Stephanie A Schnell, Alberto Ambesi-Impiombato, Marta Sanchez-Martin, Laura Belver, Luyao Xu, Yue Qin, Ryoichiro Kageyama, Adolfo A Ferrando. Blood 2015
Times Cited: 29
Times Cited: 29
Times Cited
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Similarity
Activating mutations of NOTCH1 in human T cell acute lymphoblastic leukemia.
Andrew P Weng, Adolfo A Ferrando, Woojoong Lee, John P Morris, Lewis B Silverman, Cheryll Sanchez-Irizarry, Stephen C Blacklow, A Thomas Look, Jon C Aster. Science 2004
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Mutational loss of PTEN induces resistance to NOTCH1 inhibition in T-cell leukemia.
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Gamma-secretase inhibitors reverse glucocorticoid resistance in T cell acute lymphoblastic leukemia.
Pedro J Real, Valeria Tosello, Teresa Palomero, Mireia Castillo, Eva Hernando, Elisa de Stanchina, Maria Luisa Sulis, Kelly Barnes, Catherine Sawai, Irene Homminga,[...]. Nat Med 2009
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NOTCH1 directly regulates c-MYC and activates a feed-forward-loop transcriptional network promoting leukemic cell growth.
Teresa Palomero, Wei Keat Lim, Duncan T Odom, Maria Luisa Sulis, Pedro J Real, Adam Margolin, Kelly C Barnes, Jennifer O'Neil, Donna Neuberg, Andrew P Weng,[...]. Proc Natl Acad Sci U S A 2006
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FBW7 mutations in leukemic cells mediate NOTCH pathway activation and resistance to gamma-secretase inhibitors.
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The SCFFBW7 ubiquitin ligase complex as a tumor suppressor in T cell leukemia.
Benjamin J Thompson, Silvia Buonamici, Maria Luisa Sulis, Teresa Palomero, Tomas Vilimas, Giuseppe Basso, Adolfo Ferrando, Iannis Aifantis. J Exp Med 2007
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c-Myc is an important direct target of Notch1 in T-cell acute lymphoblastic leukemia/lymphoma.
Andrew P Weng, John M Millholland, Yumi Yashiro-Ohtani, Marie Laure Arcangeli, Arthur Lau, Carol Wai, Cristina Del Bianco, Carlos G Rodriguez, Hong Sai, John Tobias,[...]. Genes Dev 2006
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Exclusive development of T cell neoplasms in mice transplanted with bone marrow expressing activated Notch alleles.
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A NOTCH1-driven MYC enhancer promotes T cell development, transformation and acute lymphoblastic leukemia.
Daniel Herranz, Alberto Ambesi-Impiombato, Teresa Palomero, Stephanie A Schnell, Laura Belver, Agnieszka A Wendorff, Luyao Xu, Mireia Castillo-Martin, David Llobet-Navás, Carlos Cordon-Cardo,[...]. Nat Med 2014
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High-level IGF1R expression is required for leukemia-initiating cell activity in T-ALL and is supported by Notch signaling.
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Metabolic reprogramming induces resistance to anti-NOTCH1 therapies in T cell acute lymphoblastic leukemia.
Daniel Herranz, Alberto Ambesi-Impiombato, Jessica Sudderth, Marta Sánchez-Martín, Laura Belver, Valeria Tosello, Luyao Xu, Agnieszka A Wendorff, Mireia Castillo, J Erika Haydu,[...]. Nat Med 2015
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Hes1 is a critical but context-dependent mediator of canonical Notch signaling in lymphocyte development and transformation.
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Genome-wide mapping and characterization of Notch-regulated long noncoding RNAs in acute leukemia.
Thomas Trimarchi, Erhan Bilal, Panagiotis Ntziachristos, Giulia Fabbri, Riccardo Dalla-Favera, Aristotelis Tsirigos, Iannis Aifantis. Cell 2014
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Long-range enhancer activity determines Myc sensitivity to Notch inhibitors in T cell leukemia.
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Notch signals positively regulate activity of the mTOR pathway in T-cell acute lymphoblastic leukemia.
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Notch/gamma-secretase inhibition turns proliferative cells in intestinal crypts and adenomas into goblet cells.
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Therapeutic antibody targeting of individual Notch receptors.
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CSL-MAML-dependent Notch1 signaling controls T lineage-specific IL-7R{alpha} gene expression in early human thymopoiesis and leukemia.
Sara González-García, Marina García-Peydró, Enrique Martín-Gayo, Esteban Ballestar, Manel Esteller, Rafael Bornstein, José Luis de la Pompa, Adolfo A Ferrando, María L Toribio. J Exp Med 2009
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Notch1 contributes to mouse T-cell leukemia by directly inducing the expression of c-myc.
