Luisa Iommarini, Ivana Kurelac, Mariantonietta Capristo, Maria Antonietta Calvaruso, Valentina Giorgio, Christian Bergamini, Anna Ghelli, Patrizia Nanni, Carla De Giovanni, Valerio Carelli, Romana Fato, Pier Luigi Lollini, Michela Rugolo, Giuseppe Gasparre, Anna Maria Porcelli. Hum Mol Genet 2014
Times Cited: 64
Times Cited: 64
Times Cited
Times Co-cited
Similarity
Respiratory complex I is essential to induce a Warburg profile in mitochondria-defective tumor cells.
Claudia Calabrese, Luisa Iommarini, Ivana Kurelac, Maria Antonietta Calvaruso, Mariantonietta Capristo, Pier-Luigi Lollini, Patrizia Nanni, Christian Bergamini, Giordano Nicoletti, Carla De Giovanni,[...]. Cancer Metab 2013
Claudia Calabrese, Luisa Iommarini, Ivana Kurelac, Maria Antonietta Calvaruso, Mariantonietta Capristo, Pier-Luigi Lollini, Patrizia Nanni, Christian Bergamini, Giordano Nicoletti, Carla De Giovanni,[...]. Cancer Metab 2013
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ROS-generating mitochondrial DNA mutations can regulate tumor cell metastasis.
Kaori Ishikawa, Keizo Takenaga, Miho Akimoto, Nobuko Koshikawa, Aya Yamaguchi, Hirotake Imanishi, Kazuto Nakada, Yoshio Honma, Jun-Ichi Hayashi. Science 2008
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A mutation threshold distinguishes the antitumorigenic effects of the mitochondrial gene MTND1, an oncojanus function.
Giuseppe Gasparre, Ivana Kurelac, Mariantonietta Capristo, Luisa Iommarini, Anna Ghelli, Claudio Ceccarelli, Giordano Nicoletti, Patrizia Nanni, Carla De Giovanni, Katia Scotlandi,[...]. Cancer Res 2011
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Complex I impairment in mitochondrial diseases and cancer: parallel roads leading to different outcomes.
Luisa Iommarini, Maria Antonietta Calvaruso, Ivana Kurelac, Giuseppe Gasparre, Anna Maria Porcelli. Int J Biochem Cell Biol 2013
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Mitochondrial respiratory complex I dysfunction promotes tumorigenesis through ROS alteration and AKT activation.
Lokendra Kumar Sharma, Hezhi Fang, Jiangtao Liu, Rasika Vartak, Janice Deng, Yidong Bai. Hum Mol Genet 2011
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The genetic and metabolic signature of oncocytic transformation implicates HIF1alpha destabilization.
Anna Maria Porcelli, Anna Ghelli, Claudio Ceccarelli, Martin Lang, Giovanna Cenacchi, Mariantonietta Capristo, Lucia Fiammetta Pennisi, Isabella Morra, Enrica Ciccarelli, Antonio Melcarne,[...]. Hum Mol Genet 2010
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Disruptive mitochondrial DNA mutations in complex I subunits are markers of oncocytic phenotype in thyroid tumors.
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A heteroplasmic, not homoplasmic, mitochondrial DNA mutation promotes tumorigenesis via alteration in reactive oxygen species generation and apoptosis.
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Origins and functional consequences of somatic mitochondrial DNA mutations in human cancer.
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Metformin inhibits mitochondrial complex I of cancer cells to reduce tumorigenesis.
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Spectrum of somatic mitochondrial mutations in five cancers.
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mtDNA mutations increase tumorigenicity in prostate cancer.
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Somatic complex I disruptive mitochondrial DNA mutations are modifiers of tumorigenesis that correlate with low genomic instability in pituitary adenomas.
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Somatic mutations of the mitochondrial genome in human colorectal tumours.
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Loss of complex I due to mitochondrial DNA mutations in renal oncocytoma.
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Succinate links TCA cycle dysfunction to oncogenesis by inhibiting HIF-alpha prolyl hydroxylase.
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Relevance of mitochondrial genetics and metabolism in cancer development.
Giuseppe Gasparre, Anna Maria Porcelli, Giorgio Lenaz, Giovanni Romeo. Cold Spring Harb Perspect Biol 2013
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Clonal expansion of mutated mitochondrial DNA is associated with tumor formation and complex I deficiency in the benign renal oncocytoma.
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Positive contribution of pathogenic mutations in the mitochondrial genome to the promotion of cancer by prevention from apoptosis.
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Mitochondrial metabolism and ROS generation are essential for Kras-mediated tumorigenicity.
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HmtDB, a genomic resource for mitochondrion-based human variability studies.
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Defective oxidative phosphorylation in thyroid oncocytic carcinoma is associated with pathogenic mitochondrial DNA mutations affecting complexes I and III.
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Somatic mitochondrial DNA mutations in cancer escape purifying selection and high pathogenicity mutations lead to the oncocytic phenotype: pathogenicity analysis of reported somatic mtDNA mutations in tumors.
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Human cells lacking mtDNA: repopulation with exogenous mitochondria by complementation.
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A mitochondrial switch promotes tumor metastasis.
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Mitochondrial DNA mutation in serous ovarian cancer: implications for mitochondria-coded genes in chemoresistance.
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Mitochondrial genome acquisition restores respiratory function and tumorigenic potential of cancer cells without mitochondrial DNA.
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High frequency of homoplasmic mitochondrial DNA mutations in human tumors can be explained without selection.
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Heteroplasmic mitochondrial DNA mutations in normal and tumour cells.
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Waves of gene regulation suppress and then restore oxidative phosphorylation in cancer cells.
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An Essential Role of the Mitochondrial Electron Transport Chain in Cell Proliferation Is to Enable Aspartate Synthesis.
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Leukemic IDH1 and IDH2 mutations result in a hypermethylation phenotype, disrupt TET2 function, and impair hematopoietic differentiation.
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Facile detection of mitochondrial DNA mutations in tumors and bodily fluids.
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BAY 87-2243, a highly potent and selective inhibitor of hypoxia-induced gene activation has antitumor activities by inhibition of mitochondrial complex I.
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Frequency and phenotypic implications of mitochondrial DNA mutations in human squamous cell cancers of the head and neck.
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Mitochondrial DNA mutations regulate metastasis of human breast cancer cells.
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Co-cited is the co-citation frequency, indicating how many articles cite the article together with the query article. Similarity is the co-citation as percentage of the times cited of the query article or the article in the search results, whichever is the lowest. These numbers are calculated for the last 100 citations when articles are cited more than 100 times.