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Times Cited: 17
Times Cited: 17
Times Cited
Times Co-cited
Similarity
Autism genome-wide copy number variation reveals ubiquitin and neuronal genes.
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De novo gene disruptions in children on the autistic spectrum.
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Multiplex targeted sequencing identifies recurrently mutated genes in autism spectrum disorders.
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Insights into Autism Spectrum Disorder Genomic Architecture and Biology from 71 Risk Loci.
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Using whole-exome sequencing to identify inherited causes of autism.
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Characteristics and predictive value of blood transcriptome signature in males with autism spectrum disorders.
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Common genetic variants on 5p14.1 associate with autism spectrum disorders.
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Contactin 4 as an autism susceptibility locus.
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Prospective diagnostic analysis of copy number variants using SNP microarrays in individuals with autism spectrum disorders.
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Disruption of contactin 4 in three subjects with autism spectrum disorder.
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Association between microdeletion and microduplication at 16p11.2 and autism.
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Genetic architecture in autism spectrum disorder.
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The behavioral phenotype in MECP2 duplication syndrome: a comparison with idiopathic autism.
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A common genetic variant in the neurexin superfamily member CNTNAP2 increases familial risk of autism.
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Identifying autism loci and genes by tracing recent shared ancestry.
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Family-based genome-wide association scan of attention-deficit/hyperactivity disorder.
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Genome-wide association scan of quantitative traits for attention deficit hyperactivity disorder identifies novel associations and confirms candidate gene associations.
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Identification of a functional rare variant in autism using genome-wide screen for monoallelic expression.
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Strong association of de novo copy number mutations with autism.
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Patterns and rates of exonic de novo mutations in autism spectrum disorders.
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Sporadic autism exomes reveal a highly interconnected protein network of de novo mutations.
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17
De novo mutations revealed by whole-exome sequencing are strongly associated with autism.
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Uncovering molecular biomarkers that correlate cognitive decline with the changes of hippocampus' gene expression profiles in Alzheimer's disease.
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Gene expression profiling differentiates autism case-controls and phenotypic variants of autism spectrum disorders: evidence for circadian rhythm dysfunction in severe autism.
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Molecular genetic overlap in bipolar disorder, schizophrenia, and major depressive disorder.
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Transcriptional consequences of schizophrenia candidate miR-137 manipulation in human neural progenitor cells.
Matthew J Hill, Jacek G Donocik, Rosamond A Nuamah, Charles A Mein, Ricardo Sainz-Fuertes, Nicholas J Bray. Schizophr Res 2014
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Pooled DNA genotyping on Affymetrix SNP genotyping arrays.
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Familial clustering of schizophrenia, bipolar disorder, and major depressive disorder.
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Genetic and functional analyses of SHANK2 mutations suggest a multiple hit model of autism spectrum disorders.
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The impact of phenotypic and genetic heterogeneity on results of genome wide association studies of complex diseases.
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Genome-wide scan of copy number variation in late-onset Alzheimer's disease.
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Whole genome association study in a homogenous population in Shandong peninsula of China reveals JARID2 as a susceptibility gene for schizophrenia.
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17
Gene-network analysis identifies susceptibility genes related to glycobiology in autism.
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Absence of BRINP1 in mice causes increase of hippocampal neurogenesis and behavioral alterations relevant to human psychiatric disorders.
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A discovery resource of rare copy number variations in individuals with autism spectrum disorder.
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A novel relationship for schizophrenia, bipolar and major depressive disorder Part 7: A hint from chromosome 7 high density association screen.
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Risk of mental illness in offspring of parents with schizophrenia, bipolar disorder, and major depressive disorder: a meta-analysis of family high-risk studies.
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The bipolar disorder risk allele at CACNA1C also confers risk of recurrent major depression and of schizophrenia.
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Exome sequencing in sporadic autism spectrum disorders identifies severe de novo mutations.
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Synaptic, transcriptional and chromatin genes disrupted in autism.
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17
Co-cited is the co-citation frequency, indicating how many articles cite the article together with the query article. Similarity is the co-citation as percentage of the times cited of the query article or the article in the search results, whichever is the lowest. These numbers are calculated for the last 100 citations when articles are cited more than 100 times.