A citation-based method for searching scientific literature

Zi-Qiang Zhou, Sara Ota, Chuxia Deng, Haruhiko Akiyama, Peter J Hurlin. Hum Mol Genet 2015
Times Cited: 17







List of co-cited articles
199 articles co-cited >1



Times Cited
  Times     Co-cited
Similarity


Constitutive activation of MEK1 in chondrocytes causes Stat1-independent achondroplasia-like dwarfism and rescues the Fgfr3-deficient mouse phenotype.
Shunichi Murakami, Gener Balmes, Sandra McKinney, Zhaoping Zhang, David Givol, Benoit de Crombrugghe. Genes Dev 2004
200
47


The transcription factor Sox9 has essential roles in successive steps of the chondrocyte differentiation pathway and is required for expression of Sox5 and Sox6.
Haruhiko Akiyama, Marie-Christine Chaboissier, James F Martin, Andreas Schedl, Benoit de Crombrugghe. Genes Dev 2002
29

Sox9 directs hypertrophic maturation and blocks osteoblast differentiation of growth plate chondrocytes.
Peter Dy, Weihuan Wang, Pallavi Bhattaram, Qiuqing Wang, Lai Wang, R Tracy Ballock, Véronique Lefebvre. Dev Cell 2012
215
29

Development of the endochondral skeleton.
Fanxin Long, David M Ornitz. Cold Spring Harb Perspect Biol 2013
299
29

Skeletal overgrowth and deafness in mice lacking fibroblast growth factor receptor 3.
J S Colvin, B A Bohne, G W Harding, D G McEwen, D M Ornitz. Nat Genet 1996
688
29

Intermittent PTH (1-34) injection rescues the retarded skeletal development and postnatal lethality of mice mimicking human achondroplasia and thanatophoric dysplasia.
Yangli Xie, Nan Su, Min Jin, Huabing Qi, Junbao Yang, Can Li, Xiaolan Du, Fengtao Luo, Bo Chen, Yue Shen,[...]. Hum Mol Genet 2012
33
29

A neonatal lethal mutation in FGFR3 uncouples proliferation and differentiation of growth plate chondrocytes in embryos.
T Iwata, L Chen, C Li, D A Ovchinnikov, R R Behringer, C A Francomano, C X Deng. Hum Mol Genet 2000
157
29

Fibroblast growth factor receptor 3 is a negative regulator of bone growth.
C Deng, A Wynshaw-Boris, F Zhou, A Kuo, P Leder. Cell 1996
837
29

Mutations in the transmembrane domain of FGFR3 cause the most common genetic form of dwarfism, achondroplasia.
R Shiang, L M Thompson, Y Z Zhu, D M Church, T J Fielder, M Bocian, S T Winokur, J J Wasmuth. Cell 1994
921
29

An activating Fgfr3 mutation affects trabecular bone formation via a paracrine mechanism during growth.
Emilie Mugniery, Romain Dacquin, Caroline Marty, Catherine Benoist-Lasselin, Marie-Christine de Vernejoul, Pierre Jurdic, Arnold Munnich, Valérie Geoffroy, Laurence Legeai-Mallet. Hum Mol Genet 2012
34
29


Runx2 and Runx3 are essential for chondrocyte maturation, and Runx2 regulates limb growth through induction of Indian hedgehog.
Carolina A Yoshida, Hiromitsu Yamamoto, Takashi Fujita, Tatsuya Furuichi, Kosei Ito, Ken-ichi Inoue, Kei Yamana, Akira Zanma, Kenji Takada, Yoshiaki Ito,[...]. Genes Dev 2004
404
23

Evaluation of the therapeutic potential of a CNP analog in a Fgfr3 mouse model recapitulating achondroplasia.
Florence Lorget, Nabil Kaci, Jeff Peng, Catherine Benoist-Lasselin, Emilie Mugniery, Todd Oppeneer, Dan J Wendt, Sean M Bell, Sherry Bullens, Stuart Bunting,[...]. Am J Hum Genet 2012
104
23



Hypertrophic chondrocytes can become osteoblasts and osteocytes in endochondral bone formation.
Liu Yang, Kwok Yeung Tsang, Hoi Ching Tang, Danny Chan, Kathryn S E Cheah. Proc Natl Acad Sci U S A 2014
357
23

