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Times Cited: 582
Times Cited
Times Co-cited
Similarity
Synaptic pruning by microglia is necessary for normal brain development.
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Fate mapping analysis reveals that adult microglia derive from primitive macrophages.
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Single-Cell RNA Sequencing of Microglia throughout the Mouse Lifespan and in the Injured Brain Reveals Complex Cell-State Changes.
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Microglia sculpt postnatal neural circuits in an activity and complement-dependent manner.
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A Unique Microglia Type Associated with Restricting Development of Alzheimer's Disease.
Hadas Keren-Shaul, Amit Spinrad, Assaf Weiner, Orit Matcovitch-Natan, Raz Dvir-Szternfeld, Tyler K Ulland, Eyal David, Kuti Baruch, David Lara-Astaiso, Beata Toth,[...]. Cell 2017
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Microglia promote learning-dependent synapse formation through brain-derived neurotrophic factor.
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Microbiome Influences Prenatal and Adult Microglia in a Sex-Specific Manner.
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Host microbiota constantly control maturation and function of microglia in the CNS.
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Spatial and temporal heterogeneity of mouse and human microglia at single-cell resolution.
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Developmental Heterogeneity of Microglia and Brain Myeloid Cells Revealed by Deep Single-Cell RNA Sequencing.
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Identification of a unique TGF-β-dependent molecular and functional signature in microglia.
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Microglia modulate wiring of the embryonic forebrain.
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Microglial brain region-dependent diversity and selective regional sensitivities to aging.
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Microglia regulate the number of neural precursor cells in the developing cerebral cortex.
Christopher L Cunningham, Verónica Martínez-Cerdeño, Stephen C Noctor. J Neurosci 2013
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Microglia Heterogeneity in the Single-Cell Era.
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Microglia emerge from erythromyeloid precursors via Pu.1- and Irf8-dependent pathways.
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Resting microglial cells are highly dynamic surveillants of brain parenchyma in vivo.
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New tools for studying microglia in the mouse and human CNS.
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Microglia shape adult hippocampal neurogenesis through apoptosis-coupled phagocytosis.
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A lineage of myeloid cells independent of Myb and hematopoietic stem cells.
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A new fate mapping system reveals context-dependent random or clonal expansion of microglia.
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Microglia contact induces synapse formation in developing somatosensory cortex.
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The TREM2-APOE Pathway Drives the Transcriptional Phenotype of Dysfunctional Microglia in Neurodegenerative Diseases.
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Colony-stimulating factor 1 receptor signaling is necessary for microglia viability, unmasking a microglia progenitor cell in the adult brain.
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Deficient neuron-microglia signaling results in impaired functional brain connectivity and social behavior.
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The classical complement cascade mediates CNS synapse elimination.
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Origin, fate and dynamics of macrophages at central nervous system interfaces.
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Environment drives selection and function of enhancers controlling tissue-specific macrophage identities.
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Tissue-resident macrophage enhancer landscapes are shaped by the local microenvironment.
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Microglia contribute to normal myelinogenesis and to oligodendrocyte progenitor maintenance during adulthood.
Nora Hagemeyer, Klara-Maria Hanft, Maria-Anna Akriditou, Nicole Unger, Eun S Park, E Richard Stanley, Ori Staszewski, Leda Dimou, Marco Prinz. Acta Neuropathol 2017
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Tissue-resident macrophages originate from yolk-sac-derived erythro-myeloid progenitors.
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An environment-dependent transcriptional network specifies human microglia identity.
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A Combination of Ontogeny and CNS Environment Establishes Microglial Identity.
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Fate mapping reveals origins and dynamics of monocytes and tissue macrophages under homeostasis.
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A novel microglial subset plays a key role in myelinogenesis in developing brain.
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Microglia remodel synapses by presynaptic trogocytosis and spine head filopodia induction.
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Local self-renewal can sustain CNS microglia maintenance and function throughout adult life.
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Co-cited is the co-citation frequency, indicating how many articles cite the article together with the query article. Similarity is the co-citation as percentage of the times cited of the query article or the article in the search results, whichever is the lowest. These numbers are calculated for the last 100 citations when articles are cited more than 100 times.