Xiong Ji, Daniel B Dadon, Benjamin E Powell, Zi Peng Fan, Diego Borges-Rivera, Sigal Shachar, Abraham S Weintraub, Denes Hnisz, Gianluca Pegoraro, Tong Ihn Lee, Tom Misteli, Rudolf Jaenisch, Richard A Young. Cell Stem Cell 2016
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Times Cited: 228
Times Cited
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Similarity
Topological domains in mammalian genomes identified by analysis of chromatin interactions.
Jesse R Dixon, Siddarth Selvaraj, Feng Yue, Audrey Kim, Yan Li, Yin Shen, Ming Hu, Jun S Liu, Bing Ren. Nature 2012
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A 3D map of the human genome at kilobase resolution reveals principles of chromatin looping.
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Control of cell identity genes occurs in insulated neighborhoods in mammalian chromosomes.
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Comprehensive mapping of long-range interactions reveals folding principles of the human genome.
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Disruptions of topological chromatin domains cause pathogenic rewiring of gene-enhancer interactions.
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Spatial partitioning of the regulatory landscape of the X-inactivation centre.
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Chromatin architecture reorganization during stem cell differentiation.
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Activation of proto-oncogenes by disruption of chromosome neighborhoods.
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Targeted Degradation of CTCF Decouples Local Insulation of Chromosome Domains from Genomic Compartmentalization.
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CTCF-Mediated Human 3D Genome Architecture Reveals Chromatin Topology for Transcription.
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Chromatin extrusion explains key features of loop and domain formation in wild-type and engineered genomes.
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Insulator dysfunction and oncogene activation in IDH mutant gliomas.
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Simple combinations of lineage-determining transcription factors prime cis-regulatory elements required for macrophage and B cell identities.
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Three-dimensional folding and functional organization principles of the Drosophila genome.
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YY1 Is a Structural Regulator of Enhancer-Promoter Loops.
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Mediator and cohesin connect gene expression and chromatin architecture.
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Comparative Hi-C reveals that CTCF underlies evolution of chromosomal domain architecture.
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BEDTools: a flexible suite of utilities for comparing genomic features.
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Moderated estimation of fold change and dispersion for RNA-seq data with DESeq2.
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CTCF: an architectural protein bridging genome topology and function.
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Formation of new chromatin domains determines pathogenicity of genomic duplications.
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CTCF/cohesin-binding sites are frequently mutated in cancer.
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Super-enhancers in the control of cell identity and disease.
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Super-resolution chromatin tracing reveals domains and cooperative interactions in single cells.
Bogdan Bintu, Leslie J Mateo, Jun-Han Su, Nicholas A Sinnott-Armstrong, Mirae Parker, Seon Kinrot, Kei Yamaya, Alistair N Boettiger, Xiaowei Zhuang. Science 2018
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YY1 and CTCF orchestrate a 3D chromatin looping switch during early neural lineage commitment.
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Topologically associating domains are stable units of replication-timing regulation.
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Co-cited is the co-citation frequency, indicating how many articles cite the article together with the query article. Similarity is the co-citation as percentage of the times cited of the query article or the article in the search results, whichever is the lowest. These numbers are calculated for the last 100 citations when articles are cited more than 100 times.