Neutral endopeptidase-resistant C-type natriuretic peptide variant represents a new therapeutic approach for treatment of fibroblast growth factor receptor 3-related dwarfism.
Daniel J Wendt, Melita Dvorak-Ewell, Sherry Bullens, Florence Lorget, Sean M Bell, Jeff Peng, Sianna Castillo, Mika Aoyagi-Scharber, Charles A O'Neill, Pavel Krejci,[...]. J Pharmacol Exp Ther 2015
57
23

Chondrocytes transdifferentiate into osteoblasts in endochondral bone during development, postnatal growth and fracture healing in mice.
Xin Zhou, Klaus von der Mark, Stephen Henry, William Norton, Henry Adams, Benoit de Crombrugghe. PLoS Genet 2014
284
23

Sox9 sustains chondrocyte survival and hypertrophy in part through Pik3ca-Akt pathways.
Daisuke Ikegami, Haruhiko Akiyama, Akira Suzuki, Takashi Nakamura, Toru Nakano, Hideki Yoshikawa, Noriyuki Tsumaki. Development 2011
124
23


Analysis of STAT1 activation by six FGFR3 mutants associated with skeletal dysplasia undermines dominant role of STAT1 in FGFR3 signaling in cartilage.
Pavel Krejci, Lisa Salazar, Tamara A Kashiwada, Katarina Chlebova, Alena Salasova, Leslie Michels Thompson, Vitezslav Bryja, Alois Kozubik, William R Wilcox. PLoS One 2008
32
23

Gly369Cys mutation in mouse FGFR3 causes achondroplasia by affecting both chondrogenesis and osteogenesis.
L Chen, R Adar, X Yang, E O Monsonego, C Li, P V Hauschka, A Yayon, C X Deng. J Clin Invest 1999
189
23

Developmental regulation of the growth plate.
Henry M Kronenberg. Nature 2003
23

Campomelic dysplasia and autosomal sex reversal caused by mutations in an SRY-related gene.
J W Foster, M A Dominguez-Steglich, S Guioli, C Kwok, P A Weller, M Stevanović, J Weissenbach, S Mansour, I D Young, P N Goodfellow. Nature 1994
17

A far-upstream (-70 kb) enhancer mediates Sox9 auto-regulation in somatic tissues during development and adult regeneration.
Timothy J Mead, Qiuqing Wang, Pallavi Bhattaram, Peter Dy, Solomon Afelik, Jan Jensen, Véronique Lefebvre. Nucleic Acids Res 2013
59
17

Sox9 is required for cartilage formation.
W Bi, J M Deng, Z Zhang, R R Behringer, B de Crombrugghe. Nat Genet 1999
17

Autosomal sex reversal and campomelic dysplasia are caused by mutations in and around the SRY-related gene SOX9.
T Wagner, J Wirth, J Meyer, B Zabel, M Held, J Zimmer, J Pasantes, F D Bricarelli, J Keutel, E Hustert,[...]. Cell 1994
17

The transcription factors L-Sox5 and Sox6 are essential for cartilage formation.
P Smits, P Li, J Mandel, Z Zhang, J M Deng, R R Behringer, B de Crombrugghe, V Lefebvre. Dev Cell 2001
433
17

Parallel expression of Sox9 and Col2a1 in cells undergoing chondrogenesis.
Q Zhao, H Eberspaecher, V Lefebvre, B De Crombrugghe. Dev Dyn 1997
376
17



FGF signaling inhibits chondrocyte proliferation and regulates bone development through the STAT-1 pathway.
M Sahni, D C Ambrosetti, A Mansukhani, R Gertner, D Levy, C Basilico. Genes Dev 1999
280
17

Quantitation of chondrocyte performance in growth-plate cartilage during longitudinal bone growth.
E B Hunziker, R K Schenk, L M Cruz-Orive. J Bone Joint Surg Am 1987
275
17

