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Times Cited: 844
Times Cited: 844
Times Cited
Times Co-cited
Similarity
A Unique Microglia Type Associated with Restricting Development of Alzheimer's Disease.
Hadas Keren-Shaul, Amit Spinrad, Assaf Weiner, Orit Matcovitch-Natan, Raz Dvir-Szternfeld, Tyler K Ulland, Eyal David, Kuti Baruch, David Lara-Astaiso, Beata Toth,[...]. Cell 2017
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Identification of a unique TGF-β-dependent molecular and functional signature in microglia.
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Fate mapping analysis reveals that adult microglia derive from primitive macrophages.
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The TREM2-APOE Pathway Drives the Transcriptional Phenotype of Dysfunctional Microglia in Neurodegenerative Diseases.
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New tools for studying microglia in the mouse and human CNS.
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Single-Cell RNA Sequencing of Microglia throughout the Mouse Lifespan and in the Injured Brain Reveals Complex Cell-State Changes.
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An RNA-sequencing transcriptome and splicing database of glia, neurons, and vascular cells of the cerebral cortex.
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Microglia sculpt postnatal neural circuits in an activity and complement-dependent manner.
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Transcriptomic analysis of purified human cortical microglia reveals age-associated changes.
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Synaptic pruning by microglia is necessary for normal brain development.
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Neurotoxic reactive astrocytes are induced by activated microglia.
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Microglia promote learning-dependent synapse formation through brain-derived neurotrophic factor.
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Microglia emerge from erythromyeloid precursors via Pu.1- and Irf8-dependent pathways.
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Colony-stimulating factor 1 receptor signaling is necessary for microglia viability, unmasking a microglia progenitor cell in the adult brain.
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Lipid-droplet-accumulating microglia represent a dysfunctional and proinflammatory state in the aging brain.
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Innate immune memory in the brain shapes neurological disease hallmarks.
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Developmental Heterogeneity of Microglia and Brain Myeloid Cells Revealed by Deep Single-Cell RNA Sequencing.
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TREM2 lipid sensing sustains the microglial response in an Alzheimer's disease model.
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Disease-Associated Microglia: A Universal Immune Sensor of Neurodegeneration.
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A neurodegeneration-specific gene-expression signature of acutely isolated microglia from an amyotrophic lateral sclerosis mouse model.
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Human and mouse single-nucleus transcriptomics reveal TREM2-dependent and TREM2-independent cellular responses in Alzheimer's disease.
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Microglia emerge as central players in brain disease.
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Microglia contribute to normal myelinogenesis and to oligodendrocyte progenitor maintenance during adulthood.
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Co-cited is the co-citation frequency, indicating how many articles cite the article together with the query article. Similarity is the co-citation as percentage of the times cited of the query article or the article in the search results, whichever is the lowest. These numbers are calculated for the last 100 citations when articles are cited more than 100 times.