrocytes secrete lcn2 to promote neuron death.
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Wild-type nonneuronal cells extend survival of SOD1 mutant motor neurons in ALS mice.
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TARDBP mutations in individuals with sporadic and familial amyotrophic lateral sclerosis.
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Focal transplantation-based astrocyte replacement is neuroprotective in a model of motor neuron disease.
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Co-cited is the co-citation frequency, indicating how many articles cite the article together with the query article. Similarity is the co-citation as percentage of the times cited of the query article or the article in the search results, whichever is the lowest. These numbers are calculated for the last 100 citations when articles are cited more than 100 times.