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Times Cited: 914
Times Cited: 914
Times Cited
Times Co-cited
Similarity
Fate mapping analysis reveals that adult microglia derive from primitive macrophages.
Florent Ginhoux, Melanie Greter, Marylene Leboeuf, Sayan Nandi, Peter See, Solen Gokhan, Mark F Mehler, Simon J Conway, Lai Guan Ng, E Richard Stanley,[...]. Science 2010
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Microglia promote learning-dependent synapse formation through brain-derived neurotrophic factor.
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A Unique Microglia Type Associated with Restricting Development of Alzheimer's Disease.
Hadas Keren-Shaul, Amit Spinrad, Assaf Weiner, Orit Matcovitch-Natan, Raz Dvir-Szternfeld, Tyler K Ulland, Eyal David, Kuti Baruch, David Lara-Astaiso, Beata Toth,[...]. Cell 2017
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Neurotoxic reactive astrocytes are induced by activated microglia.
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Synaptic pruning by microglia is necessary for normal brain development.
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Resting microglial cells are highly dynamic surveillants of brain parenchyma in vivo.
Axel Nimmerjahn, Frank Kirchhoff, Fritjof Helmchen. Science 2005
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Microglia sculpt postnatal neural circuits in an activity and complement-dependent manner.
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Colony-stimulating factor 1 receptor inhibition prevents microglial plaque association and improves cognition in 3xTg-AD mice.
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New tools for studying microglia in the mouse and human CNS.
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Sustained microglial depletion with CSF1R inhibitor impairs parenchymal plaque development in an Alzheimer's disease model.
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Genetic Cell Ablation Reveals Clusters of Local Self-Renewing Microglia in the Mammalian Central Nervous System.
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CSF1R inhibition by a small-molecule inhibitor is not microglia specific; affecting hematopoiesis and the function of macrophages.
Fengyang Lei, Naiwen Cui, Chengxin Zhou, James Chodosh, Demetrios G Vavvas, Eleftherios I Paschalis. Proc Natl Acad Sci U S A 2020
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Single-Cell RNA Sequencing of Microglia throughout the Mouse Lifespan and in the Injured Brain Reveals Complex Cell-State Changes.
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Repopulated microglia are solely derived from the proliferation of residual microglia after acute depletion.
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Negative feedback control of neuronal activity by microglia.
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Absence of colony stimulation factor-1 receptor results in loss of microglia, disrupted brain development and olfactory deficits.
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Microglia emerge from erythromyeloid precursors via Pu.1- and Irf8-dependent pathways.
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Replacement of microglia in the aged brain reverses cognitive, synaptic, and neuronal deficits in mice.
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Microglia Biology: One Century of Evolving Concepts.
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Microglial brain region-dependent diversity and selective regional sensitivities to aging.
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ATP mediates rapid microglial response to local brain injury in vivo.
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Local self-renewal can sustain CNS microglia maintenance and function throughout adult life.
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Identification of a unique TGF-β-dependent molecular and functional signature in microglia.
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Origin, fate and dynamics of macrophages at central nervous system interfaces.
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The TREM2-APOE Pathway Drives the Transcriptional Phenotype of Dysfunctional Microglia in Neurodegenerative Diseases.
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Eliminating microglia in Alzheimer's mice prevents neuronal loss without modulating amyloid-β pathology.
Elizabeth E Spangenberg, Rafael J Lee, Allison R Najafi, Rachel A Rice, Monica R P Elmore, Mathew Blurton-Jones, Brian L West, Kim N Green. Brain 2016
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Repopulating Microglia Promote Brain Repair in an IL-6-Dependent Manner.
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Microglia contact induces synapse formation in developing somatosensory cortex.
Akiko Miyamoto, Hiroaki Wake, Ayako Wendy Ishikawa, Kei Eto, Keisuke Shibata, Hideji Murakoshi, Schuichi Koizumi, Andrew J Moorhouse, Yumiko Yoshimura, Junichi Nabekura. Nat Commun 2016
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Developmental Heterogeneity of Microglia and Brain Myeloid Cells Revealed by Deep Single-Cell RNA Sequencing.
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Coupled Proliferation and Apoptosis Maintain the Rapid Turnover of Microglia in the Adult Brain.
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Analysis of fractalkine receptor CX(3)CR1 function by targeted deletion and green fluorescent protein reporter gene insertion.
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High-Dimensional Single-Cell Mapping of Central Nervous System Immune Cells Reveals Distinct Myeloid Subsets in Health, Aging, and Disease.
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The classical complement cascade mediates CNS synapse elimination.
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Microglia development follows a stepwise program to regulate brain homeostasis.
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To Kill a Microglia: A Case for CSF1R Inhibitors.
Kim N Green, Joshua D Crapser, Lindsay A Hohsfield. Trends Immunol 2020
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Microglia in the adult brain arise from Ly-6ChiCCR2+ monocytes only under defined host conditions.
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Peripherally derived macrophages can engraft the brain independent of irradiation and maintain an identity distinct from microglia.
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A new fate mapping system reveals context-dependent random or clonal expansion of microglia.
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Microglia: active sensor and versatile effector cells in the normal and pathologic brain.
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Microglia contribute to normal myelinogenesis and to oligodendrocyte progenitor maintenance during adulthood.
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Microglia turnover with aging and in an Alzheimer's model via long-term in vivo single-cell imaging.
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Tissue-resident macrophages originate from yolk-sac-derived erythro-myeloid progenitors.
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Fate mapping reveals origins and dynamics of monocytes and tissue macrophages under homeostasis.
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Microglia Are Irrelevant for Neuronal Degeneration and Axon Regeneration after Acute Injury.
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Microglia in Physiology and Disease.
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Emerging Roles for CSF-1 Receptor and its Ligands in the Nervous System.
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Microglia protect against brain injury and their selective elimination dysregulates neuronal network activity after stroke.
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IL-34 is a tissue-restricted ligand of CSF1R required for the development of Langerhans cells and microglia.
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Microglia modulate wiring of the embryonic forebrain.
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Microglia derive from progenitors, originating from the yolk sac, and which proliferate in the brain.
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Co-cited is the co-citation frequency, indicating how many articles cite the article together with the query article. Similarity is the co-citation as percentage of the times cited of the query article or the article in the search results, whichever is the lowest. These numbers are calculated for the last 100 citations when articles are cited more than 100 times.