G Poelmans, B Franke, D L Pauls, J C Glennon, J K Buitelaar. Transl Psychiatry 2013
Times Cited: 53
Times Cited: 53
Times Cited
Times Co-cited
Similarity
Integrated genome-wide association study findings: identification of a neurodevelopmental network for attention deficit hyperactivity disorder.
Geert Poelmans, David L Pauls, Jan K Buitelaar, Barbara Franke. Am J Psychiatry 2011
Geert Poelmans, David L Pauls, Jan K Buitelaar, Barbara Franke. Am J Psychiatry 2011
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Functional impact of global rare copy number variation in autism spectrum disorders.
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Identification of risk loci with shared effects on five major psychiatric disorders: a genome-wide analysis.
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Genetic relationship between five psychiatric disorders estimated from genome-wide SNPs.
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MAGMA: generalized gene-set analysis of GWAS data.
Christiaan A de Leeuw, Joris M Mooij, Tom Heskes, Danielle Posthuma. PLoS Comput Biol 2015
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Transcriptomic analysis of autistic brain reveals convergent molecular pathology.
Irina Voineagu, Xinchen Wang, Patrick Johnston, Jennifer K Lowe, Yuan Tian, Steve Horvath, Jonathan Mill, Rita M Cantor, Benjamin J Blencowe, Daniel H Geschwind. Nature 2011
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Sporadic autism exomes reveal a highly interconnected protein network of de novo mutations.
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De novo mutations revealed by whole-exome sequencing are strongly associated with autism.
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Synaptic, transcriptional and chromatin genes disrupted in autism.
Silvia De Rubeis, Xin He, Arthur P Goldberg, Christopher S Poultney, Kaitlin Samocha, A Erucment Cicek, Yan Kou, Li Liu, Menachem Fromer, Susan Walker,[...]. Nature 2014
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Multiple recurrent de novo CNVs, including duplications of the 7q11.23 Williams syndrome region, are strongly associated with autism.
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Patterns and rates of exonic de novo mutations in autism spectrum disorders.
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Common genetic variants on 5p14.1 associate with autism spectrum disorders.
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Coexpression networks implicate human midfetal deep cortical projection neurons in the pathogenesis of autism.
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Candidate genetic pathways for attention-deficit/hyperactivity disorder (ADHD) show association to hyperactive/impulsive symptoms in children with ADHD.
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An integrated molecular landscape implicates the regulation of dendritic spine formation through insulin-related signalling in obsessive-compulsive disorder.
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Rare de novo variants associated with autism implicate a large functional network of genes involved in formation and function of synapses.
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Genetic heritability and shared environmental factors among twin pairs with autism.
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A genome-wide scan for common alleles affecting risk for autism.
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Structural variation of chromosomes in autism spectrum disorder.
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Genetic architecture in autism spectrum disorder.
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Dendritic spine pathology in neuropsychiatric disorders.
Peter Penzes, Michael E Cahill, Kelly A Jones, Jon-Eric VanLeeuwen, Kevin M Woolfrey. Nat Neurosci 2011
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Integrative functional genomic analyses implicate specific molecular pathways and circuits in autism.
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A method and server for predicting damaging missense mutations.
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Polygenic transmission and complex neuro developmental network for attention deficit hyperactivity disorder: genome-wide association study of both common and rare variants.
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Rare chromosomal deletions and duplications in attention-deficit hyperactivity disorder: a genome-wide analysis.
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Genome-wide copy number variation study associates metabotropic glutamate receptor gene networks with attention deficit hyperactivity disorder.
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Understanding mechanisms underlying human gene expression variation with RNA sequencing.
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Orientation, distance, regulation and function of neighbouring genes.
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Candidate gene studies of ADHD: a meta-analytic review.
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High-resolution mapping of expression-QTLs yields insight into human gene regulation.
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Attention-deficit/hyperactivity disorder.
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Stephen V Faraone, Philip Asherson, Tobias Banaschewski, Joseph Biederman, Jan K Buitelaar, Josep Antoni Ramos-Quiroga, Luis Augusto Rohde, Edmund J S Sonuga-Barke, Rosemary Tannock, Barbara Franke. Nat Rev Dis Primers 2015
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The contribution of de novo coding mutations to autism spectrum disorder.
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Common genetic variants, acting additively, are a major source of risk for autism.
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Linkage, association, and gene-expression analyses identify CNTNAP2 as an autism-susceptibility gene.
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Etiological heterogeneity in autism spectrum disorders: more than 100 genetic and genomic disorders and still counting.
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Multiplex targeted sequencing identifies recurrently mutated genes in autism spectrum disorders.
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Strong association of de novo copy number mutations with autism.
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Co-cited is the co-citation frequency, indicating how many articles cite the article together with the query article. Similarity is the co-citation as percentage of the times cited of the query article or the article in the search results, whichever is the lowest. These numbers are calculated for the last 100 citations when articles are cited more than 100 times.