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Times Cited: 119
Times Cited: 119
Times Cited
Times Co-cited
Similarity
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Recent advances in the phencyclidine model of schizophrenia.
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NMDA receptor subunit diversity: impact on receptor properties, synaptic plasticity and disease.
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Synaptic and extrasynaptic NMDA receptors are gated by different endogenous coagonists.
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Multiple risk pathways for schizophrenia converge in serine racemase knockout mice, a mouse model of NMDA receptor hypofunction.
Darrick T Balu, Yan Li, Matthew D Puhl, Michael A Benneyworth, Alo C Basu, Shunsuke Takagi, Vadim Y Bolshakov, Joseph T Coyle. Proc Natl Acad Sci U S A 2013
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Molecular evidence of N-methyl-D-aspartate receptor hypofunction in schizophrenia.
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A CSF and postmortem brain study of D-serine metabolic parameters in schizophrenia.
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From revolution to evolution: the glutamate hypothesis of schizophrenia and its implication for treatment.
Bita Moghaddam, Daniel Javitt. Neuropsychopharmacology 2012
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Decreased serum levels of D-serine in patients with schizophrenia: evidence in support of the N-methyl-D-aspartate receptor hypofunction hypothesis of schizophrenia.
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D-serine is an endogenous ligand for the glycine site of the N-methyl-D-aspartate receptor.
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Postnatal NMDA receptor ablation in corticolimbic interneurons confers schizophrenia-like phenotypes.
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De novo mutations in schizophrenia implicate synaptic networks.
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Serine racemase deletion disrupts memory for order and alters cortical dendritic morphology.
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L M DeVito, D T Balu, B R Kanter, C Lykken, A C Basu, J T Coyle, H Eichenbaum. Genes Brain Behav 2011
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Targeted disruption of serine racemase affects glutamatergic neurotransmission and behavior.
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A critical role for NMDA receptors in parvalbumin interneurons for gamma rhythm induction and behavior.
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Schizophrenia risk from complex variation of complement component 4.
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Low d-serine levels in schizophrenia: A systematic review and meta-analysis.
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Adjunctive sarcosine plus benzoate improved cognitive function in chronic schizophrenia patients with constant clinical symptoms: A randomised, double-blind, placebo-controlled trial.
Chun-Yuan Lin, Sun-Yuan Liang, Yue-Cune Chang, Shuo-Yen Ting, Ching-Ling Kao, Yu-Hsin Wu, Guochuan E Tsai, Hsien-Yuan Lane. World J Biol Psychiatry 2017
Chun-Yuan Lin, Sun-Yuan Liang, Yue-Cune Chang, Shuo-Yen Ting, Ching-Ling Kao, Yu-Hsin Wu, Guochuan E Tsai, Hsien-Yuan Lane. World J Biol Psychiatry 2017
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Mechanism of NMDA Receptor Inhibition and Activation.
Shujia Zhu, Richard A Stein, Craig Yoshioka, Chia-Hsueh Lee, April Goehring, Hassane S Mchaourab, Eric Gouaux. Cell 2016
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Circuit-based framework for understanding neurotransmitter and risk gene interactions in schizophrenia.
John E Lisman, Joseph T Coyle, Robert W Green, Daniel C Javitt, Francine M Benes, Stephan Heckers, Anthony A Grace. Trends Neurosci 2008
John E Lisman, Joseph T Coyle, Robert W Green, Daniel C Javitt, Francine M Benes, Stephan Heckers, Anthony A Grace. Trends Neurosci 2008
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Add-on treatment of benzoate for schizophrenia: a randomized, double-blind, placebo-controlled trial of D-amino acid oxidase inhibitor.
Hsien-Yuan Lane, Ching-Hua Lin, Michael F Green, Gerhard Hellemann, Chih-Chia Huang, Po-Wei Chen, Rene Tun, Yue-Cung Chang, Guochuan E Tsai. JAMA Psychiatry 2013
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7
Sarcosine or D-serine add-on treatment for acute exacerbation of schizophrenia: a randomized, double-blind, placebo-controlled study.
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7
The NMDA receptor 'glycine modulatory site' in schizophrenia: D-serine, glycine, and beyond.
Darrick T Balu, Joseph T Coyle. Curr Opin Pharmacol 2015
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NMDA receptor subunits: diversity, development and disease.
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GABAergic interneuron origin of schizophrenia pathophysiology.
Kazu Nakazawa, Veronika Zsiros, Zhihong Jiang, Kazuhito Nakao, Stefan Kolata, Shuqin Zhang, Juan E Belforte. Neuropharmacology 2012
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A polygenic burden of rare disruptive mutations in schizophrenia.
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Glycine potentiates the NMDA response in cultured mouse brain neurons.
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De novo CNV analysis implicates specific abnormalities of postsynaptic signalling complexes in the pathogenesis of schizophrenia.
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High dose D-serine in the treatment of schizophrenia.
Joshua T Kantrowitz, Anil K Malhotra, Barbara Cornblatt, Gail Silipo, Andrea Balla, Raymond F Suckow, Cyril D'Souza, John Saksa, Scott W Woods, Daniel C Javitt. Schizophr Res 2010
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Improvement in mismatch negativity generation during d-serine treatment in schizophrenia: Correlation with symptoms.
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Glycine transporter I inhibitor, N-methylglycine (sarcosine), added to antipsychotics for the treatment of schizophrenia.
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Common polygenic variation contributes to risk of schizophrenia and bipolar disorder.
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D-serine added to antipsychotics for the treatment of schizophrenia.
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Serine racemase is predominantly localized in neurons in mouse brain.
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NMDA receptor hypofunction produces opposite effects on prefrontal cortex interneurons and pyramidal neurons.
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NMDA receptor antagonist rodent models for cognition in schizophrenia and identification of novel drug treatments, an update.
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NMDA receptor trafficking in synaptic plasticity and neuropsychiatric disorders.
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The Cognitive and Negative Symptoms in Schizophrenia Trial (CONSIST): the efficacy of glutamatergic agents for negative symptoms and cognitive impairments.
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Identity of endogenous NMDAR glycine site agonist in amygdala is determined by synaptic activity level.
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Pyramidal cell selective ablation of N-methyl-D-aspartate receptor 1 causes increase in cellular and network excitability.
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7
Co-cited is the co-citation frequency, indicating how many articles cite the article together with the query article. Similarity is the co-citation as percentage of the times cited of the query article or the article in the search results, whichever is the lowest. These numbers are calculated for the last 100 citations when articles are cited more than 100 times.