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TAN-1, the human homolog of the Drosophila notch gene, is broken by chromosomal translocations in T lymphoblastic neoplasms.
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The ubiquitin ligase FBXW7 modulates leukemia-initiating cell activity by regulating MYC stability.
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NOTCH1-RBPJ complexes drive target gene expression through dynamic interactions with superenhancers.
Hongfang Wang, Chongzhi Zang, Len Taing, Kelly L Arnett, Yinling Joey Wong, Warren S Pear, Stephen C Blacklow, X Shirley Liu, Jon C Aster. Proc Natl Acad Sci U S A 2014
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Therapeutic antibody targeting of Notch1 in T-acute lymphoblastic leukemia xenografts.
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The genetic basis of early T-cell precursor acute lymphoblastic leukaemia.
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Leukemia-associated NOTCH1 alleles are weak tumor initiators but accelerate K-ras-initiated leukemia.
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Complementary genomic screens identify SERCA as a therapeutic target in NOTCH1 mutated cancer.
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Leukemia-associated mutations within the NOTCH1 heterodimerization domain fall into at least two distinct mechanistic classes.
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NOTCH1 extracellular juxtamembrane expansion mutations in T-ALL.
Maria Luisa Sulis, Odette Williams, Teresa Palomero, Valeria Tosello, Sasikala Pallikuppam, Pedro J Real, Kelly Barnes, Linda Zuurbier, Jules P Meijerink, Adolfo A Ferrando. Blood 2008
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Direct inhibition of the NOTCH transcription factor complex.
Raymond E Moellering, Melanie Cornejo, Tina N Davis, Cristina Del Bianco, Jon C Aster, Stephen C Blacklow, Andrew L Kung, D Gary Gilliland, Gregory L Verdine, James E Bradner. Nature 2009
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NOTCH is a key regulator of human T-cell acute leukemia initiating cell activity.
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Delta-like 4 is the essential, nonredundant ligand for Notch1 during thymic T cell lineage commitment.
Ute Koch, Emma Fiorini, Rui Benedito, Valerie Besseyrias, Karin Schuster-Gossler, Michel Pierres, Nancy R Manley, Antonio Duarte, H Robson Macdonald, Freddy Radtke. J Exp Med 2008
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Core transcriptional regulatory circuit controlled by the TAL1 complex in human T cell acute lymphoblastic leukemia.
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The Notch/Hes1 pathway sustains NF-κB activation through CYLD repression in T cell leukemia.
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Direct reversal of glucocorticoid resistance by AKT inhibition in acute lymphoblastic leukemia.
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Gene expression signatures define novel oncogenic pathways in T cell acute lymphoblastic leukemia.
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Molecular pathogenesis and targeted therapies for NOTCH1-induced T-cell acute lymphoblastic leukemia.
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Targeting the NF-kappaB signaling pathway in Notch1-induced T-cell leukemia.
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Modulation of notch processing by gamma-secretase inhibitors causes intestinal goblet cell metaplasia and induction of genes known to specify gut secretory lineage differentiation.
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DLL4 regulates NOTCH signaling and growth of T acute lymphoblastic leukemia cells in NOD/SCID mice.
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Whole-genome sequencing identifies recurrent mutations in chronic lymphocytic leukaemia.
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Analysis of the chronic lymphocytic leukemia coding genome: role of NOTCH1 mutational activation.
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NOTCH1/FBXW7 mutation identifies a large subgroup with favorable outcome in adult T-cell acute lymphoblastic leukemia (T-ALL): a Group for Research on Adult Acute Lymphoblastic Leukemia (GRAALL) study.
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CUTLL1, a novel human T-cell lymphoma cell line with t(7;9) rearrangement, aberrant NOTCH1 activation and high sensitivity to gamma-secretase inhibitors.
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Characterization of Notch1 antibodies that inhibit signaling of both normal and mutated Notch1 receptors.
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Mutations of NOTCH1 are an independent predictor of survival in chronic lymphocytic leukemia.
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An epigenetic mechanism of resistance to targeted therapy in T cell acute lymphoblastic leukemia.
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Structure of the Notch1-negative regulatory region: implications for normal activation and pathogenic signaling in T-ALL.
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A Phase 1 study of the novel gamma-secretase inhibitor PF-03084014 in patients with T-cell acute lymphoblastic leukemia and T-cell lymphoblastic lymphoma.
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ChIP-on-chip significance analysis reveals large-scale binding and regulation by human transcription factor oncogenes.
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Co-cited is the co-citation frequency, indicating how many articles cite the article together with the query article. Similarity is the co-citation as percentage of the times cited of the query article or the article in the search results, whichever is the lowest. These numbers are calculated for the last 100 citations when articles are cited more than 100 times.