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Times Cited: 80
Times Cited: 80
Times Cited
Times Co-cited
Similarity
Early increase in extrasynaptic NMDA receptor signaling and expression contributes to phenotype onset in Huntington's disease mice.
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Endocytosis and synaptic removal of NR3A-containing NMDA receptors by PACSIN1/syndapin1.
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NMDA receptor subunit diversity: impact on receptor properties, synaptic plasticity and disease.
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Exon 1 of the HD gene with an expanded CAG repeat is sufficient to cause a progressive neurological phenotype in transgenic mice.
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Balance between synaptic versus extrasynaptic NMDA receptor activity influences inclusions and neurotoxicity of mutant huntingtin.
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Expression of cocaine-evoked synaptic plasticity by GluN3A-containing NMDA receptors.
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Downregulation of NR3A-containing NMDARs is required for synapse maturation and memory consolidation.
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Excitatory glycine receptors containing the NR3 family of NMDA receptor subunits.
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Selective striatal neuronal loss in a YAC128 mouse model of Huntington disease.
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Pathophysiology of Huntington's disease: time-dependent alterations in synaptic and receptor function.
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Impaired TrkB receptor signaling underlies corticostriatal dysfunction in Huntington's disease.
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Emerging roles of GluN3-containing NMDA receptors in the CNS.
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13
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Calpain and STriatal-Enriched protein tyrosine phosphatase (STEP) activation contribute to extrasynaptic NMDA receptor localization in a Huntington's disease mouse model.
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12
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Dendritic spine pathology and deficits in experience-dependent dendritic plasticity in R6/1 Huntington's disease transgenic mice.
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Full-length human mutant huntingtin with a stable polyglutamine repeat can elicit progressive and selective neuropathogenesis in BACHD mice.
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12
Co-cited is the co-citation frequency, indicating how many articles cite the article together with the query article. Similarity is the co-citation as percentage of the times cited of the query article or the article in the search results, whichever is the lowest. These numbers are calculated for the last 100 citations when articles are cited more than 100 times.