Marcello Venzi, François David, Joachim Bellet, Anna Cavaccini, Cristiano Bombardi, Vincenzo Crunelli, Giuseppe Di Giovanni. Neuropharmacology 2016
Times Cited: 38
Times Cited: 38
Times Cited
Times Co-cited
Similarity
New therapeutic opportunities for 5-HT2C receptor ligands in neuropsychiatric disorders.
Giuseppe Di Giovanni, Philippe De Deurwaerdère. Pharmacol Ther 2016
Giuseppe Di Giovanni, Philippe De Deurwaerdère. Pharmacol Ther 2016
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Role(s) of the 5-HT2C receptor in the development of maximal dentate activation in the hippocampus of anesthetized rats.
Gergely Orban, Cristiano Bombardi, Antonella Marino Gammazza, Roberto Colangeli, Massimo Pierucci, Cristoforo Pomara, Mauro Pessia, Fabio Bucchieri, Arcangelo Benigno, Ilse Smolders,[...]. CNS Neurosci Ther 2014
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Eating disorder and epilepsy in mice lacking 5-HT2c serotonin receptors.
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Central serotonin-2A (5-HT2A) receptor dysfunction in depression and epilepsy: the missing link?
Bruno P Guiard, Giuseppe Di Giovanni. Front Pharmacol 2015
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Sound-induced seizures in serotonin 5-HT2c receptor mutant mice.
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Serotonergic 5-HT2C receptors as a potential therapeutic target for the design antiepileptic drugs.
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Lorcaserin, a novel selective human 5-hydroxytryptamine2C agonist: in vitro and in vivo pharmacological characterization.
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Serotonergic modulation of the activity of mesencephalic dopaminergic systems: Therapeutic implications.
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Clemizole and modulators of serotonin signalling suppress seizures in Dravet syndrome.
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High dose of 8-OH-DPAT decreases maximal dentate gyrus activation and facilitates granular cell plasticity in vivo.
Gergely Orban, Massimo Pierucci, Arcangelo Benigno, Mauro Pessia, Salvatore Galati, Mario Valentino, Richard Muscat, Giuseppe Di Giovanni. Exp Brain Res 2013
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Enhanced tonic GABAA inhibition in typical absence epilepsy.
David W Cope, Giuseppe Di Giovanni, Sarah J Fyson, Gergely Orbán, Adam C Errington, Magor L Lorincz, Timothy M Gould, David A Carter, Vincenzo Crunelli. Nat Med 2009
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5-HT2C receptors inhibit and 5-HT1A receptors activate the generation of spike-wave discharges in a genetic rat model of absence epilepsy.
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Studies on the role of 5-HT2C and 5-HT2B receptors in regulating generalised seizure threshold in rodents.
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RNA-editing of the 5-HT(2C) receptor alters agonist-receptor-effector coupling specificity.
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Regulation of serotonin-2C receptor G-protein coupling by RNA editing.
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SB 242084, a selective and brain penetrant 5-HT2C receptor antagonist.
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Directly Observable Behavioral Effects of Lorcaserin in Rats.
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Monoaminergic Mechanisms in Epilepsy May Offer Innovative Therapeutic Opportunity for Monoaminergic Multi-Target Drugs.
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Immunohistochemical localisation of the 5-HT2C receptor protein in the rat CNS.
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Lorcaserin therapy for severe epilepsy of childhood onset: A case series.
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5-HT1A receptor agonists modify epileptic seizures in three experimental models in rats.
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Serotonergic Modulation as Effective Treatment for Dravet Syndrome in a Zebrafish Mutant Model.
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5-HT2CRs expressed by pro-opiomelanocortin neurons regulate energy homeostasis.
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International Union of Pharmacology classification of receptors for 5-hydroxytryptamine (Serotonin).
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Localization of the 5-hydroxytryptamine2C receptor protein in human and rat brain using specific antisera.
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Serotonin2C ligands exhibiting full negative and positive intrinsic activity elicit purposeless oral movements in rats: distinct effects of agonists and inverse agonists in a rat model of Parkinson's disease.
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The 5-HT2C receptor agonist lorcaserin reduces nicotine self-administration, discrimination, and reinstatement: relationship to feeding behavior and impulse control.
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CP-809,101, a selective 5-HT2C agonist, shows activity in animal models of antipsychotic activity.
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Serotonin 5-HT2C receptor RNA editing alters receptor basal activity: implications for serotonergic signal transduction.
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Biochemical and electrophysiological evidence that RO 60-0175 inhibits mesolimbic dopaminergic function through serotonin(2C) receptors.
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Characterization of the discriminative stimulus effects of lorcaserin in rats.
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In vivo evidence that 5-HT(2C) receptors inhibit 5-HT neuronal activity via a GABAergic mechanism.
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Cortical drive and thalamic feed-forward inhibition control thalamic output synchrony during absence seizures.
Cian McCafferty, François David, Marcello Venzi, Magor L Lőrincz, Francis Delicata, Zoe Atherton, Gregorio Recchia, Gergely Orban, Régis C Lambert, Giuseppe Di Giovanni,[...]. Nat Neurosci 2018
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Co-cited is the co-citation frequency, indicating how many articles cite the article together with the query article. Similarity is the co-citation as percentage of the times cited of the query article or the article in the search results, whichever is the lowest. These numbers are calculated for the last 100 citations when articles are cited more than 100 times.