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Times Cited: 698
Times Cited: 698
Times Cited
Times Co-cited
Similarity
CSF-1R inhibition alters macrophage polarization and blocks glioma progression.
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Tumour-associated macrophages as treatment targets in oncology.
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Targeting both tumour-associated CXCR2+ neutrophils and CCR2+ macrophages disrupts myeloid recruitment and improves chemotherapeutic responses in pancreatic ductal adenocarcinoma.
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PD-1 expression by tumour-associated macrophages inhibits phagocytosis and tumour immunity.
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Targeting tumour-associated macrophages with CCR2 inhibition in combination with FOLFIRINOX in patients with borderline resectable and locally advanced pancreatic cancer: a single-centre, open-label, dose-finding, non-randomised, phase 1b trial.
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Targeting tumor-infiltrating macrophages decreases tumor-initiating cells, relieves immunosuppression, and improves chemotherapeutic responses.
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Class IIa HDAC inhibition reduces breast tumours and metastases through anti-tumour macrophages.
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Cancer-Associated Fibroblasts Neutralize the Anti-tumor Effect of CSF1 Receptor Blockade by Inducing PMN-MDSC Infiltration of Tumors.
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Inflammatory monocyte mobilization decreases patient survival in pancreatic cancer: a role for targeting the CCL2/CCR2 axis.
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Macrophages impede CD8 T cells from reaching tumor cells and limit the efficacy of anti-PD-1 treatment.
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Macrophage regulation of tumor responses to anticancer therapies.
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CSF1R inhibition delays cervical and mammary tumor growth in murine models by attenuating the turnover of tumor-associated macrophages and enhancing infiltration by CD8+ T cells.
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Overcoming resistance to checkpoint blockade therapy by targeting PI3Kγ in myeloid cells.
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Coordinated regulation of myeloid cells by tumours.
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Perivascular M2 Macrophages Stimulate Tumor Relapse after Chemotherapy.
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Primary, Adaptive, and Acquired Resistance to Cancer Immunotherapy.
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The CD47-signal regulatory protein alpha (SIRPa) interaction is a therapeutic target for human solid tumors.
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Projecting cancer incidence and deaths to 2030: the unexpected burden of thyroid, liver, and pancreas cancers in the United States.
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Mutations Associated with Acquired Resistance to PD-1 Blockade in Melanoma.
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Myeloid cells are required for PD-1/PD-L1 checkpoint activation and the establishment of an immunosuppressive environment in pancreatic cancer.
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CSF1R+ Macrophages Sustain Pancreatic Tumor Growth through T Cell Suppression and Maintenance of Key Gene Programs that Define the Squamous Subtype.
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Reprogramming Tumor-Associated Macrophages by Antibody Targeting Inhibits Cancer Progression and Metastasis.
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Tumor-associated macrophages in tumor metastasis: biological roles and clinical therapeutic applications.
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8
Co-cited is the co-citation frequency, indicating how many articles cite the article together with the query article. Similarity is the co-citation as percentage of the times cited of the query article or the article in the search results, whichever is the lowest. These numbers are calculated for the last 100 citations when articles are cited more than 100 times.