Genetic inactivation of ERK1 and ERK2 in chondrocytes promotes bone growth and enlarges the spinal canal.
Arjun Sebastian, Takehiko Matsushita, Aya Kawanami, Susan Mackem, Gary E Landreth, Shunichi Murakami. J Orthop Res 2011
32
17

Skeletal overgrowth is mediated by deficiency in a specific isoform of fibroblast growth factor receptor 3.
Veraragavan P Eswarakumar, Joseph Schlessinger. Proc Natl Acad Sci U S A 2007
40
17

Fibroblast growth factor receptor 3 mutations promote apoptosis but do not alter chondrocyte proliferation in thanatophoric dysplasia.
L Legeai-Mallet, C Benoist-Lasselin, A L Delezoide, A Munnich, J Bonaventure. J Biol Chem 1998
115
17

Statin treatment rescues FGFR3 skeletal dysplasia phenotypes.
Akihiro Yamashita, Miho Morioka, Hiromi Kishi, Takeshi Kimura, Yasuhito Yahara, Minoru Okada, Kaori Fujita, Hideaki Sawai, Shiro Ikegawa, Noriyuki Tsumaki. Nature 2014
129
17



Coordination of chondrogenesis and osteogenesis by fibroblast growth factor 18.
Zhonghao Liu, Jingsong Xu, Jennifer S Colvin, David M Ornitz. Genes Dev 2002
338
17

Activating Fgfr3 Y367C mutation causes hearing loss and inner ear defect in a mouse model of chondrodysplasia.
Stéphanie Pannier, Vincent Couloigner, Nadia Messaddeq, Monique Elmaleh-Bergès, Arnold Munnich, Raymond Romand, Laurence Legeai-Mallet. Biochim Biophys Acta 2009
38
17

A subset of chondrogenic cells provides early mesenchymal progenitors in growing bones.
Noriaki Ono, Wanida Ono, Takashi Nagasawa, Henry M Kronenberg. Nat Cell Biol 2014
225
17

Gain-of-function mutation in FGFR3 in mice leads to decreased bone mass by affecting both osteoblastogenesis and osteoclastogenesis.
Nan Su, Qidi Sun, Can Li, Xiumin Lu, Huabing Qi, Siyu Chen, Jing Yang, Xiaolan Du, Ling Zhao, Qifen He,[...]. Hum Mol Genet 2010
64
17

FGFR3 mutation causes abnormal membranous ossification in achondroplasia.
Federico Di Rocco, Martin Biosse Duplan, Yann Heuzé, Nabil Kaci, Davide Komla-Ebri, Arnold Munnich, Emilie Mugniery, Catherine Benoist-Lasselin, Laurence Legeai-Mallet. Hum Mol Genet 2014
34
17

A mouse model for achondroplasia produced by targeting fibroblast growth factor receptor 3.
Y Wang, M K Spatz, K Kannan, H Hayk, A Avivi, M Gorivodsky, M Pines, A Yayon, P Lonai, D Givol. Proc Natl Acad Sci U S A 1999
167
17

Activation of Stat1 by mutant fibroblast growth-factor receptor in thanatophoric dysplasia type II dwarfism.
W C Su, M Kitagawa, N Xue, B Xie, S Garofalo, J Cho, C Deng, W A Horton, X Y Fu. Nature 1997
253
17

Common mutations in the fibroblast growth factor receptor 3 (FGFR 3) gene account for achondroplasia, hypochondroplasia, and thanatophoric dwarfism.
J Bonaventure, F Rousseau, L Legeai-Mallet, M Le Merrer, A Munnich, P Maroteaux. Am J Med Genet 1996
85
17

Meclozine promotes longitudinal skeletal growth in transgenic mice with achondroplasia carrying a gain-of-function mutation in the FGFR3 gene.
Masaki Matsushita, Satoru Hasegawa, Hiroshi Kitoh, Kensaku Mori, Bisei Ohkawara, Akihiro Yasoda, Akio Masuda, Naoki Ishiguro, Kinji Ohno. Endocrinology 2015
29
17



Co-cited is the co-citation frequency, indicating how many articles cite the article together with the query article. Similarity is the co-citation as percentage of the times cited of the query article or the article in the search results, whichever is the lowest. These numbers are calculated for the last 100 citations when articles are cited more than 100 times.