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Times Cited: 35
Times Cited: 35
Times Cited
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Extrachromosomal oncogene amplification drives tumour evolution and genetic heterogeneity.
Kristen M Turner, Viraj Deshpande, Doruk Beyter, Tomoyuki Koga, Jessica Rusert, Catherine Lee, Bin Li, Karen Arden, Bing Ren, David A Nathanson,[...]. Nature 2017
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Targeted therapy resistance mediated by dynamic regulation of extrachromosomal mutant EGFR DNA.
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Discordant inheritance of chromosomal and extrachromosomal DNA elements contributes to dynamic disease evolution in glioblastoma.
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Extrachromosomal circular DNA drives oncogenic genome remodeling in neuroblastoma.
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Functional Enhancers Shape Extrachromosomal Oncogene Amplifications.
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Circular DNA elements of chromosomal origin are common in healthy human somatic tissue.
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Inhibiting homologous recombination decreases extrachromosomal amplification but has no effect on intrachromosomal amplification in methotrexate-resistant colon cancer cells.
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Extrachromosomal circular DNA is common in yeast.
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MYC-containing double minutes in hematologic malignancies: evidence in favor of the episome model and exclusion of MYC as the target gene.
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Genomic organization and evolution of double minutes/homogeneously staining regions with MYC amplification in human cancer.
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Co-cited is the co-citation frequency, indicating how many articles cite the article together with the query article. Similarity is the co-citation as percentage of the times cited of the query article or the article in the search results, whichever is the lowest. These numbers are calculated for the last 100 citations when articles are cited more than 100 times.