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Times Cited: 859
Times Cited
Times Co-cited
Similarity
De novo mutations in schizophrenia implicate synaptic networks.
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Common schizophrenia alleles are enriched in mutation-intolerant genes and in regions under strong background selection.
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Contribution of copy number variants to schizophrenia from a genome-wide study of 41,321 subjects.
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Gene expression elucidates functional impact of polygenic risk for schizophrenia.
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Increased burden of ultra-rare protein-altering variants among 4,877 individuals with schizophrenia.
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Common polygenic variation contributes to risk of schizophrenia and bipolar disorder.
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De novo CNV analysis implicates specific abnormalities of postsynaptic signalling complexes in the pathogenesis of schizophrenia.
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Genetic relationship between five psychiatric disorders estimated from genome-wide SNPs.
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Rare loss-of-function variants in SETD1A are associated with schizophrenia and developmental disorders.
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Genome-wide association analysis identifies 30 new susceptibility loci for schizophrenia.
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The contribution of rare variants to risk of schizophrenia in individuals with and without intellectual disability.
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Genome-wide association study identifies 30 loci associated with bipolar disorder.
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Psychiatric genome-wide association study analyses implicate neuronal, immune and histone pathways.
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CNVs: harbingers of a rare variant revolution in psychiatric genetics.
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Genome-wide association analyses identify 44 risk variants and refine the genetic architecture of major depression.
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Estimating the proportion of variation in susceptibility to schizophrenia captured by common SNPs.
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Schizophrenia risk from complex variation of complement component 4.
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Fast and accurate short read alignment with Burrows-Wheeler transform.
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Synaptic, transcriptional and chromatin genes disrupted in autism.
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Shared molecular neuropathology across major psychiatric disorders parallels polygenic overlap.
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Genetic identification of brain cell types underlying schizophrenia.
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Modelling schizophrenia using human induced pluripotent stem cells.
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Developmental and genetic regulation of the human cortex transcriptome illuminate schizophrenia pathogenesis.
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Gene expression imputation across multiple brain regions provides insights into schizophrenia risk.
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Prefrontal cortical dendritic spine pathology in schizophrenia and bipolar disorder.
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From FastQ data to high confidence variant calls: the Genome Analysis Toolkit best practices pipeline.
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Co-cited is the co-citation frequency, indicating how many articles cite the article together with the query article. Similarity is the co-citation as percentage of the times cited of the query article or the article in the search results, whichever is the lowest. These numbers are calculated for the last 100 citations when articles are cited more than 100 times.