Susan Price, Pamela A Shaw, Amy Seitz, Gyan Joshi, Joie Davis, Julie E Niemela, Katie Perkins, Ronald L Hornung, Les Folio, Philip S Rosenberg, Jennifer M Puck, Amy P Hsu, Bernice Lo, Stefania Pittaluga, Elaine S Jaffe, Thomas A Fleisher, V Koneti Rao, Michael J Lenardo. Blood 2014
Times Cited: 125
Times Cited: 125
Times Cited
Times Co-cited
Similarity
Revised diagnostic criteria and classification for the autoimmune lymphoproliferative syndrome (ALPS): report from the 2009 NIH International Workshop.
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The development of lymphomas in families with autoimmune lymphoproliferative syndrome with germline Fas mutations and defective lymphocyte apoptosis.
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Dominant interfering Fas gene mutations impair apoptosis in a human autoimmune lymphoproliferative syndrome.
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A survey of 90 patients with autoimmune lymphoproliferative syndrome related to TNFRSF6 mutation.
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How I treat autoimmune lymphoproliferative syndrome.
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Autosomal dominant immune dysregulation syndrome in humans with CTLA4 mutations.
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Immune dysregulation in human subjects with heterozygous germline mutations in CTLA4.
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AUTOIMMUNE DISEASE. Patients with LRBA deficiency show CTLA4 loss and immune dysregulation responsive to abatacept therapy.
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Autoimmune lymphoproliferative syndrome with somatic Fas mutations.
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Early-onset lymphoproliferation and autoimmunity caused by germline STAT3 gain-of-function mutations.
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Using biomarkers to predict the presence of FAS mutations in patients with features of the autoimmune lymphoproliferative syndrome.
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Somatic FAS mutations are common in patients with genetically undefined autoimmune lymphoproliferative syndrome.
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Autoimmune lymphoproliferative syndrome: an update and review of the literature.
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Treatment with sirolimus results in complete responses in patients with autoimmune lymphoproliferative syndrome.
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Sirolimus is effective in relapsed/refractory autoimmune cytopenias: results of a prospective multi-institutional trial.
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Inherited human Caspase 10 mutations underlie defective lymphocyte and dendritic cell apoptosis in autoimmune lymphoproliferative syndrome type II.
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Dominant-activating germline mutations in the gene encoding the PI(3)K catalytic subunit p110δ result in T cell senescence and human immunodeficiency.
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FAS-L, IL-10, and double-negative CD4- CD8- TCR alpha/beta+ T cells are reliable markers of autoimmune lymphoproliferative syndrome (ALPS) associated with FAS loss of function.
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New advances in the diagnosis and treatment of autoimmune lymphoproliferative syndrome.
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A homozygous Fas ligand gene mutation in a patient causes a new type of autoimmune lymphoproliferative syndrome.
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NRAS mutation causes a human autoimmune lymphoproliferative syndrome.
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Phosphoinositide 3-kinase δ gene mutation predisposes to respiratory infection and airway damage.
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10
Genetic alterations in caspase-10 may be causative or protective in autoimmune lymphoproliferative syndrome.
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Evolution of disease activity and biomarkers on and off rapamycin in 28 patients with autoimmune lymphoproliferative syndrome.
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Autoimmune lymphoproliferative syndrome caused by a homozygous null FAS ligand (FASLG) mutation.
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Autoimmune lymphoproliferative syndrome: more than a FAScinating disease.
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Use of mycophenolate mofetil for chronic, refractory immune cytopenias in children with autoimmune lymphoproliferative syndrome.
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10
Co-cited is the co-citation frequency, indicating how many articles cite the article together with the query article. Similarity is the co-citation as percentage of the times cited of the query article or the article in the search results, whichever is the lowest. These numbers are calculated for the last 100 citations when articles are cited more than 100 times.